Inhibition of osteoclast differentiation and collagen antibody-induced arthritis by CTHRC1.

[1]  D. Sterchi Bone , 2019, Bancroft's Theory and Practice of Histological Techniques.

[2]  R. Baron,et al.  The Actin‐Binding Protein Cofilin and Its Interaction With Cortactin Are Required for Podosome Patterning in Osteoclasts and Bone Resorption In Vivo and In Vitro , 2016, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[3]  V. Lindner,et al.  Collagen triple helix repeat containing 1 is a new promigratory marker of arthritic pannus , 2016, Arthritis Research & Therapy.

[4]  L. Liaw,et al.  Cthrc1 controls adipose tissue formation, body composition, and physical activity , 2015, Obesity.

[5]  Christine W. Duarte,et al.  Elevated Plasma Levels of the Pituitary Hormone Cthrc1 in Individuals with Red Hair but Not in Patients with Solid Tumors , 2014, PloS one.

[6]  D. Moore,et al.  Corrigendum: Ptpn11 deletion in a novel progenitor causes metachondromatosis by inducing hedgehog signalling , 2014, Nature.

[7]  D. Ornitz,et al.  Osx-Cre Targets Multiple Cell Types besides Osteoblast Lineage in Postnatal Mice , 2014, PloS one.

[8]  T. Martin,et al.  Coupling the activities of bone formation and resorption: a multitude of signals within the basic multicellular unit. , 2014, BoneKEy reports.

[9]  H. Aburatani,et al.  Osteoclast-secreted CTHRC1 in the coupling of bone resorption to formation. , 2013, The Journal of clinical investigation.

[10]  N. Miyasaka,et al.  miR-31 controls osteoclast formation and bone resorption by targeting RhoA , 2013, Arthritis Research & Therapy.

[11]  M. Bouxsein,et al.  Sclerostin antibody inhibits skeletal deterioration due to reduced mechanical loading , 2013, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[12]  O. MacDougald,et al.  Marrow fat and bone--new perspectives. , 2013, The Journal of clinical endocrinology and metabolism.

[13]  J. Kanis,et al.  Standardized nomenclature, symbols, and units for bone histomorphometry: A 2012 update of the report of the ASBMR Histomorphometry Nomenclature Committee , 2013, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[14]  L. Liaw,et al.  Cthrc1, a Novel Circulating Hormone Regulating Metabolism , 2012, PloS one.

[15]  R. Baron,et al.  Energy expenditure and bone formation share a common sensitivity to AP‐1 transcription in the hypothalamus , 2012, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[16]  R. Kladney,et al.  Cathepsin K-Cre Causes Unexpected Germline Deletion of Genes in Mice , 2012, PloS one.

[17]  T. Martin,et al.  Wnt5a-Ror2 signaling between osteoblast-lineage cells and osteoclast precursors enhances osteoclastogenesis , 2012, Nature Medicine.

[18]  L. Maile,et al.  Insulin‐like growth factor‐binding protein‐2 is required for osteoclast differentiation , 2012, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[19]  M. Schuenke,et al.  Collagen Triple Helix Repeat Containing-1 (cthrc1) Attenuates Skeletal Muscle Atrophy: 1757 , 2011 .

[20]  V. Vives,et al.  The Rac1 exchange factor Dock5 is essential for bone resorption by osteoclasts , 2011, Journal of Bone and Mineral Research.

[21]  Ralph Müller,et al.  Guidelines for assessment of bone microstructure in rodents using micro–computed tomography , 2010, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[22]  C. Ackert-Bicknell,et al.  A circadian-regulated gene, Nocturnin, promotes adipogenesis by stimulating PPAR-γ nuclear translocation , 2010, Proceedings of the National Academy of Sciences.

[23]  C. Rosen,et al.  Fat targets for skeletal health , 2009, Nature Reviews Rheumatology.

[24]  A. Klibanski,et al.  Bone, fat, and body composition: evolving concepts in the pathogenesis of osteoporosis. , 2009, The American journal of medicine.

[25]  J. Deng,et al.  Cthrc1 Is a Positive Regulator of Osteoblastic Bone Formation , 2008, PloS one.

[26]  Y. Minami,et al.  Cthrc1 selectively activates the planar cell polarity pathway of Wnt signaling by stabilizing the Wnt-receptor complex. , 2008, Developmental Cell.

[27]  Soo Young Lee,et al.  Rac1 GTPase regulates osteoclast differentiation through TRANCE-induced NF-kappa B activation. , 2006, Molecular and cellular biochemistry.

[28]  Soo Young Lee,et al.  Rac1 GTPase regulates osteoclast differentiation through TRANCE-induced NF-κB activation , 2006, Molecular and Cellular Biochemistry.

[29]  L. Liaw,et al.  Collagen Triple Helix Repeat Containing 1, a Novel Secreted Protein in Injured and Diseased Arteries, Inhibits Collagen Expression and Promotes Cell Migration , 2005, Circulation research.

[30]  S. Cowin,et al.  Ultrastructure of the osteocyte process and its pericellular matrix. , 2004, The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology.

[31]  David L. Lacey,et al.  Osteoclast differentiation and activation , 2003, Nature.

[32]  Steffen Jung,et al.  Regulation of Peripheral Lymph Node Genesis by the Tumor Necrosis Factor Family Member Trance , 2000, The Journal of experimental medicine.

[33]  S. Morony,et al.  OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis , 1999, Nature.