Expression patterns of the HIV type 1 coreceptors CCR5 and CXCR4 on CD4+ T cells and monocytes from cord and adult blood.
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J. Moore | D. Ho | J. Hoxie | John P. Moore | W. Borkowsky | H. Mo | H. Pollack | S. Monard | D D Ho | S Monard | J P Moore | H Mo | L. Wu | W Borkowsky | L Wu | H Pollack | J Ip | G Rochford | J Hoxie | G. Rochford | James Ip | J. Ip | Lijun Wu | L. Wu
[1] W. Scott,et al. Longitudinal studies of viral sequence, viral phenotype, and immunologic parameters of human immunodeficiency virus type 1 infection in perinatally infected twins with discordant disease courses , 1996, Journal of virology.
[2] V. Deneys,et al. Age-related changes in human blood lymphocyte subpopulations. , 1992, The Journal of pediatrics.
[3] J. Moore,et al. Chemoattractants attract HIV researchers , 1996, The Journal of experimental medicine.
[4] C. Mackay,et al. Interaction of Chemokine Receptor CCR5 with its Ligands: Multiple Domains for HIV-1 gp120 Binding and a Single Domain for Chemokine Binding , 1997, The Journal of experimental medicine.
[5] Stephen C. Peiper,et al. Identification of a major co-receptor for primary isolates of HIV-1 , 1996, Nature.
[6] F. Miedema,et al. Functional and phenotypic evidence for a selective loss of memory T cells in asymptomatic human immunodeficiency virus-infected men. , 1990, The Journal of clinical investigation.
[7] Bruce L. Levine,et al. Antiviral Effect and Ex Vivo CD4+ T Cell Proliferation in HIV-Positive Patients as a Result of CD28 Costimulation , 1996, Science.
[8] C. Broder,et al. CC CKR5: A RANTES, MIP-1α, MIP-1ॆ Receptor as a Fusion Cofactor for Macrophage-Tropic HIV-1 , 1996, Science.
[9] Luc Montagnier,et al. T-lymphocyte T4 molecule behaves as the receptor for human retrovirus LAV , 1984, Nature.
[10] D. Richman,et al. The impact of the syncytium-inducing phenotype of human immunodeficiency virus on disease progression. , 1994, The Journal of infectious diseases.
[11] A. Namazi,et al. Human immunodeficiency virus-type 1 replication can be increased in peripheral blood of seropositive patients after influenza vaccination , 1995 .
[12] J. Justement,et al. Preferential infection of CD4+ memory T cells by human immunodeficiency virus type 1: evidence for a role in the selective T-cell functional defects observed in infected individuals. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[13] C. Chappey,et al. Genetic analysis of human immunodeficiency virus type 1 envelope V3 region isolates from mothers and infants after perinatal transmission , 1995, Journal of virology.
[14] S. Spector,et al. Clinical significance of human immunodeficiency virus type 1 phenotypes in infected children. , 1994, The Journal of infectious diseases.
[15] Ying Sun,et al. The β-Chemokine Receptors CCR3 and CCR5 Facilitate Infection by Primary HIV-1 Isolates , 1996, Cell.
[16] D. Goeddel,et al. Selective attraction of monocytes and T lymphocytes of the memory phenotype by cytokine RANTES , 1990, Nature.
[17] R. Doms,et al. CD4-Independent Infection by HIV-2 Is Mediated by Fusin/CXCR4 , 1996, Cell.
[18] J K Nicholson,et al. Binding of HTLV-III/LAV to T4+ T cells by a complex of the 110K viral protein and the T4 molecule. , 1986, Science.
[19] Marc Parmentier,et al. A Dual-Tropic Primary HIV-1 Isolate That Uses Fusin and the β-Chemokine Receptors CKR-5, CKR-3, and CKR-2b as Fusion Cofactors , 1996, Cell.
[20] R. Connor,et al. Change in Coreceptor Use Correlates with Disease Progression in HIV-1–Infected Individuals , 1997, The Journal of experimental medicine.
[21] L. Saravolatz,et al. Changes in virus load markers during AIDS-associated opportunistic diseases in human immunodeficiency virus-infected persons. , 1996, The Journal of infectious diseases.
[22] Virginia Litwin,et al. HIV-1 entry into CD4+ cells is mediated by the chemokine receptor CC-CKR-5 , 1996, Nature.
[23] I. Chen,et al. Increased susceptibility of neonatal monocyte/macrophages to HIV-1 infection. , 1993, AIDS research and human retroviruses.
[24] J. Hoxie,et al. Inhibition of human immunodeficiency virus fusion by a monoclonal antibody to a coreceptor (CXCR4) is both cell type and virus strain dependent , 1997, Journal of virology.
[25] D. Littman,et al. Expression cloning of new receptors used by simian and human immunodeficiency viruses , 1997, Nature.
[26] R. Weiss,et al. Primary, syncytium-inducing human immunodeficiency virus type 1 isolates are dual-tropic and most can use either Lestr or CCR5 as coreceptors for virus entry , 1996, Journal of virology.
[27] Ying Sun,et al. Two Orphan Seven-Transmembrane Segment Receptors Which Are Expressed in CD4-positive Cells Support Simian Immunodeficiency Virus Infection , 1997, The Journal of experimental medicine.
[28] D. Taub,et al. Preferential migration of activated CD4+ and CD8+ T cells in response to MIP-1 alpha and MIP-1 beta , 1993, Science.
