Central Role for the Cardiotonic Steroid Marinobufagenin in the Pathogenesis of Experimental Uremic Cardiomyopathy
暂无分享,去创建一个
Deepak Malhotra | Samer Khouri | Edward G Lakatta | E. Lakatta | M. Kanj | Larisa V. Fedorova | J. Shapiro | Zijian Xie | O. Fedorova | Zijian Xie | D. Kennedy | D. Malhotra | A. Bagrov | M. Kahaleh | Mohamed Kanj | Joseph I Shapiro | S. Khouri | Larisa Fedorova | David J Kennedy | Sandeep Vetteth | Sankaridrug M Periyasamy | M Bashar Kahaleh | Nikolai I Kolodkin | Olga V Fedorova | Alexei Y Bagrov | S. Periyasamy | S. Vetteth | N. I. Kolodkin
[1] J. Himmelfarb,et al. Plasma aminothiol oxidation in chronic hemodialysis patients. , 2002, Kidney international.
[2] R. Foley,et al. Left ventricular hypertrophy in the renal patient. , 2001, Journal of the American Society of Nephrology : JASN.
[3] Chi Han,et al. Effect of green tea extract on cardiac hypertrophy following 5/6 nephrectomy in the rat. , 2003, Kidney international.
[4] H. Masaki,et al. A novel endogenous digitalis, telocinobufagin, exhibits elevated plasma levels in patients with terminal renal failure. , 2005, Clinical biochemistry.
[5] B. Pitt. Effectiveness of spironolactone added to an angiotensin-converting enzyme inhibitor and a loop diuretic for severe chronic congestive heart failure (the Randomized Aldactone Evaluation Study [RALES]). , 1996, The American journal of cardiology.
[6] L. Bouter,et al. Mild renal insufficiency is associated with increased cardiovascular mortality: The Hoorn Study. , 2002, Kidney international.
[7] N. Bricker. On the pathogenesis of the uremic state--an exposition of the trade-off hypothesis. , 1972, Nihon Jinzo Gakkai shi.
[8] A. Askari,et al. Involvement of Src and epidermal growth factor receptor in the signal-transducing function of Na+/K+-ATPase. , 2000, The Journal of biological chemistry.
[9] W. Border,et al. Transforming growth factor beta in tissue fibrosis. , 1994, The New England journal of medicine.
[10] J. Willey,et al. Effect of chronic renal failure on cardiac contractile function, calcium cycling, and gene expression of proteins important for calcium homeostasis in the rat. , 2003, Journal of the American Society of Nephrology : JASN.
[11] P. Douglas,et al. Serial echocardiographic assessment of left ventricular geometry and function after large myocardial infarction in the rat. , 1994, Circulation.
[12] H. D. de Wardener. Franz Volhard Lecture 1996. Sodium transport inhibitors and hypertension. , 1996, Journal of hypertension. Supplement : official journal of the International Society of Hypertension.
[13] E. Lakatta,et al. Brain ouabain stimulates peripheral marinobufagenin via angiotensin II signalling in NaCl-loaded Dahl-S rats , 2005, Journal of hypertension.
[14] Jiang Tian,et al. Ouabain interaction with cardiac Na+/K+-ATPase initiates signal cascades independent of changes in intracellular Na+ and Ca2+ concentrations. , 2000, The Journal of biological chemistry.
[15] E. Zvartau,et al. Involvement of endogenous digitalis-like factors in voluntary selection of alcohol by rats. , 1999, Life sciences.
[16] B. Kahn,et al. Multiple Signal Transduction Pathways Link Na+/K+-ATPase to Growth-related Genes in Cardiac Myocytes , 1998, The Journal of Biological Chemistry.
[17] O. Fedorova,et al. Plasma marinobufagenin-like and ouabain-like immunoreactivity in adrenocorticotropin-treated rats. , 1998, American journal of hypertension.
[18] Sfj Langer. Differential laws of left ventricular isovolumic pressure fall. , 2002, Physiological research.
[19] R. Foley,et al. Congestive heart failure in renal transplant recipients: risk factors, outcomes, and relationship with ischemic heart disease. , 2002, Journal of the American Society of Nephrology : JASN.
