Inflammation induced by infection potentiates tau pathological features in transgenic mice.
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Thomas E Lane | F. LaFerla | M. Kitazawa | T. Lane | Masashi Kitazawa | R. Medeiros | David Cheng | Michael Sy | Rodrigo Medeiros | Michael Sy | Lucia Whitman | David Cheng | Frank M Laferla | L. Whitman
[1] M. Wozniak,et al. Alzheimer's disease and infection: Do infectious agents contribute to progression of Alzheimer's disease? , 2009, Alzheimer's & Dementia.
[2] M. Tsolaki,et al. Relationship between Helicobacter pylori infection and Alzheimer disease , 2006, Neurology.
[3] V. Thulasiraman,et al. Use of an antibody-coupled array and mass spectrometry for simultaneous detection of multiple amyloid beta fragments , 2009, Alzheimer's & Dementia.
[4] F. LaFerla,et al. Genetically augmenting tau levels does not modulate the onset or progression of Aβ pathology in transgenic mice , 2007, Journal of neurochemistry.
[5] D. Morgan,et al. Microglial Activation is Required for Aβ Clearance After Intracranial Injection of Lipopolysaccharide in APP Transgenic Mice , 2007, Journal of Neuroimmune Pharmacology.
[6] M. Wozniak,et al. Herpes simplex virus infection causes cellular β-amyloid accumulation and secretase upregulation , 2007, Neuroscience Letters.
[7] D. Morgan,et al. Time-dependent reduction in Aβ levels after intracranial LPS administration in APP transgenic mice , 2004, Experimental Neurology.
[8] R. Maccioni,et al. Interleukin-6 induces Alzheimer-type phosphorylation of tau protein by deregulating the cdk5/p35 pathway. , 2004, Experimental cell research.
[9] Khadija Iqbal,et al. Abnormal phosphorylation of tau and the mechanism of Alzheimer neurofibrillary degeneration: sequestration of microtubule-associated proteins 1 and 2 and the disassembly of microtubules by the abnormal tau. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[10] F. LaFerla,et al. Lipopolysaccharide-Induced Inflammation Exacerbates Tau Pathology by a Cyclin-Dependent Kinase 5-Mediated Pathway in a Transgenic Model of Alzheimer's Disease , 2005, The Journal of Neuroscience.
[11] J. Alonso,et al. Lithium blocks the PKB and GSK3 dephosphorylation induced by ceramide through protein phosphatase-2A. , 2002, Cellular signalling.
[12] A. Takashima,et al. GSK-3β Is Required for Memory Reconsolidation in Adult Brain , 2008, PloS one.
[13] P. Cohen,et al. Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B , 1995, Nature.
[14] C. Schaumburg,et al. T Cell Antiviral Effector Function Is Not Dependent on CXCL10 Following Murine Coronavirus Infection1 , 2006, The Journal of Immunology.
[15] N. Robakis,et al. It May Take Inflammation, Phosphorylation and Ubiquitination to ‘Tangle’ in Alzheimer’s Disease , 2007, Neurodegenerative Diseases.
[16] D. Appelt,et al. Chlamydia pneumoniae induces Alzheimer-like amyloid plaques in brains of BALB/c mice , 2004, Neurobiology of Aging.
[17] F. LaFerla,et al. Lithium reduces tau phosphorylation but not A beta or working memory deficits in a transgenic model with both plaques and tangles. , 2007, The American journal of pathology.
[18] M. Gurney,et al. Tau Phosphorylation at Serine 396 and Serine 404 by Human Recombinant Tau Protein Kinase II Inhibits Tau's Ability to Promote Microtubule Assembly* , 2000, The Journal of Biological Chemistry.
[19] S. Barger,et al. Interleukin-1 Mediates Pathological Effects of Microglia on Tau Phosphorylation and on Synaptophysin Synthesis in Cortical Neurons through a p38-MAPK Pathway , 2003, The Journal of Neuroscience.
[20] M. Buchmeier,et al. Dynamic regulation of alpha- and beta-chemokine expression in the central nervous system during mouse hepatitis virus-induced demyelinating disease. , 1998, Journal of immunology.
[21] E. Mandelkow,et al. Clogging of axons by tau, inhibition of axonal traffic and starvation of synapses , 2003, Neurobiology of Aging.
[22] J. Rogers,et al. Inflammation, anti-inflammatory agents and Alzheimer disease: the last 12 years. , 2006, Journal of Alzheimer's disease : JAD.
[23] Bin Zhang,et al. Synapse Loss and Microglial Activation Precede Tangles in a P301S Tauopathy Mouse Model , 2007, Neuron.
[24] Jae Woong Lee,et al. Journal of Neuroinflammation Neuro-inflammation Induced by Lipopolysaccharide Causes Cognitive Impairment through Enhancement of Beta-amyloid Generation , 2022 .
[25] F. LaFerla,et al. Inhibition of soluble TNF signaling in a mouse model of Alzheimer's disease prevents pre-plaque amyloid-associated neuropathology , 2009, Neurobiology of Disease.
[26] H. Schätzl,et al. Lithium induces clearance of protease resistant prion protein in prion‐infected cells by induction of autophagy , 2009, Journal of neurochemistry.
[27] S. Edland,et al. in Alzheimer's Disease , 1994 .
