Innate partnership of HLA-B and KIR3DL1 subtypes against HIV-1
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Amalio Telenti | Peter Parham | Stephen J O'Brien | Mark Connors | James J Goedert | Mary Carrington | J. Goedert | F. Pereyra | M. Carrington | B. Walker | J. Phair | S. O’Brien | Y. Qi | A. Telenti | P. Parham | Jeffrey N. Martin | S. Buchbinder | M. Connors | W. L. Shupert | S. Deeks | E. Brown | G. Kirk | Daniel W McVicar | Steven G Deeks | Maureen P. Martin | Ying Qi | Jeffrey N Martin | Florencia Pereyra | Sara Colombo | Susan Buchbinder | Elizabeth E Brown | Bruce D Walker | Gregory D Kirk | Xiaojiang Gao | John Phair | S. Colombo | Maureen P Martin | Eriko Yamada | W Lesley Shupert | D. McVicar | Eriko Yamada | Stephen J. O’Brien | Xiaojiang Gao | W. Shupert | E. Brown | Jeffrey N. Martin
[1] P. Parham. Taking license with natural killer cell maturation and repertoire development , 2006, Immunological reviews.
[2] B. Vuylsteke,et al. Cutting Edge: Resistance to HIV-1 Infection among African Female Sex Workers Is Associated with Inhibitory KIR in the Absence of Their HLA Ligands1 , 2006, The Journal of Immunology.
[3] D. Middleton,et al. Human NK cell education by inhibitory receptors for MHC class I. , 2006, Immunity.
[4] J. Goedert,et al. KIR/HLA Pleiotropism: Protection against Both HIV and Opportunistic Infections , 2006, PLoS pathogens.
[5] R. Vance,et al. Self-tolerance of natural killer cells , 2006, Nature Reviews Immunology.
[6] P. Parham,et al. Roles for HLA and KIR polymorphisms in natural killer cell repertoire selection and modulation of effector function , 2006, The Journal of experimental medicine.
[7] W. Yokoyama,et al. How do natural killer cells find self to achieve tolerance? , 2006, Immunity.
[8] J. Goedert,et al. AIDS restriction HLA allotypes target distinct intervals of HIV-1 pathogenesis , 2005, Nature Medicine.
[9] Jeffrey N. Martin,et al. Phenotypic, Functional, and Kinetic Parameters Associated with Apparent T-Cell Control of Human Immunodeficiency Virus Replication in Individuals with and without Antiretroviral Treatment , 2005, Journal of Virology.
[10] P. Parham,et al. KIR3DL1 Polymorphisms That Affect NK Cell Inhibition by HLA-Bw4 Ligand1 , 2005, The Journal of Immunology.
[11] L. Lybarger,et al. Licensing of natural killer cells by host major histocompatibility complex class I molecules , 2005, Nature.
[12] R. Vance,et al. A subset of natural killer cells achieves self-tolerance without expressing inhibitory receptors specific for self-MHC molecules. , 2005, Blood.
[13] P. Parham,et al. Natural selection drives recurrent formation of activating killer cell immunoglobulin-like receptor and Ly49 from inhibitory homologues , 2005, The Journal of experimental medicine.
[14] Andrew Glass,et al. Hierarchy of resistance to cervical neoplasia mediated by combinations of killer immunoglobulin-like receptor and human leukocyte antigen loci , 2005 .
[15] L. Rodrigo,et al. Interaction between KIR3DL1 and HLA-B*57 supertype alleles influences the progression of HIV-1 infection in a Zambian population. , 2005, Human immunology.
[16] Lewis L Lanier,et al. NK cell recognition. , 2005, Annual review of immunology.
[17] Bette Korber,et al. Dominant influence of HLA-B in mediating the potential co-evolution of HIV and HLA , 2004, Nature.
[18] Salim I. Khakoo,et al. HLA and NK Cell Inhibitory Receptor Genes in Resolving Hepatitis C Virus Infection , 2004, Science.
[19] Philip J. R. Goulder,et al. HIV and SIV CTL escape: implications for vaccine design , 2004, Nature Reviews Immunology.
