Selective effects of ketamine on amino acid-mediated pathways in neonatal rat spinal cord.
暂无分享,去创建一个
[1] I. Módy,et al. The Role of the GABAA Receptor/Chloride Channel Complex in Anesthesia , 1993, Anesthesiology.
[2] E. Brunner,et al. Inducing Anesthesia with a GABA Analog, THIP , 1985, Anesthesiology.
[3] J. Dubois,et al. Mechanism of action of ketamine in the current and voltage clamped myelinated nerve fibre of the frog , 1986, British journal of pharmacology.
[4] R. Siarey,et al. Effect of 6‐cyano‐2,3‐dihydroxy‐7‐nitro‐quinoxaline (CNQX) on dorsal root‐, NMDA‐, kainate‐ and quisqualate‐mediated depolarization of rat motoneurones in vitro , 1990, British journal of pharmacology.
[5] S. Konishi,et al. A substance P antagonist inhibits a slow reflex response in the spinal cord of the newborn rat. , 1982, Acta physiologica Scandinavica.
[6] J. Kendig,et al. Substance P and NMDA receptor-mediated slow potentials in neonatal rat spinal cord: age-related changes , 1992, Brain Research.
[7] R. Wachtel. Ketamine Decreases the Open Time of Single‐channel Currents Activated by Acetylcholine , 1988, Anesthesiology.
[8] G. Biggio,et al. Biochemical and electrophysiologic evidence that propofol enhances GABAergic transmission in the rat brain. , 1991, Anesthesiology.
[9] W. J. Russell,et al. The action of ether and methoxyflurane on synaptic transmission in isolated preparations of the mammalian cortex. , 1975, The Journal of physiology.
[10] T. Narahashi,et al. General anesthetics modulate GABA receptor channel complex in rat dorsal root ganglion neurons , 1989, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[11] F. F. Weight,et al. GABA- and glutamate-gated ion channels as molecular sites of alcohol and anesthetic action. , 1992, Advances in biochemical psychopharmacology.
[12] K. Krnjević,et al. Halothane suppresses slow inward currents in hippocampal slices. , 1988, Canadian journal of physiology and pharmacology.
[13] J. Kendig,et al. Propofol and barbiturate depression of spinal nociceptive neurotransmission. , 1992, Anesthesiology.
[14] M. Maze,et al. Isoflurane and an α2-Adrenoceptor Agonist Suppress Nociceptive Neurotransmission in Neonatal Rat Spinal Cord , 1991 .
[15] V. Baum,et al. Distinctive Effects of Three Intravenous Anesthetics on the Inward Rectifier (IK1) and the Delayed Rectifier (IK) Potassium Currents in Myocardium: Implications for the Mechanism of Action , 1993, Anesthesia and analgesia.
[16] N. Akaike,et al. Effects of two volatile anesthetics and a volatile convulsant on the excitatory and inhibitory amino acid responses in dissociated CNS neurons of the rat. , 1991, Journal of neurophysiology.
[17] R. Dingledine. New wave of non-NMDA excitatory amino acid receptors. , 1991, Trends in pharmacological sciences.
[18] D. Lodge,et al. Effects of ketamine and three other anaesthetics on spinal reflexes and inhibitions in the cat. , 1984, British journal of anaesthesia.
[19] A. Malmberg,et al. Hyperalgesia mediated by spinal glutamate or substance P receptor blocked by spinal cyclooxygenase inhibition. , 1992, Science.
[20] W. R. Lieb,et al. Molecular and cellular mechanisms of general anaesthesia , 1994, Nature.
[21] G. D. Lange,et al. Potentiation of gamma‐aminobutyric‐acid‐activated chloride conductance by a steroid anaesthetic in cultured rat spinal neurones. , 1987, The Journal of physiology.
[22] W. L. Way,et al. On the pharmacology of the ketamine enantiomorphs in the rat. , 1977, The Journal of pharmacology and experimental therapeutics.
[23] Longtang L. Chen,et al. Enflurane inhibits NMDA, AMPA, and kainate‐induced currents in Xenopus oocytes expressing mouse and human brain mRNA , 1993, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[24] R. Nicoll,et al. Presynaptic inhibition: transmitter and ionic mechanisms. , 1979, International review of neurobiology.
[25] F. F. Weight,et al. Ethanol inhibits NMDA-activated current but does not alter GABA-activated current in an isolated adult mammalian neuron , 1990, Brain Research.
