HIV-1 adaptation to NK cell mediated immune pressure

[1]  M. Altfeld,et al.  Control of human viral infections by natural killer cells. , 2013, Annual review of immunology.

[2]  G. d’Ettorre,et al.  Soluble ligands for the NKG2D receptor are released during HIV‐1 infection and impair NKG2D expression and cytotoxicity of NK cells , 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[3]  M. Altfeld,et al.  Evasion from NK cell-mediated immune responses by HIV-1. , 2012, Microbes and infection.

[4]  P. Parham,et al.  Mutation at Positively Selected Positions in the Binding Site for HLA-C Shows That KIR2DL1 Is a More Refined but Less Adaptable NK Cell Receptor Than KIR2DL3 , 2012, The Journal of Immunology.

[5]  Morten Nielsen,et al.  Peptide‐MHC class I stability is a better predictor than peptide affinity of CTL immunogenicity , 2012, European journal of immunology.

[6]  Guido Ferrari,et al.  Immune-correlates analysis of an HIV-1 vaccine efficacy trial. , 2012, The New England journal of medicine.

[7]  A. Santoni,et al.  The Human Immunodeficiency Virus Type 1 Nef and Vpu Proteins Downregulate the Natural Killer Cell-Activating Ligand PVR , 2012, Journal of Virology.

[8]  Søren Buus,et al.  Real-time, high-throughput measurements of peptide-MHC-I dissociation using a scintillation proximity assay. , 2011, Journal of immunological methods.

[9]  K. Shianna,et al.  Copy Number Variation of KIR Genes Influences HIV-1 Control , 2011, PLoS biology.

[10]  M. Altfeld,et al.  Common HIV-1 Peptide Variants Mediate Differential Binding of KIR3DL1 to HLA-Bw4 Molecules , 2011, Journal of Virology.

[11]  Angela R. McLean,et al.  Modelling the Evolution and Spread of HIV Immune Escape Mutants , 2010, PLoS pathogens.

[12]  S. Buus,et al.  Efficacious Early Antiviral Activity of HIV Gag- and Pol-Specific HLA-B*2705-Restricted CD8+ T Cells , 2010, Journal of Virology.

[13]  T. Elliott,et al.  Peptide antagonism as a mechanism for NK cell activation , 2010, Proceedings of the National Academy of Sciences.

[14]  E. Zimmerman,et al.  HIV-1 Vpr Triggers Natural Killer Cell–Mediated Lysis of Infected Cells through Activation of the ATR-Mediated DNA Damage Response , 2009, PLoS pathogens.

[15]  S. Rowland-Jones,et al.  Reciprocal recognition of an HLA-Cw4-restricted HIV-1 gp120 epitope by CD8+ T cells and NK cells , 2009, AIDS.

[16]  Morten Nielsen,et al.  Pan-specific MHC class I predictors: a benchmark of HLA class I pan-specific prediction methods , 2009, Bioinform..

[17]  Todd M. Allen,et al.  Protective HLA Class I Alleles That Restrict Acute-Phase CD8+ T-Cell Responses Are Associated with Viral Escape Mutations Located in Highly Conserved Regions of Human Immunodeficiency Virus Type 1 , 2008, Journal of Virology.

[18]  David Heckerman,et al.  Phylogenetic Dependency Networks: Inferring Patterns of CTL Escape and Codon Covariation in HIV-1 Gag , 2008, PLoS Comput. Biol..

[19]  J. Routy,et al.  A combined genotype of KIR3DL1 high expressing alleles and HLA-B*57 is associated with a reduced risk of HIV infection , 2008, AIDS.

[20]  Lewis L Lanier,et al.  Up on the tightrope: natural killer cell activation and inhibition , 2008, Nature Immunology.

[21]  P. Parham,et al.  Synergistic Polymorphism at Two Positions Distal to the Ligand-Binding Site Makes KIR2DL2 a Stronger Receptor for HLA-C Than KIR2DL31 , 2008, The Journal of Immunology.

[22]  J. Routy,et al.  Increased proportion of KIR3DS1 homozygotes in HIV-exposed uninfected individuals , 2008, AIDS.

[23]  W. Klitz,et al.  Corrigendum to “High-resolution HLA alleles and haplotypes in the United States population” [Hum Imunol 68 (2007) 779–788] , 2008 .

[24]  O. Lund,et al.  NetMHCpan, a method for MHC class I binding prediction beyond humans , 2008, Immunogenetics.

[25]  William H. Carr,et al.  Differential natural killer cell–mediated inhibition of HIV-1 replication based on distinct KIR/HLA subtypes , 2007, The Journal of experimental medicine.

[26]  B. Walker,et al.  Portable flanking sequences modulate CTL epitope processing. , 2007, The Journal of clinical investigation.

[27]  J. Borghans,et al.  HLA Alleles Associated with Slow Progression to AIDS Truly Prefer to Present HIV-1 p24 , 2007, PLoS ONE.

[28]  Todd M. Allen,et al.  Escape from the Dominant HLA-B27-Restricted Cytotoxic T-Lymphocyte Response in Gag Is Associated with a Dramatic Reduction in Human Immunodeficiency Virus Type 1 Replication , 2007, Journal of Virology.

[29]  Loren Gragert,et al.  High-resolution HLA alleles and haplotypes in the United States population. , 2007, Human immunology.

