The Mechanism of Ca2+-Dependent Regulation of Kinesin-Mediated Mitochondrial Motility
暂无分享,去创建一个
[1] L. Hsieh‐Wilson,et al. The chemical neurobiology of carbohydrates. , 2008, Chemical reviews.
[2] Cuiling Li,et al. Docking of Axonal Mitochondria by Syntaphilin Controls Their Mobility and Affects Short-Term Facilitation , 2008, Cell.
[3] I. Boldogh,et al. Mitochondria on the move. , 2007, Trends in cell biology.
[4] S. Gross,et al. Cargo Transport: Two Motors Are Sometimes Better Than One , 2007, Current Biology.
[5] J. Swanson,et al. Kinesin-1 structural organization and conformational changes revealed by FRET stoichiometry in live cells , 2007, The Journal of cell biology.
[6] M. Beal. Mitochondria and neurodegeneration. , 2007, Novartis Foundation symposium.
[7] Dawen Cai,et al. Microtubule Acetylation Promotes Kinesin-1 Binding and Transport , 2006, Current Biology.
[8] K. Pozo,et al. Mapping the GRIF-1 Binding Domain of the Kinesin, KIF5C, Substantiates a Role for GRIF-1 as an Adaptor Protein in the Anterograde Trafficking of Cargoes* , 2006, Journal of Biological Chemistry.
[9] A. Spradling,et al. Milton controls the early acquisition of mitochondria by Drosophila oocytes , 2006, Development.
[10] D. Chan,et al. Critical dependence of neurons on mitochondrial dynamics. , 2006, Current opinion in cell biology.
[11] I. Reynolds,et al. Mitochondrial Trafficking to Synapses in Cultured Primary Cortical Neurons , 2006, The Journal of Neuroscience.
[12] A. Ruusala,et al. The atypical Rho GTPases Miro-1 and Miro-2 have essential roles in mitochondrial trafficking. , 2006, Biochemical and biophysical research communications.
[13] T. Schwarz,et al. Axonal transport of mitochondria requires milton to recruit kinesin heavy chain and is light chain independent , 2006, The Journal of cell biology.
[14] R. Rizzuto,et al. Mitochondrial dynamics and Ca2+ signaling. , 2006, Biochimica et biophysica acta.
[15] C. Lively,et al. Kinesin-1 and Dynein are the primary motors for fast transport of mitochondria in Drosophila motor axons. , 2006, Molecular biology of the cell.
[16] Tony Pawson,et al. Modification of the Creator recombination system for proteomics applications – improved expression by addition of splice sites , 2006, BMC biotechnology.
[17] G. Woehlke,et al. Review: regulation mechanisms of Kinesin-1 , 2006, Journal of Muscle Research & Cell Motility.
[18] M. Charlton,et al. The GTPase dMiro Is Required for Axonal Transport of Mitochondria to Drosophila Synapses , 2005, Neuron.
[19] P. Verstreken,et al. Synaptic Mitochondria Are Critical for Mobilization of Reserve Pool Vesicles at Drosophila Neuromuscular Junctions , 2005, Neuron.
[20] F. Stephenson,et al. GRIF-1 and OIP106, Members of a Novel Gene Family of Coiled-Coil Domain Proteins , 2005, Journal of Biological Chemistry.
[21] Yasunori Hayashi,et al. The Importance of Dendritic Mitochondria in the Morphogenesis and Plasticity of Spines and Synapses , 2004, Cell.
[22] S. Chasserot-Golaz,et al. Coupling actin and membrane dynamics during calcium-regulated exocytosis: a role for Rho and ARF GTPases. , 2004, Biochimica et biophysica acta.
[23] G. Hajnóczky,et al. Control of mitochondrial motility and distribution by the calcium signal , 2004, The Journal of cell biology.
[24] Rebecca L. Frederick,et al. Yeast Miro GTPase, Gem1p, regulates mitochondrial morphology via a novel pathway , 2004, The Journal of cell biology.
[25] P. Hollenbeck,et al. Nerve Growth Factor Signaling Regulates Motility and Docking of Axonal Mitochondria , 2004, Current Biology.
[26] R. Fletterick,et al. Crystal Structure of Kinesin Regulated by Ca2+-Calmodulin* , 2004, Journal of Biological Chemistry.
[27] J. Saras,et al. Rho GTPases have diverse effects on the organization of the actin filament system. , 2004, The Biochemical journal.
[28] W. Chazin,et al. Structures of EF-hand Ca 2+-binding proteins: Diversity in the organization, packing and response to Ca 2+ Binding , 1998, Biometals.
[29] G. Kress,et al. Glutamate Decreases Mitochondrial Size and Movement in Primary Forebrain Neurons , 2003, The Journal of Neuroscience.
[30] G. Hart,et al. Roles of the Tetratricopeptide Repeat Domain in O-GlcNAc Transferase Targeting and Protein Substrate Specificity* , 2003, Journal of Biological Chemistry.
[31] P. Hollenbeck,et al. Mitochondrial movement and positioning in axons: the role of growth factor signaling , 2003, Journal of Experimental Biology.
[32] A. Ruusala,et al. Atypical Rho GTPases Have Roles in Mitochondrial Homeostasis and Apoptosis* , 2003, The Journal of Biological Chemistry.
[33] I. Meinertzhagen,et al. Axonal Transport of Mitochondria to Synapses Depends on Milton, a Novel Drosophila Protein , 2002, Neuron.
[34] Seema Sharma,et al. Identification, Molecular Cloning, and Characterization of a Novel GABAA Receptor-associated Protein, GRIF-1* , 2002, The Journal of Biological Chemistry.
[35] K. Homma,et al. Ca2+-dependent Regulation of the Motor Activity of Myosin V* , 2000, The Journal of Biological Chemistry.
[36] D. Steele,et al. Effects of cytosolic ATP on spontaneous and triggered Ca2+‐induced Ca2+ release in permeabilised rat ventricular myocytes , 2000, The Journal of physiology.
[37] T. Rapoport,et al. Light Chain– dependent Regulation of Kinesin's Interaction with Microtubules , 1998, Journal of Cell Biology.
[38] N. Hirokawa,et al. Targeted Disruption of Mouse Conventional Kinesin Heavy Chain kif5B, Results in Abnormal Perinuclear Clustering of Mitochondria , 1998, Cell.
[39] A. Reddy,et al. Ca2+/calmodulin regulation of the Arabidopsis kinesin-like calmodulin-binding protein. , 1998, Cell motility and the cytoskeleton.
[40] W. Saxton,et al. Kinesin mutations cause motor neuron disease phenotypes by disrupting fast axonal transport in Drosophila. , 1996, Genetics.
[41] C. Overly,et al. Organelle motility and metabolism in axons vs dendrites of cultured hippocampal neurons. , 1996, Journal of cell science.
[42] P. Hollenbeck,et al. Axonal transport of mitochondria along microtubules and F-actin in living vertebrate neurons , 1995, The Journal of cell biology.
[43] T. Stossel,et al. Gelsolin inhibition of fast axonal transport indicates a requirement for actin microfilaments , 1984, Nature.