[20] Bertram L Kasiske,et al. Kidney disease as a risk factor for development of cardiovascular disease: a statement from the American Heart Association Councils on Kidney in Cardiovascular Disease, High Blood Pressure Research, Clinical Cardiology, and Epidemiology and Prevention. , 2003, Circulation.
[21] R. Buñag,et al. Tail‐Cuff Blood Pressure Measurement without External Preheating in Awake Rats , 1982, Hypertension.
[22] H. E. Wardener. Franz Volhard Lecture 1996. Sodium transport inhibitors and hypertension. , 1996 .
[23] C. van Hardeveld,et al. An assay for sarcoplasmic reticulum Ca2(+)-ATPase activity in muscle homogenates. , 1990, Analytical biochemistry.
[24] E. Lakatta,et al. Endogenous Ligand of &agr;1 Sodium Pump, Marinobufagenin, Is a Novel Mediator of Sodium Chloride–Dependent Hypertension , 2002, Circulation.
[25] P. Parfrey. Is renal insufficiency an atherogenic state? Reflections on prevalence, incidence, and risk. , 2001, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[26] J. Himmelfarb,et al. Plasma protein thiol oxidation and carbonyl formation in chronic renal failure. , 2000, Kidney international.
[27] A. Pinaev,et al. Effects of two endogenous Na+,K(+)-ATPase inhibitors, marinobufagenin and ouabain, on isolated rat aorta. , 1995, European journal of pharmacology.
[28] J. Himmelfarb,et al. Albumin is the major plasma protein target of oxidant stress in uremia. , 2001, Kidney international.
[29] H. Li,et al. Ouabain-induced hypertrophy in cultured cardiac myocytes is accompanied by changes in expression of several late response genes. , 1997, Journal of molecular and cellular cardiology.
[30] G. Bianchi,et al. Organ Hypertrophic Signaling within Caveolae Membrane Subdomains Triggered by Ouabain and Antagonized by PST 2238* , 2004, Journal of Biological Chemistry.
[31] P. Parfrey,et al. Risk factors for the development of left ventricular hypertrophy in a prospectively followed cohort of dialysis patients. , 1994, Journal of the American Society of Nephrology : JASN.
[32] N S Bricker,et al. The control of sodium excretion with normal and reduced nephron populations. The pre-eminence of third factor. , 1967, The American journal of medicine.
[33] J. Shapiro,et al. Intracellular Reactive Oxygen Species Mediate the Linkage of Na+/K+-ATPase to Hypertrophy and Its Marker Genes in Cardiac Myocytes* , 1999, The Journal of Biological Chemistry.
[34] M. Young,et al. Mineralocorticoid receptors and pathophysiological roles for aldosterone in the cardiovascular system. , 2002, Journal of hypertension.
[35] R. Foley,et al. Risk factors for cardiac morbidity and mortality in dialysis patients. , 1994, Current opinion in nephrology and hypertension.
[36] T. Hostetter,et al. Role of aldosterone in the remnant kidney model in the rat. , 1996, The Journal of clinical investigation.
[37] A. Garg,et al. Moderate renal insufficiency and the risk of cardiovascular mortality: results from the NHANES I. , 2002, Kidney international.
[38] J. Shapiro,et al. Ouabain-induced endocytosis of the plasmalemmal Na/K-ATPase in LLC-PK1 cells requires caveolin-1. , 2005, Kidney international.
[39] W. Border,et al. Transforming Growth Factor β in Tissue Fibrosis , 1994 .
[40] J. Shapiro,et al. Salt loading induces redistribution of the plasmalemmal Na/K-ATPase in proximal tubule cells. , 2005, Kidney international.
[41] Bertram L Kasiske,et al. Kidney disease as a risk factor for development of cardiovascular disease: a statement from the American Heart Association Councils on Kidney in Cardiovascular Disease, High Blood Pressure Research, Clinical Cardiology, and Epidemiology and Prevention. , 2003, Hypertension.
[42] Zijian Xie,et al. Role of caveolae in signal-transducing function of cardiac Na+/K+-ATPase. , 2003, American journal of physiology. Cell physiology.
[43] B. Brenner,et al. Hyperfiltration in remnant nephrons: a potentially adverse response to renal ablation. , 1981, The American journal of physiology.
[44] O. Fedorova,et al. Endogenous marinobufagenin-like factor in acute plasma volume expansion. , 1998, Clinical and experimental hypertension.