[30]  O. Lund,et al.  NetMHCpan, a Method for Quantitative Predictions of Peptide Binding to Any HLA-A and -B Locus Protein of Known Sequence , 2007, PloS one.

[31]  J. Ward,et al.  HIV modulates the expression of ligands important in triggering natural killer cell cytotoxic responses on infected primary T-cell blasts. , 2007, Blood.

[32]  M. Altfeld,et al.  'Unleashed' natural killers hinder HIV , 2007, Nature Genetics.

[33]  Amalio Telenti,et al.  Innate partnership of HLA-B and KIR3DL1 subtypes against HIV-1 , 2007, Nature Genetics.

[34]  B. Walker,et al.  Effective T-Cell Responses Select Human Immunodeficiency Virus Mutants and Slow Disease Progression , 2007, Journal of Virology.

[35]  M. Carrington,et al.  Cutting Edge: Allele-Specific and Peptide-Dependent Interactions between KIR3DL1 and HLA-A and HLA-B12 , 2007, The Journal of Immunology.

[36]  A. Santoni,et al.  Human immunodeficiency virus 1 Nef protein downmodulates the ligands of the activating receptor NKG2D and inhibits natural killer cell-mediated cytotoxicity. , 2007, The Journal of general virology.

[37]  Morten Nielsen,et al.  A Community Resource Benchmarking Predictions of Peptide Binding to MHC-I Molecules , 2006, PLoS Comput. Biol..

[38]  David Heckerman,et al.  Leveraging Information Across HLA Alleles/Supertypes Improves Epitope Prediction , 2006, RECOMB.

[39]  Becca Asquith,et al.  Inefficient Cytotoxic T Lymphocyte–Mediated Killing of HIV-1–Infected Cells In Vivo , 2006, PLoS biology.

[40]  N. Crepaz,et al.  Higher incidence of clear cell adenocarcinoma of the cervix and vagina among women born between 1947 and 1971 in the United States , 2011, Cancer Causes & Control.

[41]  Eric Vivier,et al.  Recognition of peptide-MHC class I complexes by activating killer immunoglobulin-like receptors. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[42]  A. McMichael,et al.  Crystal structures and KIR3DL1 recognition of three immunodominant viral peptides complexed to HLA‐B*2705 , 2005, European journal of immunology.

[43]  W. Yokoyama Specific and non-specific natural killer cell responses to viral infection. , 2005, Advances in experimental medicine and biology.

[44]  Lewis L Lanier,et al.  NK cell recognition. , 2005, Annual review of immunology.

[45]  Jessica M Malenfant,et al.  CD107a as a functional marker for the identification of natural killer cell activity. , 2004, Journal of immunological methods.

[46]  Salim I. Khakoo,et al.  HLA and NK Cell Inhibitory Receptor Genes in Resolving Hepatitis C Virus Infection , 2004, Science.

[47]  Philip J. R. Goulder,et al.  HIV and SIV CTL escape: implications for vaccine design , 2004, Nature Reviews Immunology.

[48]  M. Leppert,et al.  SNP haplotypes and allele frequencies show evidence for disruptive and balancing selection in the human leukocyte receptor complex , 2004, Immunogenetics.

[49]  S. Rowland-Jones,et al.  Recognition of HLA‐A3 and HLA‐A11 by KIR3DL2 is peptide‐specific , 2004, European journal of immunology.

[50]  Keith Hoots,et al.  Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS , 2002, Nature Genetics.

[51]  C. Moore,et al.  Evidence of HIV-1 Adaptation to HLA-Restricted Immune Responses at a Population Level , 2002, Science.

[52]  Peter Parham,et al.  KIR: diverse, rapidly evolving receptors of innate and adaptive immunity. , 2002, Annual review of immunology.

[53]  Jeffrey C. Boyington,et al.  Crystal structure of an NK cell immunoglobulin-like receptor in complex with its class I MHC ligand , 2000, Nature.

[54]  D. Baltimore,et al.  The selective downregulation of class I major histocompatibility complex proteins by HIV-1 protects HIV-infected cells from NK cells. , 1999, Immunity.

[55]  O. Mandelboim,et al.  Self and viral peptides can initiate lysis by autologous natural killer cells. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[56]  Eric O Long,et al.  The Direct Binding of a p58 Killer Cell Inhibitory Receptor to Human Histocompatibility Leukocyte Antigen (HLA)-Cw4 Exhibits Peptide Selectivity , 1997, The Journal of experimental medicine.

[57]  O. Mandelboim,et al.  The binding site of NK receptors on HLA-C molecules. , 1997, Immunity.

[58]  Martin A. Nowak,et al.  Late escape from an immunodominant cytotoxic T-lymphocyte response associated with progression to AIDS , 1997, Nature Medicine.

[59]  Eric O Long,et al.  Peptide sequence requirements for the recognition of HLA-B*2705 by specific natural killer cells. , 1996, Journal of immunology.

[60]  Eric O Long,et al.  Peptide specificity in the recognition of MHC class I by natural killer cell clones , 1995, Science.

[61]  Charles R. M. Bangham,et al.  Human immunodeficiency virus genetic variation that can escape cytotoxic T cell recognition , 1991, Nature.

[62]  H. Gendelman,et al.  Production of acquired immunodeficiency syndrome-associated retrovirus in human and nonhuman cells transfected with an infectious molecular clone , 1986, Journal of virology.