Developmental toxicity of dioxin to mouse embryonic teeth in vitro: arrest of tooth morphogenesis involves stimulation of apoptotic program in the dental epithelium.
暂无分享,去创建一个
I. Thesleff | R. Pohjanvirta | P. Lukinmaa | A. Partanen | C. Sahlberg | S. Alaluusua | A. Kiukkonen
[1] J. Tuomisto,et al. Response of the incisor tooth to 2,3,7,8-tetrachlorodibenzo-p-dioxin in a dioxin-resistant and a dioxin-sensitive rat strain. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[2] J. Tuomisto,et al. Effect of in utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on rat molar development: the role of exposure time. , 2002, Toxicology and applied pharmacology.
[3] Michael T. Fisher,et al. Analysis of 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced gene expression profile in vivo using pathway-specific cDNA arrays. , 2002, Toxicology.
[4] R. Peterson,et al. In utero exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin causes accelerated terminal differentiation in fetal mouse skin. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[5] L. Birnbaum,et al. Persistent abnormalities in the rat mammary gland following gestational and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[6] R. Pohjanvirta,et al. Expression of the mediators of dioxin toxicity, aryl hydrocarbon receptor (AHR) and the AHR nuclear translocator (ARNT), is developmentally regulated in mouse teeth. , 2002, The International journal of developmental biology.
[7] R. Peterson,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin toxicity in the zebrafish embryo: altered regional blood flow and impaired lower jaw development. , 2002, Toxicological sciences : an official journal of the Society of Toxicology.
[8] N. Maihle,et al. The EGF/ErbB Receptor Family and Apoptosis , 2002, Growth factors.
[9] M. Walker,et al. 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) inhibition of coronary development is preceded by a decrease in myocyte proliferation and an increase in cardiac apoptosis. , 2001, Teratology.
[10] J. Loertscher,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin alters the differentiation pattern of human keratinocytes in organotypic culture. , 2001, Toxicology and applied pharmacology.
[11] J. Tuukkanen,et al. In utero/lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure impairs molar tooth development in rats. , 2001, Toxicology and applied pharmacology.
[12] D. Tillitt,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin induces apoptotic cell death and cytochrome P4501A expression in developing Fundulus heteroclitus embryos. , 2001, Aquatic toxicology.
[13] R. Peterson,et al. In utero and lactational treatment with 2,3,7,8-tetrachlorodibenzo-p-dioxin impairs mammary gland differentiation but does not block the response to exogenous estrogen in the postpubertal female rat. , 2001, Toxicological sciences : an official journal of the Society of Toxicology.
[14] L. Leamy,et al. 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Affects Size and Shape, But Not Asymmetry, of Mandibles in Mice , 2001, Ecotoxicology.
[15] S. Kondo,et al. The immunohistochemical localization of Bax and Bcl-2 and their relation to apoptosis during amelogenesis in developing rat molars. , 2001, Archives of oral biology.
[16] R. Pohjanvirta,et al. Arrest of rat molar tooth development by lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 2001, Toxicology and applied pharmacology.
[17] Rachel L. Allen,et al. Defying death after DNA damage , 2000, Nature.
[18] M. Hengartner. The biochemistry of apoptosis , 2000, Nature.
[19] M. Walker,et al. Characterization of cardiotoxicity induced by 2,3,7, 8-tetrachlorodibenzo-p-dioxin and related chemicals during early chick embryo development. , 2000, Toxicology and applied pharmacology.
[20] J. Davis,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) inhibits growth factor withdrawal-induced apoptosis in the human mammary epithelial cell line, MCF-10A. , 2000, Carcinogenesis.
[21] I. Thesleff,et al. Reiterative signaling and patterning during mammalian tooth morphogenesis , 2000, Mechanisms of Development.
[22] M. DeVito,et al. Acute administration of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in pregnant Long Evans rats: association of measured tissue concentrations with developmental effects. , 2000, Toxicological sciences : an official journal of the Society of Toxicology.
[23] A. Ullrich,et al. Epidermal growth factor receptors: critical mediators of multiple receptor pathways. , 1999, Current opinion in cell biology.
[24] S. Korsmeyer,et al. Cell Death in Development , 1999, Cell.
[25] T. Vartiainen,et al. Developing teeth as biomarker of dioxin exposure , 1999, The Lancet.
[26] I. Thesleff,et al. Epidermal growth factor receptor as a mediator of developmental toxicity of dioxin in mouse embryonic teeth. , 1998, Laboratory investigation; a journal of technical methods and pathology.
[27] John Calvin Reed,et al. Developmental expression patterns of Bcl-2, Bcl-x, Bax, and Bak in Teeth , 1998, Cell Death and Differentiation.
[28] I. Thesleff,et al. Expression and function of FGFs‐4, ‐8, and ‐9 suggest functional redundancy and repetitive use as epithelial signals during tooth morphogenesis , 1998, Developmental dynamics : an official publication of the American Association of Anatomists.
[29] S. Keränen,et al. The life history of an embryonic signaling center: BMP-4 induces p21 and is associated with apoptosis in the mouse tooth enamel knot. , 1998, Development.