[20] P. Parham,et al. The Protein Made from a Common Allele of KIR3DL1 (3DL1*004) Is Poorly Expressed at Cell Surfaces due to Substitution at Positions 86 in Ig Domain 0 and 182 in Ig Domain 1 1 , 2003, The Journal of Immunology.
[21] Stephen J O'Brien,et al. The influence of HLA genotype on AIDS. , 2003, Annual review of medicine.
[22] John D. Storey,et al. Statistical significance for genomewide studies , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[23] C. Hallahan,et al. HIV-specific CD8+ T cell proliferation is coupled to perforin expression and is maintained in nonprogressors , 2002, Nature Immunology.
[24] P. Leibson,et al. Natural killer cell activation in mice and men: different triggers for similar weapons? , 2002, Nature Immunology.
[25] Keith Hoots,et al. Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS , 2002, Nature Genetics.
[26] P. Parham,et al. Variation within the human killer cell immunoglobulin-like receptor (KIR) gene family. , 2002, Critical reviews in immunology.
[27] Peter Parham,et al. KIR: diverse, rapidly evolving receptors of innate and adaptive immunity. , 2002, Annual review of immunology.
[28] M. Carrington,et al. HLA and AIDS: a cautionary tale. , 2001, Trends in molecular medicine.
[29] J J Goedert,et al. Effect of a single amino acid change in MHC class I molecules on the rate of progression to AIDS. , 2001, The New England journal of medicine.
[30] E. Rosenberg,et al. Control of HIV-1 viremia and protection from AIDS are associated with HLA-Bw4 homozygosity , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[31] William H. Carr,et al. Different NK Cell Surface Phenotypes Defined by the DX9 Antibody Are Due to KIR3DL1 Gene Polymorphism1 , 2001, The Journal of Immunology.
[32] D. G. Muir,et al. Rapid evolution of NK cell receptor systems demonstrated by comparison of chimpanzees and humans. , 2000, Immunity.
[33] H. Rammensee,et al. SYFPEITHI: database for MHC ligands and peptide motifs , 1999, Immunogenetics.
[34] C. Biron,et al. Natural killer cells in antiviral defense: function and regulation by innate cytokines. , 1999, Annual review of immunology.
[35] J. Dorfman,et al. Acquisition of Ly49 Receptor Expression by Developing Natural Killer Cells , 1998, The Journal of experimental medicine.
[36] J. Margolick,et al. Prognostic indicators for AIDS and infectious disease death in HIV-infected injection drug users: plasma viral load and CD4+ cell count. , 1998, JAMA.
[37] P Parham,et al. Functionally and structurally distinct NK cell receptor repertoires in the peripheral blood of two human donors. , 1997, Immunity.
[38] P Parham,et al. Conserved and variable residues within the Bw4 motif of HLA-B make separable contributions to recognition by the NKB1 killer cell-inhibitory receptor. , 1997, Journal of immunology.
[39] J. Dorfman,et al. Major histocompatibility complex class I‐dependent skewing of the natural killer cell Ly49 receptor reportoire , 1996 .
[40] J. Dorfman,et al. Major histocompatibility complex class I-dependent skewing of the natural killer cell Ly49 receptor repertoire. , 1996, European journal of immunology.
[41] G. Ferrara,et al. NK3-specific natural killer cells are selectively inhibited by Bw4- positive HLA alleles with isoleucine 80 , 1994, The Journal of experimental medicine.
[42] S. Buchbinder,et al. Long‐term HIV‐1 infection without immunologic progression , 1994, AIDS.
[43] R. Oswald. A cautionary tale. , 1994, Accident and emergency nursing.
[44] J. Klein,et al. The molecular descent of the major histocompatibility complex. , 1993, Annual review of immunology.
[45] J. Phair,et al. Acquired immune deficiency syndrome occurring within 5 years of infection with human immunodeficiency virus type-1: the Multicenter AIDS Cohort Study. , 1992, Journal of acquired immune deficiency syndromes.
[46] J. Goedert,et al. A prospective study of human immunodeficiency virus type 1 infection and the development of AIDS in subjects with hemophilia. , 1989, The New England journal of medicine.