[29] R. Koup,et al. Macrophages and CD4+ T lymphocytes from two multiply exposed, uninfected individuals resist infection with primary non-syncytium-inducing isolates of human immunodeficiency virus type 1 , 1996, Journal of virology.
[30] Robin A. Weiss,et al. The T4 gene encodes the AIDS virus receptor and is expressed in the immune system and the brain , 1986, Cell.
[31] M. Baggiolini,et al. Interleukin-2 regulates CC chemokine receptor expression and chemotactic responsiveness in T lymphocytes , 1996, The Journal of experimental medicine.
[32] R. Doms,et al. Role of CCR5 in infection of primary macrophages and lymphocytes by macrophage-tropic strains of human immunodeficiency virus: resistance to patient-derived and prototype isolates resulting from the delta ccr5 mutation , 1997, Journal of virology.
[33] D. Ho,et al. Human immunodeficiency virus type 1 variants with increased replicative capacity develop during the asymptomatic stage before disease progression , 1994, Journal of virology.
[34] L. Bradley,et al. Predominance of T cells that express CD45R in the CD4+ helper/inducer lymphocyte subset of neonates. , 1989, Clinical immunology and immunopathology.
[35] K. Peden,et al. STRL33, A Novel Chemokine Receptor–like Protein, Functions as a Fusion Cofactor for Both Macrophage-tropic and T Cell Line–tropic HIV-1 , 1997, The Journal of experimental medicine.
[36] T. Elbeik,et al. Activation of virus replication after vaccination of HIV-1-infected individuals , 1995, The Journal of experimental medicine.
[37] Nancy Sullivan,et al. CCR5 Levels and Expression Pattern Correlate with Infectability by Macrophage-tropic HIV-1, In Vitro , 1997, The Journal of experimental medicine.
[38] Paul E. Kennedy,et al. HIV-1 Entry Cofactor: Functional cDNA Cloning of a Seven-Transmembrane, G Protein-Coupled Receptor , 1996, Science.
[39] G. Santini,et al. Age-related changes in human lymphocyte subsets: progressive reduction of the CD4 CD45R (suppressor inducer) population. , 1988, Clinical immunology and immunopathology.
[40] C. Fox,et al. Dynamics of virus versus host interaction in children with human immunodeficiency virus type 1 infection. , 1996, The Journal of infectious diseases.
[41] Bernhard Moser,et al. The CXC chemokine SDF-1 is the ligand for LESTR/fusin and prevents infection by T-cell-line-adapted HIV-1 , 1996, Nature.
[42] J. Moore. Coreceptors--Implications for HIV Pathogenesis and Therapy , 1997, Science.
[43] C. Peckham,et al. Mother-to-child transmission of the human immunodeficiency virus. , 1995, The New England journal of medicine.
[44] C. Mackay,et al. The HIV coreceptors CXCR4 and CCR5 are differentially expressed and regulated on human T lymphocytes. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[45] William C. Olson,et al. CD4-dependent, antibody-sensitive interactions between HIV-1 and its co-receptor CCR-5 , 1996, Nature.
[46] J. Albert,et al. Transmission of human immunodeficiency virus type 1 (HIV-1) from mother to child correlates with viral phenotype. , 1993, Virology.
[47] D. Scott‐Algara,et al. Differential requirements for HIV‐1 replication in naive and memory CD4 T cells from asymptomatic HIV‐1 seropositive carriers and AIDS patients , 1993, Clinical and experimental immunology.
[48] A. Ammann. Mother–infant HIV transmission: Making the most of what we know , 1996, Nature Medicine.
[49] H. Ullum,et al. Increased losses of CD4+CD45RA+ cells in late stages of HIV infection is related to increased risk of death: evidence from a cohort of 347 HIV‐infected individuals , 1997, AIDS.
[50] B. Levine,et al. Differential regulation of HIV-1 fusion cofactor expression by CD28 costimulation of CD4+ T cells. , 1997, Science.
[51] M. Moroni,et al. HIV type 1 phenotype correlates with the stage of infection in vertically infected children. , 1996, AIDS research and human retroviruses.
[52] E. Meese,et al. TYMSTR, a putative chemokine receptor selectively expressed in activated T cells, exhibits HIV-1 coreceptor function , 1997, Current Biology.
[53] D. Taub,et al. CD8+ and CD45RA+ human peripheral blood lymphocytes are potent sources of macrophage inflammatory protein 1α, interleukin‐8 and RANTES , 1995, European journal of immunology.
[54] D. Montefiori,et al. Antibody-mediated neutralization of primary isolates of human immunodeficiency virus type 1 in peripheral blood mononuclear cells is not affected by the initial activation state of the cells , 1997, Journal of virology.
[55] D. Mosier,et al. Macrophage-tropic HIV: critical for AIDS pathogenesis? , 1994, Immunology today.
[56] S. Skoda-Smith,et al. CD4+ memory T cells are the predominant population of HIV‐1‐infected lymphocytes in neonates and children , 1996, AIDS.
[57] C. Macken,et al. Maternal HIV-1 viral load and vertical transmission of infection: The Ariel Project for the prevention of HIV transmission from mother to infant , 1997, Nature Medicine.
[58] M. Greaves,et al. The CD4 (T4) antigen is an essential component of the receptor for the AIDS retrovirus , 1984, Nature.