Adrenergic modulation of cytokine release in bone marrow progenitor-derived macrophage following polymicrobial sepsis
暂无分享,去创建一个
[1] R. Gamelli,et al. Bone Marrow Norepinephrine Mediates Development of Functionally Different Macrophages After Thermal Injury and Sepsis , 2004, Annals of surgery.
[2] C. Dinarello. Anti-cytokine therapeutics and infections. , 2003, Vaccine.
[3] D. Mannino,et al. The epidemiology of sepsis in the United States from 1979 through 2000. , 2003, The New England journal of medicine.
[4] Y. Suh,et al. Cyclic AMP inhibition of tumor necrosis factor alpha production induced by amyloidogenic C-terminal peptide of Alzheimer's amyloid precursor protein in macrophages: involvement of multiple intracellular pathways and cyclic AMP response element binding protein. , 2003, Molecular pharmacology.
[5] M. Bergmann,et al. Immunomodulatory effects of vasoactive catecholamines. , 2002, Wiener klinische Wochenschrift.
[6] G. Bancroft,et al. Adrenaline suppression of the macrophage nitric oxide response to lipopolysaccharide is associated with differential regulation of tumour necrosis factor‐α and interleukin‐10 , 2001, Immunology.
[7] L. Moldawer,et al. SEPSIS SYNDROMES: UNDERSTANDING THE ROLE OF INNATE AND ACQUIRED IMMUNITY , 2001, Shock.
[8] G. Clermont,et al. Epidemiology of severe sepsis in the United States: Analysis of incidence, outcome, and associated costs of care , 2001, Critical care medicine.
[9] R. Gamelli,et al. Norepinephrine Modulates Myelopoiesis After Experimental Thermal Injury With Sepsis , 2001, Annals of surgery.
[10] R. Gamelli,et al. Myeloid Commitment Shifts Toward Monocytopoiesis After Thermal Injury and Sepsis , 2001, Annals of surgery.
[11] Petra Reinke,et al. Catecholamines induce IL-10 release in patients suffering from acute myocardial infarction by transactivating its promoter in monocytic but not in T-cells , 2000, Molecular and Cellular Biochemistry.
[12] L. Sigola,et al. Adrenaline inhibits macrophage nitric oxide production through β1 and β2 adrenergic receptors , 2000, Immunology.
[13] M. Mittlböck,et al. Attenuation of catecholamine-induced immunosuppression in whole blood from patients with sepsis. , 1999, Shock.
[14] I. C. Almeida,et al. Differential inhibitory mechanism of cyclic AMP on TNF‐α and IL‐12 synthesis by macrophages exposed to microbial stimuli , 1999, British journal of pharmacology.
[15] S. Akira,et al. Interleukin-10 Receptor Signaling through the JAK-STAT Pathway , 1999, The Journal of Biological Chemistry.
[16] R. Gamelli,et al. Dynamic norepinephrine alterations in bone marrow: evidence of functional innervation , 1999, Journal of Neuroimmunology.
[17] J. Lederer,et al. The effects of injury on the adaptive immune response. , 1999, Shock.
[18] D. H. Lee,et al. Release of tumor necrosis factor-α from coronary smooth muscle : Activation of NF-κB and inhibition by elevated cyclic AMP , 1998 .
[19] Khusru Asadullah,et al. Sympathetic activation triggers systemic interleukin-10 release in immunodepression induced by brain injury , 1998, Nature Medicine.
[20] E. Vizi,et al. Isoproterenol regulates tumour necrosis factor, interleukin‐10, interleukin‐6 and nitric oxide production and protects against the development of vascular hyporeactivity in endotoxaemia , 1997, Immunology.
[21] O. Lantz,et al. Regulation of interleukin‐10 production by β‐adrenergic agonists , 1996, European journal of immunology.
[22] Gregory J. Goodall,et al. Differential Regulation of the Stability of Cytokine mRNAs in Lipopolysaccharide-activated Blood Monocytes in Response to Interleukin-10* , 1996, The Journal of Biological Chemistry.
[23] S. Opal,et al. Treatment of septic shock with the tumor necrosis factor receptor:Fc fusion protein. The Soluble TNF Receptor Sepsis Study Group. , 1996, The New England journal of medicine.
[24] T. van der Poll,et al. Epinephrine inhibits tumor necrosis factor-alpha and potentiates interleukin 10 production during human endotoxemia. , 1996, The Journal of clinical investigation.
[25] J. Keiser,et al. Role of epinephrine in TNF and IL-6 production from isolated perfused rat liver. , 1995, The American journal of physiology.
[26] T. van der Poll,et al. Noradrenaline inhibits lipopolysaccharide-induced tumor necrosis factor and interleukin 6 production in human whole blood , 1994, Infection and immunity.
[27] M. Howard,et al. Biological properties of interleukin 10 , 1992, Journal of Clinical Immunology.
[28] Y. Vodovotz,et al. Macrophage deactivation by interleukin 10 , 1991, The Journal of experimental medicine.
[29] T. Mosmann,et al. IL-10 inhibits cytokine production by activated macrophages. , 1991, Journal of immunology.
[30] C G Figdor,et al. Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes , 1991, The Journal of experimental medicine.
[31] M. Westfall,et al. Plasma catecholamines during E. coli bacteremia in conscious rats. , 1988, The American journal of physiology.
[32] F. Romano,et al. Cardiac and splenic norepinephrine turnover during septic peritonitis. , 1986, The American journal of physiology.
[33] I. Chaudry,et al. Sepsis and septic shock--a review of laboratory models and a proposal. , 1980, The Journal of surgical research.
[34] R. Bone,et al. Toward a theory regarding the pathogenesis of the systemic inflammatory response syndrome: what we do and do not know about cytokine regulation. , 1996, Critical care medicine.
[35] E. Faist. The mechanisms of host defense dysfunction following shock and trauma. , 1996, Current topics in microbiology and immunology.
[36] F. Romano,et al. Plasma catecholamines following cecal ligation and puncture in the rat. , 1987, Circulatory shock.
[37] C. Benedict,et al. Plasma noradrenaline and adrenaline concentrations and dopamine-beta-hydroxylase activity in patients with shock due to septicaemia, trauma and haemorrhage. , 1978, The Quarterly journal of medicine.