Kainic acid-induced seizure upregulates Na(+)/myo-inositol cotransporter mRNA in rat brain.

[1]  T. Yamashita,et al.  Induction of Na+/myo-inositol co-transporter mRNA after rat cryogenic injury. , 1997, Brain research. Molecular brain research.

[2]  J. Willoughby,et al.  Fos Induction Following Systemic Kainic Acid: Early Expression in Hippocampus and Later Widespread Expression Correlated With Seizure , 1997, Neuroscience.

[3]  J. L. Stringer,et al.  Effect of seizures and diuretics on the osmolality of the cerebrospinal fluid , 1997, Brain Research.

[4]  T. Yamashita,et al.  Induction of Na+/Myo-Inositol Cotransporter mRNA after Focal Cerebral Ischemia: Evidence for Extensive Osmotic Stress in Remote Areas , 1996, Journal of Cerebral Blood Flow and Metabolism.

[5]  M. Tohyama,et al.  Rapid and transient up-regulation of Na+/myo-inositol cotransporter transcription in the brain of acute hypernatremic rats. , 1996, Brain research. Molecular brain research.

[6]  R. Belmaker,et al.  A comparison of the ability of myo-inositol and epi-inositol to attenuate lithium-plocarpine seizures in rats , 1996, Biological Psychiatry.

[7]  W. Lowe,et al.  Osmotic regulation of Na-myo-inositol cotransporter mRNA level and activity in endothelial and neural cells. , 1996, The American journal of physiology.

[8]  G. Holmes,et al.  Effect of kainic acid-induced status epilepticus on inositol-trisphosphate and seizure-induced brain damage in mature and immature animals. , 1995, Brain research. Developmental brain research.

[9]  M. Tohyama,et al.  Localization and rapid regulation of Na+/myo-inositol cotransporter in rat kidney. , 1995, The Journal of clinical investigation.

[10]  R. Jope,et al.  Modulation by inositol of cholinergic-and serotonergic-induced seizures in lithium-treated rats , 1995, Brain Research.

[11]  A. Planas,et al.  Regional expression of inducible heat shock protein-70 mRNA in the rat brain following administration of convulsant drugs. , 1994, Brain research. Molecular brain research.

[12]  T. Noguchi,et al.  In vivo osmoregulation of Na/myo-inositol cotransporter mRNA in rat kidney medulla. , 1994, Journal of the American Society of Nephrology : JASN.

[13]  P. Gass,et al.  High induction threshold for transcription factor KROX-20 in the rat brain: partial co-expression with heat shock protein 70 following limbic seizures. , 1994, Brain research. Molecular brain research.

[14]  J. L. Stringer,et al.  Kainic acid, bicuculline, pentylenetetrazol and pilocarpine elicit maximal dentate activation in the anesthetized rat , 1994, Epilepsy Research.

[15]  M. Baudry,et al.  Co-expression of HSP72 and c-fos in rat brain following kainic acid treatment. , 1993, Neuroreport.

[16]  P. Gass,et al.  Spatiotemporal Induction of Immediate Early Genes in the Rat Brain after Limbic Seizures: Effects of NMDA Receptor Antagonist MK‐801 , 1993, The European journal of neuroscience.

[17]  A. Yamauchi,et al.  Cloning of the cDNa for a Na+/myo-inositol cotransporter, a hypertonicity stress protein. , 1992, The Journal of biological chemistry.

[18]  S. Gullans,et al.  Upregulation of inositol transport mediates inositol accumulation in hyperosmolar brain cells. , 1991, The American journal of physiology.

[19]  M. Goldberg,et al.  Non-NMDA receptor-mediated neurotoxicity in cortical culture , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[20]  S. Gullans,et al.  Characterization of the major brain osmolytes that accumulate in salt-loaded rats. , 1989, The American journal of physiology.

[21]  F. Meyer Calcium, neuronal hyperexcitability and ischemic injury , 1989, Brain Research Reviews.

