Anti-hypertrophic and anti-oxidant effect of beta3-adrenergic stimulation in myocytes requires differential neuronal NOS phosphorylation.

[1]  M. Crabtree,et al.  Regulation of Endothelial Nitric-oxide Synthase (NOS) S-Glutathionylation by Neuronal NOS , 2012, The Journal of Biological Chemistry.

[2]  D. Kass,et al.  Erratum: Cardioprotective effect of beta-3 adrenergic receptor agonism: Role of neuronal nitric oxide synthase (Journal of the American College of Cardiology (2012) 59 (1979-1987)) , 2012 .

[3]  D. Kass,et al.  Cardioprotective effect of beta-3 adrenergic receptor agonism: role of neuronal nitric oxide synthase. , 2012, Journal of the American College of Cardiology.

[4]  B. Casadei,et al.  Sub-cellular targeting of constitutive NOS in health and disease. , 2012, Journal of molecular and cellular cardiology.

[5]  S. Jha,et al.  Beta3-adrenoreceptor stimulation ameliorates myocardial ischemia-reperfusion injury via endothelial nitric oxide synthase and neuronal nitric oxide synthase activation. , 2011, Journal of the American College of Cardiology.

[6]  I-Chin Chen,et al.  Protein kinase A-dependent Neuronal Nitric Oxide Synthase Activation Mediates the Enhancement of Baroreflex Response by Adrenomedullin in the Nucleus Tractus Solitarii of Rats , 2011, Journal of Biomedical Sciences.

[7]  A. Burnett,et al.  Activity-Dependent Regulation of Surface Glucose Transporter-3 , 2011, The Journal of Neuroscience.

[8]  L. Barouch,et al.  Beta 3-adrenoreceptor regulation of nitric oxide in the cardiovascular system. , 2010, Journal of molecular and cellular cardiology.

[9]  Weimin Li,et al.  [Association between beta3-adrenergic receptor and oxidative stress in chronic heart failure rats]. , 2010, Zhonghua xin xue guan bing za zhi.

[10]  D. Kass,et al.  Adverse ventricular remodeling and exacerbated NOS uncoupling from pressure-overload in mice lacking the beta3-adrenoreceptor. , 2009, Journal of molecular and cellular cardiology.

[11]  E. Motley,et al.  Role of Protease-Activated Receptor-1 in Endothelial Nitric Oxide Synthase-Thr495 Phosphorylation , 2009, Experimental biology and medicine.

[12]  R. Schwinger,et al.  Effects of the beta3-adrenergic agonist BRL 37344 on endothelial nitric oxide synthase phosphorylation and force of contraction in human failing myocardium. , 2009, Journal of cardiac failure.

[13]  R. Andriantsitohaina,et al.  Involvement of β3-Adrenoceptor in Altered β-Adrenergic Response in Senescent Heart: Role of Nitric Oxide Synthase 1–derived Nitric Oxide , 2008, Anesthesiology.

[14]  Guang-yi Zhang,et al.  Neuroprotection of γ‐aminobutyric acid receptor agonists via enhancing neuronal nitric oxide synthase (Ser847) phosphorylation through increased neuronal nitric oxide synthase and PSD95 interaction and inhibited protein phosphatase activity in cerebral ischemia , 2008, Journal of Neuroscience Research.

[15]  A. Recchia,et al.  β3‐Adrenoceptors modulate left ventricular relaxation in the rat heart via the NO‐cGMP‐PKG pathway , 2008, Acta Physiologica.

[16]  R. Andriantsitohaina,et al.  Altered Contractile Response due to Increased &bgr;3-Adrenoceptor Stimulation in Diabetic Cardiomyopathy: The Role of Nitric Oxide Synthase 1–derived Nitric Oxide , 2007, Anesthesiology.

[17]  E. Getzoff,et al.  Biphasic Coupling of Neuronal Nitric Oxide Synthase Phosphorylation to the NMDA Receptor Regulates AMPA Receptor Trafficking and Neuronal Cell Death , 2007, The Journal of Neuroscience.

[18]  K. Osuka,et al.  Phosphorylation of neuronal nitric oxide synthase at Ser847 in the nucleus intermediolateralis after spinal cord injury in mice , 2007, Neuroscience.

