Dopamine Controls Systemic Inflammation through Inhibition of NLRP3 Inflammasome
暂无分享,去创建一个
Lei Liu | Z. Tian | R. Zhou | Wei Jiang | Yiqing Yan | Xiaqiong Wang | Chen Ding | Rongbin Zhou
[1] Jorge Oliver-De La Cruz,et al. Role of mitochondria ROS generation in ethanol-induced NLRP3 inflammasome activation and cell death in astroglial cells , 2014, Front. Cell. Neurosci..
[2] P. Sanberg,et al. The innate and adaptive immunological aspects in neurodegenerative diseases , 2014, Journal of Neuroimmunology.
[3] S. Ray,et al. Understanding heterogeneity in the host response to Staphylococcus aureus infection for prognostic biomarker discovery , 2014, Critical Care.
[4] Zhigang Tian,et al. Omega-3 fatty acids prevent inflammation and metabolic disorder through inhibition of NLRP3 inflammasome activation. , 2013, Immunity.
[5] M. Brucale,et al. Triggering of Inflammasome by Aggregated α–Synuclein, an Inflammatory Response in Synucleinopathies , 2013, PloS one.
[6] Junying Yuan,et al. Deubiquitination of NLRP3 by BRCC3 critically regulates inflammasome activity. , 2013, Molecular cell.
[7] W. Shao,et al. Suppression of neuroinflammation by astrocytic dopamine D2 receptors via αB-crystallin , 2012, Nature.
[8] Hardy Kornfeld,et al. Nitric oxide controls the immunopathology of tuberculosis by inhibiting NLRP3 inflammasome–dependent processing of IL-1β , 2012, Nature Immunology.
[9] R. Germain,et al. The calcium-sensing receptor regulates the NLRP3 inflammasome through Ca2+ and cAMP , 2012, Nature.
[10] Vishva M Dixit,et al. Inflammasomes and their roles in health and disease. , 2012, Annual review of cell and developmental biology.
[11] E. Alnemri,et al. Non-transcriptional Priming and Deubiquitination Regulate NLRP3 Inflammasome Activation* , 2012, The Journal of Biological Chemistry.
[12] Perry Vh. Innate Inflammation in Parkinson’s Disease , 2012 .
[13] K. Tracey,et al. Rethinking inflammation: neural circuits in the regulation of immunity , 2012, Immunological reviews.
[14] A. Nishi,et al. Mechanisms for the Modulation of Dopamine D1 Receptor Signaling in Striatal Neurons , 2011, Front. Neuroanat..
[15] Haitao Wen,et al. The inflammasome NLRs in immunity, inflammation, and associated diseases. , 2011, Annual review of immunology.
[16] R. Gainetdinov,et al. The Physiology, Signaling, and Pharmacology of Dopamine Receptors , 2011, Pharmacological Reviews.
[17] Jonathan L. Schmid-Burgk,et al. Inflammasomes: current understanding and open questions , 2011, Cellular and Molecular Life Sciences.
[18] Pedro Romero,et al. Type I interferon inhibits interleukin-1 production and inflammasome activation. , 2011, Immunity.
[19] S. Basu,et al. The immunoregulatory role of dopamine: An update , 2010, Brain, Behavior, and Immunity.
[20] J. Vincent,et al. Comparison of dopamine and norepinephrine in the treatment of shock. , 2010, The New England journal of medicine.
[21] J. Bos,et al. Epac: defining a new mechanism for cAMP action. , 2010, Annual review of pharmacology and toxicology.
[22] Tony Wyss-Coray,et al. Immune Activation in Brain Aging and Neurodegeneration: Too Much or Too Little? , 2009, Neuron.
[23] E. Alnemri,et al. Cutting Edge: NF-κB Activating Pattern Recognition and Cytokine Receptors License NLRP3 Inflammasome Activation by Regulating NLRP3 Expression1 , 2009, The Journal of Immunology.