[22]  M. Burg,et al.  Signal for induction of aldose reductase in renal medullary cells by high external NaCl. , 1989, The American journal of physiology.

[23]  M. Rizzo,et al.  Blindsight: A case study and implications by L. Weiskrantz, Oxford University Press, 1986. £ 19.50 (viii + 187 pages) ISBN 9 18 852129 4 , 1987, Trends in Neurosciences.

[24]  P. Chomczyński,et al.  Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction. , 1987, Analytical biochemistry.

[25]  D. Choi Ionic dependence of glutamate neurotoxicity , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[26]  O. Hornykiewicz,et al.  Kainic acid-induced seizures: Dose-relationship of behavioural neurochemical and histopathological changes , 1985, Brain Research.

[27]  S. Rothman The neurotoxicity of excitatory amino acids is produced by passive chloride influx , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[28]  Y. Ben-Ari,et al.  Limbic seizure and brain damage produced by kainic acid: Mechanisms and relevance to human temporal lobe epilepsy , 1985, Neuroscience.

[29]  S. Kish,et al.  Kainic acid induced seizures: Neurochemical and histopathological changes , 1983, Neuroscience.

[30]  G. Westbrook,et al.  Cellular and synaptic basis of kainic acid-induced hippocampal epileptiform activity , 1983, Brain Research.

[31]  J. Wamsley,et al.  Autoradiographic localization of high-affinity [3H]kainic acid binding sites in the rat forebrain. , 1983, European journal of pharmacology.

[32]  C. Cotman,et al.  The distribution of [3H]kainic acid binding sites in rat CNS as determined by autoradiography , 1982, Brain Research.

[33]  M. E. Clark,et al.  Living with water stress: evolution of osmolyte systems. , 1982, Science.

[34]  S. Deadwyler,et al.  Kainic acid produces depolarization of CA3 pyramidal cells in the in vitro hippocampal slice , 1981, Brain Research.

[35]  R. C. Collins,et al.  Kainic acid induced limbic seizures: metabolic, behavioral, electroencephalographic and neuropathological correlates , 1981, Brain Research.

[36]  Y. Ben-Ari,et al.  Electrographic, clinical and pathological alterations following systemic administration of kainic acid, bicuculline or pentetrazole: Metabolic mapping using the deoxyglucose method with special reference to the pathology of epilepsy , 1981, Neuroscience.

[37]  J. Price,et al.  Widespread patterns of neuronal damage following systemic or intracerebral injections of kainic acid: A histological study , 1980, Neuroscience.

[38]  Y. Tano,et al.  Expression of Na+/myo-inositol cotransporter mRNA in normal and hypertonic stress rat eyes. , 1996, Brain research. Molecular brain research.

[39]  W. Lowe,et al.  Erratum: Osmotic regulation of Na-myo-inositol cotransporter mRNA level and activity in endothelial and neural cells (American Journal of Cell Physiology (1996) 270 (C990-C997)) , 1996 .

[40]  E. Imai,et al.  Na+/myo-inositol transport is regulated by basolateral tonicity in Madin-Darby canine kidney cells. , 1996, The Journal of clinical investigation.

[41]  A. Yamauchi,et al.  Hypertonicity stimulates transcription of gene for Na(+)-myo-inositol cotransporter in MDCK cells. , 1993, The American journal of physiology.

[42]  S. Gullans,et al.  Control of brain volume during hyperosmolar and hypoosmolar conditions. , 1993, Annual review of medicine.

[43]  J. Coyle,et al.  Developmental time course and ionic dependence of kainate-mediated toxicity in rat cerebellar granule cell cultures. , 1991, The Journal of pharmacology and experimental therapeutics.

[44]  J. Olney Inciting excitotoxic cytocide among central neurons. , 1986, Advances in experimental medicine and biology.

[45]  C. Cotman,et al.  Synaptic localization of kainic acid binding sites , 1981, Nature.