Organ Damage

[1]  D. Rizzoni,et al.  Hemodynamic Consequences of Changes in Microvascular Structure , 2017, American journal of hypertension.

[2]  P. Gosse,et al.  High Prevalence of Multiple Arterial Bed Lesions in Patients With Fibromuscular Dysplasia: The ARCADIA Registry (Assessment of Renal and Cervical Artery Dysplasia) , 2017, Hypertension.

[3]  D. Rizzoni,et al.  Microvascular structure as a prognostically relevant endpoint. , 2017, Journal of hypertension.

[4]  P. Ambrosino,et al.  Markers of atherosclerosis in patients with Cushing’s syndrome: a meta-analysis of literature studies , 2017, Annals of medicine.

[5]  R. Netea-Maier,et al.  Vascular Health in Patients in Remission of Cushing's Syndrome Is Comparable With That in BMI-Matched Controls. , 2016, The Journal of clinical endocrinology and metabolism.

[6]  S. Y-Hassan Clinical Features and Outcome of Pheochromocytoma-Induced Takotsubo Syndrome: Analysis of 80 Published Cases. , 2016, The American journal of cardiology.

[7]  M. Robson,et al.  Pheochromocytoma Is Characterized by Catecholamine-Mediated Myocarditis, Focal and Diffuse Myocardial Fibrosis, and Myocardial Dysfunction. , 2016, Journal of the American College of Cardiology.

[8]  M. Muiesan,et al.  Atrial fibrillation and arterial hypertension: A common duet with dangerous consequences where the renin angiotensin-aldosterone system plays an important role. , 2016, International journal of cardiology.

[9]  C. Hung,et al.  Hypokalemia correlated with arterial stiffness but not microvascular endothelial function in patients with primary aldosteronism , 2015, Journal of the renin-angiotensin-aldosterone system : JRAAS.

[10]  P. Chanson,et al.  Cardiac Structure and Function in Cushing's Syndrome: A Cardiac Magnetic Resonance Imaging Study , 2014, The Journal of clinical endocrinology and metabolism.

[11]  F. Turchi,et al.  Primary aldosteronism and essential hypertension: assessment of cardiovascular risk at diagnosis and after treatment. , 2014, Nutrition, metabolism, and cardiovascular diseases : NMCD.

[12]  P. Mark,et al.  Alterations in Vascular Function in Primary Aldosteronism – a Cardiovascular Magnetic Resonance Imaging Study Running Title: Vascular Function in Primary Aldosteronism , 2013 .

[13]  C. Catena,et al.  Predictive Factors of Left Ventricular Mass Changes after Treatment of Primary Aldosteronism , 2012, Hormone and Metabolic Research.

[14]  T. Zelinka,et al.  Peripheral arterial stiffness in primary aldosteronism. , 2012, Physiological research.

[15]  Yen-Hung Lin,et al.  Endothelial progenitor cells in primary aldosteronism: a biomarker of severity for aldosterone vasculopathy and prognosis. , 2011, The Journal of clinical endocrinology and metabolism.

[16]  Christodoulos Stefanadis,et al.  Prediction of cardiovascular events and all-cause mortality with central haemodynamics: a systematic review and meta-analysis. , 2010, European heart journal.

[17]  T. Zelinka,et al.  Factors influencing arterial stiffness in pheochromocytoma and effect of adrenalectomy , 2010, Hypertension Research.

[18]  Jeroen J. Bax,et al.  Cardiac dysfunction is reversed upon successful treatment of Cushing's syndrome. , 2010, European journal of endocrinology.

[19]  C. de Ciuceis,et al.  Hypertrophic remodeling of subcutaneous small resistance arteries in patients with Cushing's syndrome. , 2009, The Journal of clinical endocrinology and metabolism.

[20]  D. Rizzoni,et al.  Effects of antihypertensive treatment on small artery remodelling , 2009, Journal of hypertension.

[21]  T. Zelinka,et al.  Increased carotid intima-media thickness in patients with pheochromocytoma in comparison to essential hypertension , 2009, Journal of Human Hypertension.

[22]  M. Bazzocchi,et al.  Intrarenal hemodynamics in primary aldosteronism before and after treatment. , 2009, Journal of Clinical Endocrinology and Metabolism.

[23]  R. Holle,et al.  Risk factors associated with a low glomerular filtration rate in primary aldosteronism. , 2009, The Journal of clinical endocrinology and metabolism.

[24]  T. Zelinka,et al.  Adrenalectomy improves arterial stiffness in primary aldosteronism. , 2008, American journal of hypertension.

[25]  I. Kallikazaros,et al.  Myocardial and Aortic Stiffening in the Early Course of Primary Aldosteronism , 2008, Clinical cardiology.

[26]  L. Sechi,et al.  Cardiovascular outcomes in patients with primary aldosteronism after treatment. , 2008, Archives of internal medicine.

[27]  T. Zelinka,et al.  Increased intima–media thickness of the common carotid artery in primary aldosteronism in comparison with essential hypertension , 2007, Journal of hypertension.

[28]  T. Zelinka,et al.  Increased arterial wall stiffness in primary aldosteronism in comparison with essential hypertension. , 2006, American journal of hypertension.

[29]  M. Novello,et al.  Long-term renal outcomes in patients with primary aldosteronism. , 2006, JAMA.

[30]  M. Muiesan,et al.  Evaluation of subclinical target organ damage for risk assessment and treatment in the hypertensive patients: left ventricular hypertrophy. , 2006, Journal of the American Society of Nephrology : JASN.

[31]  C. de Ciuceis,et al.  Changes in extracellular matrix in subcutaneous small resistance arteries of patients with primary aldosteronism. , 2006, The Journal of clinical endocrinology and metabolism.

[32]  J. Sharman,et al.  Evidence for abnormal left ventricular structure and function in normotensive individuals with familial hyperaldosteronism type I. , 2005, The Journal of clinical endocrinology and metabolism.

[33]  P. Fesler,et al.  Relative glomerular hyperfiltration in primary aldosteronism. , 2005, Journal of the American Society of Nephrology : JASN.

[34]  P. Milliez,et al.  Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism. , 2005, Journal of the American College of Cardiology.

[35]  D. Calhoun,et al.  Impaired Endothelium-Dependent Flow-Mediated Vasodilation in Hypertensive Subjects With Hyperaldosteronism , 2004, Circulation.

[36]  E. Schiffrin The many targets of aldosterone. , 2004, Hypertension.

[37]  E. Porteri,et al.  Structural changes in small resistance arteries and left ventricular geometry in patients with primary and secondary hypertension , 2002, Journal of hypertension.

[38]  T. Yoshimoto,et al.  Left ventricular hypertrophy is more prominent in patients with primary aldosteronism than in patients with other types of secondary hypertension. , 1997, Hypertension research : official journal of the Japanese Society of Hypertension.

[39]  A. Pessina,et al.  Left ventricular anatomy and function in primary aldosteronism and renovascular hypertension. , 1997, Advances in experimental medicine and biology.

[40]  D. Rizzoni,et al.  Association of change in left ventricular mass with prognosis during long-term antihypertensive treatment , 1995, Journal of hypertension.

[41]  A. Pessina,et al.  Prevalence of extracranial carotid artery lesions at duplex in primary aldosteronism. , 1993, American journal of hypertension.

[42]  M. Mulvany,et al.  Structure and function of small arteries. , 1990, Physiological reviews.

[43]  K P Offord,et al.  Association of hypokalemia, aldosteronism, and renal cysts. , 1990, The New England journal of medicine.

[44]  N. Javadpour,et al.  Renal changes in primary aldosteronism. , 1977, Journal of Urology.