[24] G. Núñez,et al. Cutting Edge: TNF-α Mediates Sensitization to ATP and Silica via the NLRP3 Inflammasome in the Absence of Microbial Stimulation1 , 2009, The Journal of Immunology.
[25] K. Tracey. Reflex control of immunity , 2009, Nature Reviews Immunology.
[26] F. Martinon,et al. The inflammasomes: guardians of the body. , 2009, Annual review of immunology.
[27] E. Hirsch,et al. Infiltration of CD4+ lymphocytes into the brain contributes to neurodegeneration in a mouse model of Parkinson disease. , 2008, The Journal of clinical investigation.
[28] M. Goldman. Faculty Opinions recommendation of The NALP3 inflammasome is involved in the innate immune response to amyloid-beta. , 2008 .
[29] U. R. Chowdhury,et al. Dopamine regulates endothelial progenitor cell mobilization from mouse bone marrow in tumor vascularization. , 2008, The Journal of clinical investigation.
[30] Keith F. Tipton,et al. The therapeutic potential of monoamine oxidase inhibitors , 2006, Nature Reviews Neuroscience.
[31] F. Martinon,et al. Gout-associated uric acid crystals activate the NALP3 inflammasome , 2006, Nature.
[32] N. Barnes,et al. Close encounters of the monoamine kind: immune cells betray their nervous disposition , 2005, Immunology.
[33] B. Yard,et al. Clinical review: Immunomodulatory effects of dopamine in general inflammation , 2004, Critical care.
[34] T. Sotnikova,et al. Lithium antagonizes dopamine-dependent behaviors mediated by an AKT/glycogen synthase kinase 3 signaling cascade. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[35] J. Serratosa,et al. High‐yield isolation of murine microglia by mild trypsinization , 2003, Glia.
[36] A. Mondal,et al. Dopamine inhibits cytokine release and expression of tyrosine kinases, Lck and Fyn in activated T cells. , 2003, International immunopharmacology.
[37] R. Moots,et al. Dopamine receptor expression on human T- and B-lymphocytes, monocytes, neutrophils, eosinophils and NK cells: a flow cytometric study , 2002, Journal of Neuroimmunology.
[38] F. Gonon,et al. Changes in Extracellular Dopamine Induced by Morphine and Cocaine: Crucial Control by D2 Receptors , 2002, The Journal of Neuroscience.
[39] Janice A. Nagy,et al. The neurotransmitter dopamine inhibits angiogenesis induced by vascular permeability factor/vascular endothelial growth factor , 2001, Nature Medicine.
[40] S. Basu,et al. Dopamine, a neurotransmitter, influences the immune system , 2000, Journal of Neuroimmunology.
[41] S. Tayebati,et al. Dopamine D1-like receptor subtypes in human peripheral blood lymphocytes , 1999, Journal of Neuroimmunology.
[42] R. G. Allen,et al. Pituitary Lactotroph Hyperplasia and Chronic Hyperprolactinemia in Dopamine D2 Receptor-Deficient Mice , 1997, Neuron.
[43] J. Bergquist,et al. Catecholamines are synthesized by mouse lymphocytes and regulate function of these cells by induction of apoptosis , 1996, Immunology.
[44] C. Gerfen,et al. Altered striatal function in a mutant mouse lacking D1A dopamine receptors. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[45] J. Bergquist,et al. Discovery of endogenous catecholamines in lymphocytes and evidence for catecholamine regulation of lymphocyte function via an autocrine loop. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[46] V. Perry. Innate inflammation in Parkinson's disease. , 2012, Cold Spring Harbor perspectives in medicine.
[47] J. Tschopp,et al. The Inflammasomes , 2010, Cell.
[48] Ashley Harms. Inflammation in Parkinson's disease. , 2001, Advances in neurology.
[49] S. R. Nash,et al. Dopamine receptors: from structure to function. , 1998, Physiological reviews.