c‐FLIPL is a dual function regulator for caspase‐8 activation and CD95‐mediated apoptosis
暂无分享,去创建一个
Xiaolu Yang | M. Peter | Xiaolu Yang | A. Algeciras-Schimnich | B. Barnhart | David W. Chang | Zheng Xing | Yi Pan | Alicia Algeciras‐Schimnich | Bryan C. Barnhart | Shoshanit Yaish‐Ohad | Marcus E. Peter | D. W. Chang | S. Yaish-Ohad | Yi Pan | Zheng Xing
[1] D. Baltimore,et al. Autoproteolytic activation of pro-caspases by oligomerization. , 1998, Molecular cell.
[2] P. Krammer,et al. TCR-Mediated Up-Regulation of c-FLIPshort Correlates with Resistance Toward CD95-Mediated Apoptosis by Blocking Death-Inducing Signaling Complex Activity1 , 2000, The Journal of Immunology.
[3] M. Peter,et al. The death receptors. , 1999, Results and problems in cell differentiation.
[4] H. Perlman,et al. Flice-Inhibitory Protein Expression during Macrophage Differentiation Confers Resistance to FAS-Mediated Apoptosis , 1999, The Journal of experimental medicine.
[5] S. Schreiber,et al. Identification of an 11-kDa FKBP12-rapamycin-binding domain within the 289-kDa FKBP12-rapamycin-associated protein and characterization of a critical serine residue. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[6] Brent R. Stockwell,et al. An Induced Proximity Model for Caspase-8 Activation* , 1998, The Journal of Biological Chemistry.
[7] R. Siegel,et al. Membrane Oligomerization and Cleavage Activates the Caspase-8 (FLICE/MACHα1) Death Signal* , 1998, The Journal of Biological Chemistry.
[8] B. Trask,et al. MRIT, a novel death-effector domain-containing protein, interacts with caspases and BclXL and initiates cell death. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[9] M. Peter,et al. Cytotoxicity‐dependent APO‐1 (Fas/CD95)‐associated proteins form a death‐inducing signaling complex (DISC) with the receptor. , 1995, The EMBO journal.
[10] J. Blenis,et al. Death Receptor Recruitment of Endogenous Caspase-10 and Apoptosis Initiation in the Absence of Caspase-8* , 2001, The Journal of Biological Chemistry.
[11] M. Peter,et al. Two CD95 (APO‐1/Fas) signaling pathways , 1998, The EMBO journal.
[12] D. Goeddel,et al. Casper is a FADD- and caspase-related inducer of apoptosis. , 1997, Immunity.
[13] Howard Y. Chang,et al. Proteases for Cell Suicide: Functions and Regulation of Caspases , 2000, Microbiology and Molecular Biology Reviews.
[14] D. Baltimore,et al. Essential role of CED-4 oligomerization in CED-3 activation and apoptosis. , 1998, Science.
[15] T. Griffith,et al. Intracellular regulation of TRAIL-induced apoptosis in human melanoma cells. , 1998, Journal of immunology.
[16] S. Srinivasula,et al. FLAME-1, a Novel FADD-like Anti-apoptotic Molecule That Regulates Fas/TNFR1-induced Apoptosis* , 1997, The Journal of Biological Chemistry.
[17] Margot Thome,et al. Inhibition of death receptor signals by cellular FLIP , 1997, Nature.
[18] D. Baltimore,et al. Autoimmunity as a consequence of retrovirus-mediated expression of C-FLIP in lymphocytes. , 2009, Immunity.
[19] R. Gentz,et al. I-FLICE, a Novel Inhibitor of Tumor Necrosis Factor Receptor-1- and CD-95-induced Apoptosis* , 1997, The Journal of Biological Chemistry.
[20] Shannon R. Magari,et al. Redesigning an FKBP-ligand interface to generate chemical dimerizers with novel specificity. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[21] P. Krammer,et al. Cellular FLICE-inhibitory Protein Splice Variants Inhibit Different Steps of Caspase-8 Activation at the CD95 Death-inducing Signaling Complex* , 2001, The Journal of Biological Chemistry.
[22] J. Tschopp,et al. NF-κB Signals Induce the Expression of c-FLIP , 2001, Molecular and Cellular Biology.
[23] Ingo Schmitz,et al. The Role of c-FLIP in Modulation of CD95-induced Apoptosis* , 1999, The Journal of Biological Chemistry.
[24] M. Hayden,et al. Cell death attenuation by `Usurpin', a mammalian DED-caspase homologue that precludes caspase-8 recruitment and activation by the CD-95 (Fas, APO-1) receptor complex , 1998, Cell Death and Differentiation.
[25] Peter Scheurich,et al. NF-κB Inducers Upregulate cFLIP, a Cycloheximide-Sensitive Inhibitor of Death Receptor Signaling , 2001, Molecular and Cellular Biology.
[26] J. Blenis,et al. Essential requirement for caspase-8/FLICE in the initiation of the Fas-induced apoptotic cascade , 1998, Current Biology.
[27] S. Chen,et al. CLARP, a death effector domain-containing protein interacts with caspase-8 and regulates apoptosis. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[28] D. Goeddel,et al. Requirement for Casper (c-FLIP) in regulation of death receptor-induced apoptosis and embryonic development. , 2000, Immunity.
[29] H. Chun,et al. Caspase-10 is an initiator caspase in death receptor signaling , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[30] R. Offringa,et al. Immune Escape of Tumors in Vivo by Expression of Cellular Flice-Inhibitory Protein , 1999, The Journal of experimental medicine.
[31] Matthias Mann,et al. FLICE is activated by association with the CD95 death‐inducing signaling complex (DISC) , 1997, The EMBO journal.
[32] Y. Goltsev,et al. CASH, a Novel Caspase Homologue with Death Effector Domains* , 1997, The Journal of Biological Chemistry.
[33] P. Krammer,et al. FLICE-Inhibitory Proteins: Regulators of Death Receptor-Mediated Apoptosis , 2001, Molecular and Cellular Biology.