Up-regulation and redistribution of protein kinase C-δ in chronically hypoxic heart
暂无分享,去创建一个
O. Novakova | R. Musters | J. Neckář | F. Kolář | R. J. Musters | M. Hlavackova | F. Novák | K. Kožichová
[1] K. Nakanishi,et al. Protein kinase C mRNA and protein expressions in hypobaric hypoxia‐induced cardiac hypertrophy in rats , 2010, Acta physiologica.
[2] Chengqun Huang,et al. Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole. , 2010, American journal of physiology. Heart and circulatory physiology.
[3] A. Boveris,et al. Mitochondrial nitric oxide metabolism during rat heart adaptation to high altitude: effect of sildenafil, L-NAME, and L-arginine treatments. , 2009, American journal of physiology. Heart and circulatory physiology.
[4] Qingbo Xu,et al. Proteomic and metabolomic analysis of cardioprotection: Interplay between protein kinase C epsilon and delta in regulating glucose metabolism of murine hearts. , 2009, Journal of molecular and cellular cardiology.
[5] Mourad Ogbi,et al. Delta Protein Kinase C Interacts with the d Subunit of the F1F0 ATPase in Neonatal Cardiac Myocytes Exposed to Hypoxia or Phorbol Ester , 2008, Journal of Biological Chemistry.
[6] M. Young,et al. Expression of mitochondrial regulatory genes parallels respiratory capacity and contractile function in a rat model of hypoxia-induced right ventricular hypertrophy , 2008, Molecular and Cellular Biochemistry.
[7] Qilin Yu,et al. Protein kinase C-epsilon coimmunoprecipitates with cytochrome oxidase subunit IV and is associated with improved cytochrome-c oxidase activity and cardioprotection. , 2007, American journal of physiology. Heart and circulatory physiology.
[8] S. Soltoff,et al. Rottlerin: an inappropriate and ineffective inhibitor of PKCdelta. , 2007, Trends in pharmacological sciences.
[9] Hideyuki Takahashi,et al. Mechanism of membrane redistribution of protein kinase C by its ATP-competitive inhibitors. , 2007, The Biochemical journal.
[10] O. Novakova,et al. Dietary Polyunsaturated Fatty Acids Alter Myocardial Protein Kinase C Expression and Affect Cardioprotection Induced by Chronic Hypoxia , 2007, Experimental biology and medicine.
[11] D. Mochly‐Rosen,et al. Cardioprotective mechanisms of PKC isozyme-selective activators and inhibitors in the treatment of ischemia-reperfusion injury. , 2007, Pharmacological research.
[12] T. Ravingerová,et al. Differential role of PI3K/Akt pathway in the infarct size limitation and antiarrhythmic protection in the rat heart , 2007, Molecular and Cellular Biochemistry.
[13] W. Paulus,et al. Cardioprotection Via Activation of Protein Kinase C-&dgr; Depends on Modulation of the Reverse Mode of the Na+/Ca2+ Exchanger , 2006, Circulation.
[14] J. Sadoshima,et al. Preconditioning effects of PKCδ , 2005 .
[15] Z. Bosnjak,et al. Preconditioning by Isoflurane Induces Lasting Sensitization of the Cardiac Sarcolemmal Adenosine Triphosphate–sensitive Potassium Channel by a Protein Kinase C-&dgr;–mediated Mechanism , 2005, Anesthesiology.
[16] D. Mochly‐Rosen,et al. δPKC-mediated activation of εPKC in ethanol-induced cardiac protection from ischemia , 2005 .
[17] D. Mochly‐Rosen,et al. Reperfusion-Induced Translocation of &dgr;PKC to Cardiac Mitochondria Prevents Pyruvate Dehydrogenase Reactivation , 2005, Circulation research.
[18] O. Novakova,et al. Increased expression and altered subcellular distribution of PKC-delta in chronically hypoxic rat myocardium: involvement in cardioprotection. , 2005, American journal of physiology. Heart and circulatory physiology.
[19] J. Mazat,et al. Time course of differential mitochondrial energy metabolism adaptation to chronic hypoxia in right and left ventricles. , 2005, Cardiovascular research.
[20] J. C. Hunter,et al. Protein kinase C distribution and translocation in rat myocardium: Methodological considerations. , 2005, Journal of pharmacological and toxicological methods.
[21] S. Steinberg. Distinctive activation mechanisms and functions for protein kinase Cdelta. , 2004, The Biochemical journal.
[22] D. Mochly‐Rosen,et al. Protein kinase Cdelta activation induces apoptosis in response to cardiac ischemia and reperfusion damage: a mechanism involving BAD and the mitochondria. , 2004, The Journal of biological chemistry.
[23] H. Ding,et al. Intermittent hypoxia protects the rat heart against ischemia/reperfusion injury by activating protein kinase C. , 2004, Life sciences.
[24] Qingbo Xu,et al. Loss of PKC-δ alters cardiac metabolism , 2004 .
[25] Otmar Pachinger,et al. Ischemic preconditioning exaggerates cardiac damage in PKC-δ null mice , 2004 .
[26] Lubo Zhang,et al. Effect of prenatal hypoxia on heat stress-mediated cardioprotection in adult rat heart. , 2004, American journal of physiology. Heart and circulatory physiology.
