Nicotine stimulates the expression of cyclooxygenase-2 mRNA via NFkappaB activation in human gingival fibroblasts.
暂无分享,去创建一个
[1] J. Bradley,et al. TNF‐mediated inflammatory disease , 2008, The Journal of pathology.
[2] L. Ulloa,et al. The alpha7 nicotinic acetylcholine receptor as a pharmacological target for inflammation , 2007, British journal of pharmacology.
[3] G. K. Johnson,et al. The impact of cigarette smoking on periodontal disease and treatment. , 2007, Periodontology 2000.
[4] B. Olson,et al. Nicotine and lipopolysaccharide affect cytokine expression from gingival fibroblasts. , 2007, Journal of periodontology.
[5] K. Pinkerton,et al. Receptor‐mediated tobacco toxicity: cooperation of the Ras/Raf‐1/MEK1/ERK and JAK‐2/STAT‐3 pathways downstream of a7 nicotinic receptor in oral keratinocytes , 2006 .
[6] E. Mazzon,et al. Pyrrolidine dithiocarbamate reduced experimental periodontitis. , 2006, European journal of pharmacology.
[7] Yu-Chao Chang,et al. Regulation of nicotine-induced cyclooxygenase-2 protein expression in human gingival fibroblasts , 2006, Acta Pharmacologica Sinica.
[8] P. Celec. Nuclear factor kappa B--molecular biomedicine: the next generation. , 2004, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[9] Yoshifumi Yamamoto,et al. Platelet‐derived growth factor‐induced arachidonic acid release for enhancement of prostaglandin E2 synthesis in human gingival fibroblasts pretreated with interleukin‐1β , 2004, Journal of cellular biochemistry.
[10] M. Yamazaki,et al. Tumor necrosis factor α (TNF-α)-induced prostaglanding E2 release is mediated by the activation of cyclooxygenase-2 (COX-2)transcription via NFκB in human gingival fibroblasts , 2002, Molecular and Cellular Biochemistry.
[11] M. Yamazaki,et al. Activation of NFκB is necessary for IL-1β-induced cyclooxygenase-2 (COX-2) expression in human gingival fibroblasts , 2000, Molecular and Cellular Biochemistry.
[12] M. Chou,et al. Induction of cyclooxygenase-2 mRNA and protein expression in human gingival fibroblasts stimulated with nicotine. , 2003, Journal of periodontal research.
[13] B. Aggarwal,et al. Cigarette smoke condensate activates nuclear transcription factor-κB through phosphorylation and degradation of IκBα: correlation with induction of cyclooxygenase-2 , 2002 .
[14] S. Stein,et al. Regulation of cytokine production in human gingival fibroblasts following treatment with nicotine and lipopolysaccharide. , 2001, Journal of periodontology.
[15] H. Sugiya,et al. Bradykinin potentiates prostaglandin E(2) release in the human gingival fibroblasts pretreated with interleukin-1beta via Ca(2+) mobilization. , 2000, European journal of pharmacology.
[16] C. Cutler,et al. Clinical benefits of oral irrigation for periodontitis are related to reduction of pro-inflammatory cytokine levels and plaque. , 2000, Journal of clinical periodontology.
[17] S. Eliasson,et al. Exposure to tobacco smoking and periodontal health. , 2000, Journal of clinical periodontology.
[18] R. Garavito,et al. Cyclooxygenases: structural, cellular, and molecular biology. , 2000, Annual review of biochemistry.
[19] N. Sayers,et al. Effects of tobacco products on the attachment and growth of periodontal ligament fibroblasts. , 1999, Journal of periodontology.
[20] M. Takano,et al. Distinct characteristics of transcellular transport between nicotine and tetraethylammonium in LLC-PK1 cells. , 1998, The Journal of pharmacology and experimental therapeutics.
[21] K. Giacomini,et al. Nicotine transport in a human choriocarcinoma cell line (JAR). , 1998, Journal of pharmaceutical sciences.
[22] M. Cattaneo,et al. Mechanisms of mitogen-activated protein kinase activation by nicotine in small-cell lung carcinoma cells. , 1997, The Biochemical journal.
[23] D. Tipton,et al. Effects of nicotine on proliferation and extracellular matrix production of human gingival fibroblasts in vitro. , 1995, Journal of periodontology.
[24] P. De Koninck,et al. Agonist and toxin sensitivities of ACh-evoked currents on neurons expressing multiple nicotinic ACh receptor subunits. , 1995, Journal of neurophysiology.
[25] K. Matsushima,et al. The IL‐1 receptor signaling pathway , 1994, Journal of leukocyte biology.
[26] I. Mandel,et al. Smoke signals: an alert for oral disease. , 1994, Journal of the American Dental Association.
[27] T. Hla,et al. Human cyclooxygenase-2 cDNA. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[28] M. Smith,et al. Effects of NSAIDs on beagle crevicular cyclooxygenase metabolites and periodontal bone loss. , 1992, Journal of periodontal research.
[29] R. Kent,et al. Cigarette smoking in a periodontal practice. , 1992, Journal of periodontology.
[30] P. Hanes,et al. Binding, uptake, and release of nicotine by human gingival fibroblasts. , 1991, Journal of periodontology.
[31] G. R. Imm,et al. A Comparative Study of Human Periodontal Ligament Cells and Gingival Fibroblasts in vitro , 1988, Journal of dental research.
[32] K. Fujimori,et al. Enhanced expression of a glyceraldehyde-3-phosphate dehydrogenase gene in human lung cancers. , 1987, Cancer research.
[33] S. Offenbacher,et al. The use of crevicular fluid prostaglandin E2 levels as a predictor of periodontal attachment loss. , 1986, Journal of periodontal research.
[34] S. Offenbacher,et al. Crevicular fluid prostaglandin E levels as a measure of the periodontal disease status of adult and juvenile periodontitis patients. , 1984, Journal of periodontal research.
[35] M. M. Bradford. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.