[26] Y. Ohno,et al. Selective depression of the segmental polysynaptic reflex by phencyclidine and its analogs in the rat in vitro: interaction with N-methyl-D-aspartate receptors. , 1990, The Journal of pharmacology and experimental therapeutics.
[27] C. Jahr,et al. Ia afferent excitation of motoneurones in the in vitro new‐born rat spinal cord is selectively antagonized by kynurenate. , 1986, The Journal of physiology.
[28] G. Collingridge,et al. Excitatory amino acid receptors in the vertebrate central nervous system. , 1989, Pharmacological reviews.
[29] R. Dingledine,et al. Molecular biology of mammalian amino acid receptors , 1990, The FASEB Journal.
[30] S. Long,et al. Primary afferent depolarization in the rat spinal cord is mediated by pathways utilising NMDA and non-NMDA receptors , 1989, Neuroscience Letters.
[31] C. Scholfield,et al. Action of general anaesthetics on unclamped Ca(2+)-mediated currents in unmyelinated axons of rat olfactory cortex. , 1991, European journal of pharmacology.
[32] R. M. Joy,et al. Modification of GABA-mediated inhibition by various injectable anesthetics. , 1992, Anesthesiology.
[33] N. Harrison,et al. Enhancement of gamma‐aminobutyric acid‐activated Cl‐ currents in cultured rat hippocampal neurones by three volatile anaesthetics. , 1992, The Journal of physiology.
[34] J. Kendig,et al. Isoflurane Depresses Both Glutamate‐ and Peptide‐Mediated Slow Synaptic Transmission in Neonatal Rat Spinal Cord , 1991, Annals of the New York Academy of Sciences.
[35] C. Scholfield,et al. General anaesthetics and field currents in unclamped, unmyelinated axons of rat olfactory cortex , 1990, British journal of pharmacology.
[36] C. D. Richards,et al. An in vitro investigation of the action of ketamine on excitatory synaptic transmission in the hippocampus of the guinea-pig. , 1988, European journal of pharmacology.
[37] M. Otsuka,et al. Pharmacological profile of a tachykinin antagonist, spantide, as examined on rat spinal motoneurones , 1990, British journal of pharmacology.
[38] J. Kendig,et al. Enflurane-induced burst discharge of hippocampal CA1 neurones is blocked by the NMDA receptor antagonist APV. , 1989, British journal of anaesthesia.
[39] C. D. Richards,et al. The actions of volatile anaesthetics on synaptic transmission in the dentate gyrus. , 1975, The Journal of physiology.
[40] J. Antognini,et al. Exaggerated Anesthetic Requirements in the Preferentially Anesthetized Brain , 1993, Anesthesiology.
[41] J. Kendig,et al. Anesthetic Actions in the Hippocampal Formation , 1991, Annals of the New York Academy of Sciences.
[42] J. Kendig,et al. Substance P and NMDA receptors mediate a slow nociceptive ventral root potential in neonatal rat spinal cord , 1991, Brain Research.
[43] J. Kendig,et al. The GABAA Receptor in Anesthesia: Isoflurane , 1994 .
[44] B. Robertson,et al. Prolongation of inhibitory postsynaptic currents by pentobarbitone, halothane and ketamine in CA1 pyramidal cells in rat hippocampus , 1985, British journal of pharmacology.
[45] G. Collingridge,et al. Ketamine blocks an NMDA receptor-mediated component of synaptic transmission in rat hippocampus in a voltage-dependent manner , 1988, Neuroscience Letters.
[46] M. Rogawski,et al. Phencyclidine block of calcium current in isolated guinea‐pig hippocampal neurones. , 1992, The Journal of physiology.
[47] R. H. Evans,et al. A comparison of excitatory amino acid antagonists acting at primary afferent C fibres and motoneurones of the isolated spinal cord of the rat , 1987, British journal of pharmacology.
[48] R. Harris,et al. General anesthetics potentiate gamma-aminobutyric acid actions on gamma-aminobutyric acidA receptors expressed by Xenopus oocytes: lack of involvement of intracellular calcium. , 1992, The Journal of pharmacology and experimental therapeutics.
[49] F. Moroni,et al. General anaesthetics inhibit the responses induced by glutamate receptor agonists in the mouse cortex , 1992, Neuroscience Letters.
[50] W. R. Lieb,et al. Selective actions of volatile general anaesthetics at molecular and cellular levels. , 1993, British journal of anaesthesia.