[27] R. Musters,et al. Reactive Oxygen Species Precede Protein Kinase C-&dgr; Activation Independent of Adenosine Triphosphate–sensitive Mitochondrial Channel Opening in Sevoflurane-induced Cardioprotection , 2004, Anesthesiology.
[28] P. Yock,et al. Inhibition of &dgr;-Protein Kinase C Protects Against Reperfusion Injury of the Ischemic Heart In Vivo , 2003, Circulation.
[29] H. Ding,et al. ATP-dependent potassium channels involved in the cardiac protection induced by intermittent hypoxia against ischemia/reperfusion injury. , 2003, Life sciences.
[30] M. Zaugg,et al. Translocation of Protein Kinase C Isoforms to Subcellular Targets in Ischemic and Anesthetic Preconditioning , 2003, Anesthesiology.
[31] F. Léon-Velarde,et al. Effects of nifedipine-induced pulmonary vasodilatation on cardiac receptors and protein kinase C isoforms in the chronically hypoxic rat , 2003, Pflügers Archiv.
[32] J. Molkentin,et al. Cross-regulation of Novel Protein Kinase C (PKC) Isoform Function in Cardiomyocytes , 2003, The Journal of Biological Chemistry.
[33] G. Grover,et al. Effects of mitochondrial K(ATP) modulators on cardioprotection induced by chronic high altitude hypoxia in rats. , 2002, Cardiovascular research.
[34] K. Pritchard,et al. Activation of Protein Kinases in Chronically Hypoxic Infant Human and Rabbit Hearts: Role in Cardioprotection , 2002, Circulation.
[35] P. Ping,et al. Nitric Oxide (NO) Induces Nitration of Protein Kinase Cε (PKCε), Facilitating PKCε Translocation via Enhanced PKCε-RACK2 Interactions , 2002, The Journal of Biological Chemistry.
[36] P. Rock,et al. H2O2 opens mitochondrial KATPchannels and inhibits GABA receptors via protein kinase C-ε in cardiomyocytes , 2002 .
[37] Jan Neckář,et al. Chronic hypoxia alters fatty acid composition of phospholipids in right and left ventricular myocardium , 2002, Molecular and Cellular Biochemistry.
[38] Y. Nishizuka,et al. Phosphorylation sites of protein kinase C δ in H2O2-treated cells and its activation by tyrosine kinase in vitro , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[39] G. Gross,et al. Sarcolemmal and mitochondrial K(atp)channels mediate cardioprotection in chronically hypoxic hearts. , 2001, Journal of molecular and cellular cardiology.
[40] Yigang Wang,et al. Essential activation of PKC-δ in opioid-initiated cardioprotection , 2001 .
[41] J. Downey,et al. Opening of Mitochondrial KATP Channels Triggers the Preconditioned State by Generating Free Radicals , 2000, Circulation research.
[42] F. Marks,et al. Rottlerin, a novel protein kinase inhibitor. , 1994, Biochemical and biophysical research communications.
[43] C. Hoppel,et al. Biochemical properties of subsarcolemmal and interfibrillar mitochondria isolated from rat cardiac muscle. , 1977, The Journal of biological chemistry.
[44] O. Novakova,et al. Role of oxidative stress in PKC-δ upregulation and cardioprotection induced by chronic intermittent hypoxia , 2007 .
[45] L. Liaudet,et al. Nitric oxide and peroxynitrite in health and disease. , 2007, Physiological reviews.
[46] Mourad Ogbi,et al. Protein kinase Cepsilon interacts with cytochrome c oxidase subunit IV and enhances cytochrome c oxidase activity in neonatal cardiac myocyte preconditioning. , 2006, The Biochemical journal.
[47] J. Sadoshima,et al. Preconditioning effects of PKCdelta. , 2005, Journal of molecular and cellular cardiology.
[48] D. Mochly‐Rosen,et al. DeltaPKC-mediated activation of epsilonPKC in ethanol-induced cardiac protection from ischemia. , 2005, Journal of molecular and cellular cardiology.
[49] F. Kolář,et al. Molecular mechanisms of cardiac protection by adaptation to chronic hypoxia. , 2004, Physiological research.
[50] Otmar Pachinger,et al. Ischemic preconditioning exaggerates cardiac damage in PKC-delta null mice. , 2004, American journal of physiology. Heart and circulatory physiology.
[51] Qingbo Xu,et al. Loss of PKC-delta alters cardiac metabolism. , 2004, American journal of physiology. Heart and circulatory physiology.
[52] P. Ping,et al. Nitric oxide (NO) induces nitration of protein kinase Cepsilon (PKCepsilon ), facilitating PKCepsilon translocation via enhanced PKCepsilon -RACK2 interactions: a novel mechanism of no-triggered activation of PKCepsilon. , 2002, The Journal of biological chemistry.
[53] P. Rock,et al. H(2)O(2) opens mitochondrial K(ATP) channels and inhibits GABA receptors via protein kinase C-epsilon in cardiomyocytes. , 2002, American journal of physiology. Heart and circulatory physiology.
[54] Y. Wang,et al. Essential activation of PKC-delta in opioid-initiated cardioprotection. , 2001, American journal of physiology. Heart and circulatory physiology.
[55] V. Pelouch,et al. Intermittent high altitude--induced changes in energy metabolism in the rat myocardium and their reversibility. , 1989, Physiologia Bohemoslovaca.