[30] J. Rubenstein,et al. Role of Dlx-1 and Dlx-2 genes in patterning of the murine dentition. , 1997, Development.
[31] R. Peterson,et al. In utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin: effects on development of the male and female reproductive system of the mouse. , 1997, Toxicology and applied pharmacology.
[32] M. Raff,et al. Programmed Cell Death in Animal Development , 1997, Cell.
[33] M. Nagarkatti,et al. Evidence for the induction of apoptosis in thymocytes by 2,3,7,8-tetrachlorodibenzo-p-dioxin in vivo. , 1997, Toxicology and applied pharmacology.
[34] D. Tillitt,et al. Embryotoxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): the embryonic vasculature is a physiological target for TCDD-induced DNA damage and apoptotic cell death in Medaka (Orizias latipes). , 1996, Toxicology and applied pharmacology.
[35] T. Vartiainen,et al. Polychlorinated dibenzo-p-dioxins and dibenzofurans via mother's milk may cause developmental defects in the child's teeth. , 1996, Environmental toxicology and pharmacology.
[36] R. Pohjanvirta,et al. Short-term toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in laboratory animals: effects, mechanisms, and animal models. , 1994, Pharmacological reviews.
[37] R. Peterson,et al. Partial demasculinization and feminization of sex behavior in male rats by in utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin is not associated with alterations in estrogen receptor binding or volumes of sexually differentiated brain nuclei. , 1994, Toxicology and applied pharmacology.
[38] R. Voutilainen,et al. EGF receptor and its ligands, EGF and TGF-alpha, in developing and neoplastic human odontogenic tissues. , 1993, The International journal of developmental biology.
[39] R. Pohjanvirta,et al. Exposure to 2,3,7,8-tetrachlorodibenzo-para-dioxin leads to defective dentin formation and pulpal perforation in rat incisor tooth. , 1993, Toxicology.
[40] L. Birnbaum,et al. TCDD alters medial epithelial cell differentiation during palatogenesis. , 1989, Toxicology and applied pharmacology.
[41] R. Michell. POST-RECEPTOR SIGNALLING PATHWAYS , 1989, The Lancet.
[42] M. Rao,et al. Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in the Syrian golden hamster. , 1988, Carcinogenesis.
[43] L. Birnbaum,et al. TCDD-induced hyperplasia of the ureteral epithelium produces hydronephrosis in murine fetuses. , 1987, Teratology.
[44] I. Thesleff,et al. Localization and quantitation of 125I-epidermal growth factor binding in mouse embryonic tooth and other embryonic tissues at different developmental stages. , 1987, Developmental biology.
[45] W. McNulty. Toxicity and fetotoxicity of TCDD, TCDF and PCB isomers in rhesus macaques (Macaca mulatta). , 1985, Environmental health perspectives.
[46] I. Thesleff,et al. Transferrin is required for early tooth morphogenesis. , 1984, Differentiation; research in biological diversity.
[47] C. Wade,et al. Results of a two-year chronic toxicity and oncogenicity study of 2,3,7,8-tetrachlorodibenzo-p-dioxin in rats. , 1978, Toxicology and applied pharmacology.
[48] J. Tuomisto,et al. In utero / lactational TCDD exposure impairs the molar tooth development in rats , 2000 .
[49] D. Nebert,et al. Role of the aromatic hydrocarbon receptor and [Ah] gene battery in the oxidative stress response, cell cycle control, and apoptosis. , 2000, Biochemical pharmacology.
[50] R. Peterson,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin alters cardiovascular and craniofacial development and function in sac fry of rainbow trout (Oncorhynchus mykiss). , 1999, Toxicological sciences : an official journal of the Society of Toxicology.
[51] J. Peters,et al. Amelioration of TCDD-induced teratogenesis in aryl hydrocarbon receptor (AhR)-null mice. , 1999, Toxicological sciences : an official journal of the Society of Toxicology.
[52] K. Weiss,et al. Perspectives on genetic aspects of dental patterning. , 1998, European journal of oral sciences.
[53] D. Tillitt,et al. Correlation of 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced apoptotic cell death in the embryonic vasculature with embryotoxicity. , 1998, Toxicology and applied pharmacology.
[54] A. Vaahtokari,et al. Apoptosis in the developing tooth: association with an embryonic signaling center and suppression by EGF and FGF-4. , 1996, Development.
[55] C. Bradfield,et al. Ah receptor signaling pathways. , 1996, Annual review of cell and developmental biology.
[56] S. Mima,et al. A morphological study of liver lesions in Xenopus larvae exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) with special reference to apoptosis of hepatocytes. , 1995, Journal of environmental pathology, toxicology and oncology : official organ of the International Society for Environmental Toxicology and Cancer.
[57] G L Kimmel,et al. Developmental and reproductive toxicity of dioxins and related compounds: cross-species comparisons. , 1993, Critical reviews in toxicology.
[58] H. Overström,et al. Polychlorinated dibenzo-p-dioxins and dibenzofurans. , 1991 .
[59] V. Diewert,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin-induced cleft palate in the mouse: evidence for alterations in palatal shelf fusion. , 1984, Teratogenesis, carcinogenesis, and mutagenesis.