[28] Lili Wang,et al. Interplay between Cyclin-Dependent Kinase 5 and Glycogen Synthase Kinase 3β Mediated by Neuregulin Signaling Leads to Differential Effects on Tau Phosphorylation and Amyloid Precursor Protein Processing , 2008, The Journal of Neuroscience.
[29] S. Kikuchi,et al. Relationship between Helicobacter pylori infection and smoking and drinking habits , 2000, Journal of gastroenterology and hepatology.
[30] G. Cole,et al. Scavenging of Alzheimer's amyloid β‐protein by microglia in culture , 1996, Journal of neuroscience research.
[31] Y. L. Dubreuil,et al. Inflammatory processes induce beta-amyloid precursor protein changes in mouse brain. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[32] Q. Tian,et al. Temporal correlation of the memory deficit with Alzheimer‐like lesions induced by activation of glycogen synthase kinase‐3 , 2008, Journal of neurochemistry.
[33] H. Keirstead,et al. Remyelination, axonal sparing, and locomotor recovery following transplantation of glial-committed progenitor cells into the MHV model of multiple sclerosis , 2004, Experimental Neurology.
[34] W. Griffin,et al. Glial‐Neuronal Interactions in Alzheimer's Disease: The Potential Role of a ‘Cytokine Cycle’ in Disease Progression , 1998, Brain pathology.
[35] Gabriele Siciliano,et al. Lithium delays progression of amyotrophic lateral sclerosis , 2008, Proceedings of the National Academy of Sciences.
[36] P. Mcgeer,et al. Granules in glial cells of patients with Alzheimer's disease are immunopositive for C-terminal sequences of β-amyloid protein , 1996, Neuroscience Letters.
[37] J. Fleming,et al. Demyelination induced by murine hepatitis virus JHM strain (MHV-4) is immunologically mediated , 1990, Journal of Neuroimmunology.
[38] John D Lambris,et al. Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[39] F. LaFerla,et al. Inflammation induces tau pathology in inclusion body myositis model via glycogen synthase kinase‐3β , 2008, Annals of neurology.
[40] N. Ip,et al. Cdk5 is involved in neuregulin-induced AChR expression at the neuromuscular junction , 2001, Nature Neuroscience.
[41] F. Plattner,et al. The Roles of Cyclin-dependent Kinase 5 and Glycogen Synthase Kinase 3 in Tau Hyperphosphorylation* , 2006, Journal of Biological Chemistry.
[42] D. Borchelt,et al. Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid β peptide in APPswe transgenic mice , 2003, Neurobiology of Disease.
[43] D. Selkoe,et al. Relationship of microglia and astrocytes to amyloid deposits of Alzheimer disease , 1989, Journal of Neuroimmunology.
[44] D. Appelt,et al. Identification and localization of Chlamydia pneumoniae in the Alzheimer's brain , 1998, Medical Microbiology and Immunology.
[45] D. Selkoe. Toward a remembrance of things past: deciphering Alzheimer disease. , 2003, Harvey lectures.
[46] R. Edwards,et al. Evidence for Differential Roles for NKG2D Receptor Signaling in Innate Host Defense against Coronavirus-Induced Neurological and Liver Disease , 2007, Journal of Virology.
[47] L. Lue,et al. Microglial chemotaxis, activation, and phagocytosis of amyloid β-peptide as linked phenomena in Alzheimer's disease , 2001, Neurochemistry International.
[48] S. Maeda,et al. Tau oligomerization: a role for tau aggregation intermediates linked to neurodegeneration. , 2008, Current Alzheimer research.
[49] E. Mandelkow,et al. Sequential phosphorylation of Tau by glycogen synthase kinase-3beta and protein kinase A at Thr212 and Ser214 generates the Alzheimer-specific epitope of antibody AT100 and requires a paired-helical-filament-like conformation. , 1998, European journal of biochemistry.
[50] K. Honjo,et al. Commentary on the letter for Alzheimer's disease and infection: Do infectious agents contribute to progression of Alzheimer's disease? , 2010, Alzheimer's & Dementia.
[51] K. Honjo,et al. Alzheimer's disease and infection: Do infectious agents contribute to progression of Alzheimer's disease? , 2009, Alzheimer's & Dementia.
[52] M. Mattson,et al. Adverse Stress, Hippocampal Networks, and Alzheimer’s Disease , 2010, NeuroMolecular Medicine.
[53] D. Rubinsztein,et al. Lithium induces autophagy by inhibiting inositol monophosphatase , 2005, The Journal of cell biology.
[54] C. Holmes,et al. Role of Infection in the Pathogenesis of Alzheimer’s Disease , 2009, CNS drugs.
[55] M. Wozniak,et al. Herpes simplex virus type 1 in Alzheimer's disease: the enemy within , 2009, BDJ.
[56] C. Tzourio,et al. Vascular risk factors and dementia , 2009, Neurology.
[57] Tony Wyss-Coray,et al. Inflammation in Alzheimer disease: driving force, bystander or beneficial response? , 2006, Nature Medicine.
[58] C. Geula,et al. Ccr2 deficiency impairs microglial accumulation and accelerates progression of Alzheimer-like disease , 2007, Nature Medicine.
[59] D. Ruano,et al. Inflammatory Response in the Hippocampus of PS1M146L/APP751SL Mouse Model of Alzheimer's Disease: Age-Dependent Switch in the Microglial Phenotype from Alternative to Classic , 2008, The Journal of Neuroscience.