Involvement of STAT3-regulated hepatic soluble factors in attenuation of stellate cell activity and liver fibrogenesis in mice.
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N. Hayashi | N. Hiramatsu | A. Hosui | T. Kodama | M. Shigekawa | H. Hikita | T. Tatsumi | T. Takehara | T. Kanto | S. Shimizu | T. Miyagi | H. Ishida | Wei Li | Satoshi Shimizu | Minoru Shigekawa | Hayato Hikita
[1] N. Hayashi,et al. The Bcl‐xL inhibitor, ABT‐737, efficiently induces apoptosis and suppresses growth of hepatoma cells in combination with sorafenib , 2010, Hepatology.
[2] J. Magarian Blander,et al. Hepatic acute-phase proteins control innate immune responses during infection by promoting myeloid-derived suppressor cell function , 2010, The Journal of experimental medicine.
[3] E. Casanova,et al. Signal transducer and activator of transcription 3 protects from liver injury and fibrosis in a mouse model of sclerosing cholangitis. , 2010, Gastroenterology.
[4] N. Hayashi,et al. Thrombocytopenia exacerbates cholestasis-induced liver fibrosis in mice. , 2010, Gastroenterology.
[5] Darjus F. Tschaharganeh,et al. Lack of glycoprotein 130/signal transducer and activator of transcription 3-mediated signaling in hepatocytes enhances chronic liver injury and fibrosis progression in a model of sclerosing cholangitis. , 2010, The American journal of pathology.
[6] S. Akira,et al. Signal transducer and activator of transcription 3 signaling within hepatocytes attenuates systemic inflammatory response and lethality in septic mice , 2007, Hepatology.
[7] C. Trautwein,et al. Signal transducer of inflammation gp130 modulates atherosclerosis in mice and man , 2007, The Journal of experimental medicine.
[8] M. Manns,et al. Erratum: Lack of gp130 expression results in more bacterial infection and higher mortality during chronic cholestasis in mice (Hepatology (2005) 42 (1082-1090)) , 2005 .
[9] M. Manns,et al. Lack of gp130 expression results in more bacterial infection and higher mortality during chronic cholestasis in mice , 2005, Hepatology.
[10] Takahiro Suzuki,et al. Hepatocyte-specific disruption of Bcl-xL leads to continuous hepatocyte apoptosis and liver fibrotic responses. , 2004, Gastroenterology.
[11] Y. Kido,et al. Role of STAT-3 in regulation of hepatic gluconeogenic genes and carbohydrate metabolism in vivo , 2004, Nature Medicine.
[12] G. Gores,et al. Kupffer cell engulfment of apoptotic bodies stimulates death ligand and cytokine expression , 2003, Hepatology.
[13] S. Akira,et al. Stat3 protects against Fas-induced liver injury by redox-dependent and -independent mechanisms. , 2003, The Journal of clinical investigation.
[14] G. Gores,et al. Apoptotic Body Engulfment by a Human Stellate Cell Line Is Profibrogenic , 2003, Laboratory Investigation.
[15] Wei Li,et al. STAT3 Contributes to the Mitogenic Response of Hepatocytes during Liver Regeneration* , 2002, The Journal of Biological Chemistry.
[16] I. Kushner,et al. Acute-phase proteins and other systemic responses to inflammation. , 1999, The New England journal of medicine.
[17] S. Akira,et al. Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[18] T. Kishimoto,et al. Gp130 and the interleukin-6 family of cytokines. , 1997, Annual review of immunology.
[19] S. Akira,et al. Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway , 1994, Cell.
[20] J. Kountouras,et al. Prolonged bile duct obstruction: a new experimental model for cirrhosis in the rat. , 1984, British journal of experimental pathology.