Insulin Receptor Substrate-2 Is Not Necessary for Insulin- and Exercise-stimulated Glucose Transport in Skeletal Muscle*
暂无分享,去创建一个
M. White | H. Towery | Y. Higaki | D. Withers | L. Goodyear | J. Wojtaszewski | M. Hirshman | Jørgen F. P. Wojtaszewski | Michael F. Hirshman | Heather Towery | Laurie J. Goodyear | Heather H. Towery
[1] M. Czech,et al. Signaling Mechanisms That Regulate Glucose Transport* , 1999, The Journal of Biological Chemistry.
[2] C. Kahn,et al. A muscle-specific insulin receptor knockout exhibits features of the metabolic syndrome of NIDDM without altering glucose tolerance. , 1998, Molecular cell.
[3] Tatsuya Hayashi,et al. Evidence for 5′AMP-Activated Protein Kinase Mediation of the Effect of Muscle Contraction on Glucose Transport , 1998, Diabetes.
[4] K. Siddle,et al. Phosphoinositide 3-kinase: the key switch mechanism in insulin signalling. , 1998, The Biochemical journal.
[5] I. Tabata,et al. Effects of high-intensity intermittent swimming on glucose transport in rat epitrochlearis muscle. , 1998, Journal of applied physiology.
[6] G. Shulman,et al. Disruption of IRS-2 causes type 2 diabetes in mice , 1998, Nature.
[7] G. Lienhard,et al. A Novel 160-kDa Phosphotyrosine Protein in Insulin-treated Embryonic Kidney Cells Is a New Member of the Insulin Receptor Substrate Family* , 1997, The Journal of Biological Chemistry.
[8] J. Eckel,et al. Molecular mechanisms of contraction-induced translocation of GLUT4 in isolated cardiomyocytes. , 1997, The American journal of cardiology.
[9] G. Lienhard,et al. The 60-kDa Phosphotyrosine Protein in Insulin-treated Adipocytes Is a New Member of the Insulin Receptor Substrate Family* , 1997, The Journal of Biological Chemistry.
[10] J. Zierath,et al. Insulin Receptor Substrate-1 Phosphorylation and Phosphatidylinositol 3-Kinase Activity in Skeletal Muscle From NIDDM Subjects After In Vivo Insulin Stimulation , 1997, Diabetes.
[11] William Arbuthnot Sir Lane,et al. Role of IRS-2 in insulin and cytokine signalling , 1995, Nature.
[12] O. Pedersen,et al. Contraction stimulates translocation of glucose transporter GLUT4 in skeletal muscle through a mechanism distinct from that of insulin. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[13] F. Giorgino,et al. Insulin receptor phosphorylation, insulin receptor substrate-1 phosphorylation, and phosphatidylinositol 3-kinase activity are decreased in intact skeletal muscle strips from obese subjects. , 1995, The Journal of clinical investigation.
[14] C. Kahn,et al. Insulin action and the insulin signaling network. , 1995, Endocrine reviews.
[15] J. Holloszy,et al. Wortmannin inhibits insulin‐stimulated but not contraction‐stimulated glucose transport activity in skeletal muscle , 1995, FEBS letters.
[16] M. Birnbaum,et al. The Effects of Wortmannin on Rat Skeletal Muscle , 1995, The Journal of Biological Chemistry.
[17] C. Kahn,et al. Alternative pathway of insulin signalling in mice with targeted disruption of the IRS-1 gene , 1994, Nature.
[18] T. Yagi,et al. Insulin resistance and growth retardation in mice lacking insulin receptor substrate-1 , 1994, Nature.
[19] J. Blenis,et al. Phosphatidylinositol 3-kinase activation is required for insulin stimulation of pp70 S6 kinase, DNA synthesis, and glucose transporter translocation , 1994, Molecular and cellular biology.
[20] M. Kasuga,et al. Inhibition of the translocation of GLUT1 and GLUT4 in 3T3-L1 cells by the phosphatidylinositol 3-kinase inhibitor, wortmannin. , 1994, The Biochemical journal.
[21] C. Kahn,et al. The insulin signaling system. , 1994, The Journal of biological chemistry.
[22] E. Ravussin,et al. Insulin resistance and insulin secretory dysfunction as precursors of non-insulin-dependent diabetes mellitus. Prospective studies of Pima Indians. , 1993, The New England journal of medicine.
[23] Dominique,et al. Defect in skeletal muscle phosphatidylinositol-3-kinase in obese insulin-resistant mice. , 1993, The Journal of clinical investigation.
[24] M J Saad,et al. Regulation of insulin receptor substrate-1 in liver and muscle of animal models of insulin resistance. , 1992, The Journal of clinical investigation.
[25] C. Kahn,et al. Structure of the insulin receptor substrate IRS-1 defines a unique signal transduction protein , 1991, Nature.
[26] A. Klip,et al. Exercise modulates the insulin‐induced translocation of glucose transporters in rat skeletal muscle , 1990, FEBS letters.
[27] R. DeFronzo. The Triumvirate: β-Cell, Muscle, Liver: A Collusion Responsible for NIDDM , 1988, Diabetes.