[19]  R. Schwinger,et al.  Beta3-adrenergic eNOS stimulation in left ventricular murine myocardium. , 2006, Canadian journal of physiology and pharmacology.

[20]  D. Kass,et al.  Tetrahydrobiopterin and Cardiovascular Disease , 2006, Arteriosclerosis, thrombosis, and vascular biology.

[21]  R. Germack,et al.  Induction of β3-Adrenergic Receptor Functional Expression following Chronic Stimulation with Noradrenaline in Neonatal Rat Cardiomyocytes , 2006, Journal of Pharmacology and Experimental Therapeutics.

[22]  S. Neubauer,et al.  nNOS Gene Deletion Exacerbates Pathological Left Ventricular Remodeling and Functional Deterioration After Myocardial Infarction , 2005, Circulation.

[23]  T. Hintze,et al.  A Defect of Neuronal Nitric Oxide Synthase Increases Xanthine Oxidase–Derived Superoxide Anion and Attenuates the Control of Myocardial Oxygen Consumption by Nitric Oxide Derived From Endothelial Nitric Oxide Synthase , 2005, Circulation research.

[24]  B. Fleischmann,et al.  Mechanisms of β3‐adrenoceptor‐induced eNOS activation in right atrial and left ventricular human myocardium , 2004, British journal of pharmacology.

[25]  J. Hare,et al.  Neuronal nitric oxide synthase negatively regulates xanthine oxidoreductase inhibition of cardiac excitation-contraction coupling. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[26]  C. Gauthier,et al.  Impairment of the low‐affinity state β1‐adrenoceptor‐induced relaxation in spontaneously hypertensive rats , 2004, British Journal of Pharmacology.

[27]  E. Ziff,et al.  Bidirectional Regulation of Neuronal Nitric-oxide Synthase Phosphorylation at Serine 847 by the N-Methyl-d-aspartate Receptor* , 2004, Journal of Biological Chemistry.

[28]  E. Ziff,et al.  NMDA receptor regulation of nNOS phosphorylation and induction of neuron death , 2003, Neurobiology of Aging.

[29]  K. Fukunaga,et al.  Phosphorylation of Neuronal Nitric Oxide Synthase at Ser847 by CaM-KII in the Hippocampus of Rat Brain after Transient Forebrain Ischemia , 2002, Journal of Cerebral Blood Flow and Metabolism.

[30]  I. Afanas’ev.  Lucigenin chemiluminescence assay for superoxide detection. , 2001, Circulation Research.

[31]  J. Balligand,et al.  Upregulation of &bgr;3-Adrenoceptors and Altered Contractile Response to Inotropic Amines in Human Failing Myocardium , 2001, Circulation.

[32]  K. Bidasee,et al.  The effect of diabetes on expression of β1-, β2-, and β3-Adrenoreceptors in rat hearts , 2001 .

[33]  L. Luttrell,et al.  The β3-Adrenergic Receptor Activates Mitogen-activated Protein Kinase in Adipocytes through a Gi-dependent Mechanism* , 1999, The Journal of Biological Chemistry.

[34]  J. Balligand,et al.  The negative inotropic effect of beta3-adrenoceptor stimulation is mediated by activation of a nitric oxide synthase pathway in human ventricle. , 1998, The Journal of clinical investigation.

[35]  S. Liggett,et al.  Structural basis for receptor subtype-specific regulation revealed by a chimeric beta 3/beta 2-adrenergic receptor. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[36]  A. Strosberg,et al.  The human beta 3-adrenergic receptor is resistant to short term agonist-promoted desensitization. , 1993, Molecular pharmacology.

[37]  J. Arch,et al.  Thermogenic and antiobesity activity of a novel beta-adrenoceptor agonist (BRL 26830A) in mice and rats. , 1983, American Journal of Clinical Nutrition.

[38]  F. Epstein,et al.  Displacement-encoded and manganese-enhanced cardiac MRI reveal that nNOS, not eNOS, plays a dominant role in modulating contraction and calcium influx in the mammalian heart. , 2012, American Journal of Physiology. Heart and Circulatory Physiology.

[39]  Joao A. C. Lima,et al.  Nitric oxide regulates the heart by spatial confinement of nitric oxide synthase isoforms , 2002, Nature.