Adenylate kinase 1 gene deletion disrupts muscle energetic economy despite metabolic rearrangement
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Arend Heerschap | Edwin Janssen | Andre Terzic | A. W. Simonetti | A. de Haan | A. Terzic | A. Heerschap | P. Dzeja | Arnold de Haan | F. Oerlemans | Petras P. Dzeja | Frank Oerlemans | Arjan W. Simonetti | Paula S. Rush | Ronald R. Terjung | Bé Wieringa | B. Wieringa | Edwin Janssen | Paula S. Rush | Ronald R. Terjung | A. Simonetti
[1] A. Koretsky,et al. Insights into cellular energy metabolism from transgenic mice. , 1995, Physiological reviews.
[2] R. Terjung,et al. Adenine nucleotide metabolism in contracting skeletal muscle. , 1991, Exercise and sport sciences reviews.
[3] M. Wyss,et al. Intracellular compartmentation, structure and function of creatine kinase isoenzymes in tissues with high and fluctuating energy demands: the 'phosphocreatine circuit' for cellular energy homeostasis. , 1992, The Biochemical journal.
[4] J. Ingwall,et al. Impaired cardiac energetics in mice lacking muscle-specific isoenzymes of creatine kinase. , 1998, Circulation research.
[5] J. Ottaway,et al. The role of compartmentation in the control of glycolysis. , 1977, Current topics in cellular regulation.
[6] A. Terzic,et al. Reversal of the ATP-liganded State of ATP-sensitive K+ Channels by Adenylate Kinase Activity* , 1996, The Journal of Biological Chemistry.
[7] R. Terjung,et al. Molecular and kinetic alterations of muscle AMP deaminase during chronic creatine depletion. , 1998, American journal of physiology. Cell physiology.
[8] B. Wieringa,et al. Direct Evidence for the Control of Mitochondrial Respiration by Mitochondrial Creatine Kinase in Oxidative Muscle Cells in Situ * , 2000, The Journal of Biological Chemistry.
[9] N D Goldberg,et al. Kinetics and compartmentation of energy metabolism in intact skeletal muscle determined from 18O labeling of metabolite phosphoryls. , 1991, The Journal of biological chemistry.
[10] B. Wieringa,et al. Targeting of the creatine kinase M gene in embryonic stem cells using isogenic and nonisogenic vectors. , 1992, Nucleic acids research.
[11] P. Diolez,et al. Quantitative studies of enzyme-substrate compartmentation, functional coupling and metabolic channelling in muscle cells , 2004, Molecular and Cellular Biochemistry.
[12] D. Hardie,et al. The AMP-activated protein kinase--fuel gauge of the mammalian cell? , 1997, European journal of biochemistry.
[13] U. Oechsner,et al. Yeast adenylate kinase is active simultaneously in mitochondria and cytoplasm and is required for non-fermentative growth. , 1988, European journal of biochemistry.
[14] L. Olson,et al. Suppression of Adenylate Kinase Catalyzed Phosphotransfer Precedes and Is Associated with Glucose-induced Insulin Secretion in Intact HIT-T15 Cells* , 1996, The Journal of Biological Chemistry.
[15] M. Yamada,et al. Mechanism of mitochondrial import of adenylate kinase isozymes. , 1998, Journal of biochemistry.
[16] C. Gibbs,et al. Efficiency of skeletal and cardiac muscle. , 1998, Advances in Experimental Medicine and Biology.
[17] M. Inouye,et al. Adenylate kinase complements nucleoside diphosphate kinase deficiency in nucleotide metabolism. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[18] Honggao Yan,et al. Nucleoside monophosphate kinases: structure, mechanism, and substrate specificity. , 1999, Advances in enzymology and related areas of molecular biology.
[19] M. Yamada,et al. Tissue-specific and developmentally regulated expression of the genes encoding adenylate kinase isozymes. , 1993, Journal of biochemistry.
[20] Arend Heerschap,et al. Altered Ca2+ Responses in Muscles with Combined Mitochondrial and Cytosolic Creatine Kinase Deficiencies , 1997, Cell.
[21] B. Wieringa,et al. Alterations in AMP deaminase activity and kinetics in skeletal muscle of creatine kinase-deficient mice. , 1998, American journal of physiology. Cell physiology.
[22] S. Bessman,et al. The creatine-creatine phosphate energy shuttle. , 1985, Annual review of biochemistry.
[23] P. Dzeja,et al. Suppression of Creatine Kinase-catalyzed Phosphotransfer Results in Increased Phosphoryl Transfer by Adenylate Kinase in Intact Skeletal Muscle* , 1996, The Journal of Biological Chemistry.
[24] C. Mathews,et al. Nucleoside diphosphokinase: a functional link between intermediary metabolism and nucleic acid synthesis. , 1992, Current topics in cellular regulation.
[25] The AMP‐Activated Protein Kinase , 1997 .
[26] Philippe Mateo,et al. Muscle Creatine Kinase-deficient Mice , 1995, The Journal of Biological Chemistry.
[27] M. Yamada,et al. Human adenylate kinase deficiency associated with hemolytic anemia. A single base substitution affecting solubility and catalytic activity of the cytosolic adenylate kinase. , 1989, Journal of Biological Chemistry.
[28] G. Schulz,et al. Structural relationships in the adenylate kinase family. , 1986, European journal of biochemistry.
[29] D R Wilkie,et al. Energetics of muscular contraction. , 1973, Clinical science.
[30] A. Terzic,et al. Phosphotransfer reactions in the regulation of ATP‐sensitive K+ channels , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[31] M. Konrad. Molecular analysis of the essential gene for adenylate kinase from the fission yeast Schizosaccharomyces pombe. , 1993, The Journal of biological chemistry.
[32] Arend Heerschap,et al. Skeletal muscles of mice deficient in muscle creatine kinase lack burst activity , 1993, Cell.
[33] Cytoarchitectural and metabolic adaptations in muscles with mitochondrial and cytosolic creatine kinase deficiencies , 1998 .
[34] S. Dawis,et al. Evidence for compartmentalized adenylate kinase catalysis serving a high energy phosphoryl transfer function in rat skeletal muscle. , 1990, The Journal of biological chemistry.
[35] A. Terzic,et al. Failing energetics in failing hearts , 2000, Current cardiology reports.
[36] P. Dzeja,et al. Adenylate Kinase-catalyzed Phosphoryl Transfer Couples ATP Utilization with Its Generation by Glycolysis in Intact Muscle (*) , 1995, The Journal of Biological Chemistry.
[37] D. Lazarević,et al. wt p53 dependent expression of a membrane-associated isoform of adenylate kinase , 1999, Oncogene.
[38] A. Terzic,et al. Adenylate kinase-catalyzed phosphotransfer in the myocardium : increased contribution in heart failure. , 1999, Circulation research.
[39] B. Wieringa,et al. Muscle Creatine Kinase-deficient Mice , 1995, The Journal of Biological Chemistry.