Penta-O-galloyl-β-D-glucose attenuates cisplatin-induced nephrotoxicity via reactive oxygen species reduction in renal epithelial cells and enhances antitumor activity in Caki-2 renal cancer cells.
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Soo-Jin Jeong | Sung-Hoon Kim | Hyo-Jung Lee | H. Ryu | K. Ahn | Seung-hoon Choi | Eun-ok Lee | H. Kwon | Min‐ho Lee
[1] Soo-Jin Jeong,et al. Oral administration of penta-O-galloyl-β-D-glucose suppresses triple-negative breast cancer xenograft growth and metastasis in strong association with JAK1-STAT3 inhibition. , 2011, Carcinogenesis.
[2] G. Türk,et al. Lycopene and ellagic acid prevent testicular apoptosis induced by cisplatin. , 2011, Phytomedicine : international journal of phytotherapy and phytopharmacology.
[3] J. Lieske,et al. 1,2,3,4,6-Penta-O-galloyl-beta-D-glucose reduces renal crystallization and oxidative stress in a hyperoxaluric rat model. , 2011, Kidney international.
[4] Soo-Jin Jeong,et al. Reactive oxygen species involved in sulforaphane-induced STAT3 inactivation and apoptosis in DU145 prostate cancer cells , 2010 .
[5] Soo-Jin Jeong,et al. 1,2,3,4,6-Penta-O-galloly-beta-D-glucose suppresses hypoxia-induced accumulation of hypoxia-inducible factor-1α and signaling in LNCaP prostate cancer cells. , 2010, Biological & pharmaceutical bulletin.
[6] Soo-Jin Jeong,et al. Penta-O-galloyl-β-D-glucose induces G1 arrest and DNA replicative S-phase arrest independently of P21 cyclin-dependent kinase inhibitor 1A, P27 cyclin-dependent kinase inhibitor 1B and P53 in human breast cancer cells and is orally active against triple-negative xenograft growth , 2010, Breast Cancer Research.
[7] Soo-Jin Jeong,et al. Paeonol Oxime Inhibits bFGF-Induced Angiogenesis and Reduces VEGF Levels in Fibrosarcoma Cells , 2010, PloS one.
[8] J. Lieske,et al. Decursin prevents cisplatin-induced apoptosis via the enhancement of antioxidant enzymes in human renal epithelial cells. , 2010, Biological & pharmaceutical bulletin.
[9] T. Chou. Drug combination studies and their synergy quantification using the Chou-Talalay method. , 2010, Cancer research.
[10] S. Saleh,et al. Protective Effects of the Angiotensin II Receptor Blocker Losartan on Cisplatin-Induced Kidney Injury , 2009, Chemotherapy.
[11] Soo-Jin Jeong,et al. Protective effect of Bojungbangdocktang on cisplatin-induced cytotoxicity and apoptosis in MCF-10A breast endothelial cells. , 2009, Environmental toxicology and pharmacology.
[12] Sara Khan,et al. Studies on the protective effect of green tea against cisplatin induced nephrotoxicity. , 2009, Pharmacological research.
[13] B. Molitoris,et al. siRNA targeted to p53 attenuates ischemic and cisplatin-induced acute kidney injury. , 2009, Journal of the American Society of Nephrology : JASN.
[14] Sung-Hoon Kim,et al. Anti-Cancer, Anti-Diabetic and Other Pharmacologic and Biological Activities of Penta-Galloyl-Glucose , 2009, Pharmaceutical Research.
[15] Sang Kook Lee,et al. Extract of Prunus persica flesh (PPFE) improves chemotherapeutic efficacy and protects against nephrotoxicity in cisplatin‐treated mice , 2009, Phytotherapy research : PTR.
[16] Jen-kun Lin,et al. Penta-O-galloyl-beta-D-glucose suppresses prostate cancer bone metastasis by transcriptionally repressing EGF-induced MMP-9 expression. , 2009, Journal of agricultural and food chemistry.
[17] J. Lieske,et al. Rhus verniciflua Stokes prevents cisplatin-induced cytotoxicity and reactive oxygen species production in MDCK-I renal cells and intact mice. , 2009, Phytomedicine : international journal of phytotherapy and phytopharmacology.
[18] B. de Camargo,et al. Omission of Day 2 of Antiemetic Medications is a Cost Saving Strategy for Improving Chemotherapy-Induced Nausea and Vomiting Control: Results of a Randomized Phase III Trial , 2009, American journal of clinical oncology.
[19] Cheng Jiang,et al. Penta-1,2,3,4,6-O-galloyl-β-d-glucose induces p53 and inhibits STAT3 in prostate cancer cells in vitro and suppresses prostate xenograft tumor growth in vivo , 2008, Molecular Cancer Therapeutics.
[20] Sang Kook Lee,et al. The extract of Prunus persica flesh (PPFE) attenuates chemotherapy‐induced hepatotoxicity in mice , 2008, Phytotherapy research : PTR.
[21] Y. Kashiwada,et al. Inhibition of P‐glycoprotein function by tannic acid and pentagalloylglucose , 2007, The Journal of pharmacy and pharmacology.
[22] Y. Masubuchi,et al. Toxicological Significance of Mechanism-Based Inactivation of Cytochrome P450 Enzymes by Drugs , 2007, Critical reviews in toxicology.
[23] Sung-Hoon Kim,et al. DMNQ S‐64 Induces Apoptosis via Caspase Activation and Cyclooxygenase‐2 Inhibition in Human Nonsmall Lung Cancer Cells , 2007, Annals of the New York Academy of Sciences.
[24] J. Lieske,et al. Renal cell adaptation to oxalate , 2005, Urological Research.
[25] Y. Surh,et al. Penta-O-galloyl-beta-D-glucose suppresses tumor growth via inhibition of angiogenesis and stimulation of apoptosis: roles of cyclooxygenase-2 and mitogen-activated protein kinase pathways. , 2005, Carcinogenesis.
[26] Y. Gho,et al. 1,2,3,4,6-Penta-O-galloyl-beta-D-glucose blocks endothelial cell growth and tube formation through inhibition of VEGF binding to VEGF receptor. , 2004, Cancer letters.
[27] Jen-kun Lin,et al. Induction of G1 phase arrest in MCF human breast cancer cells by pentagalloylglucose through the down-regulation of CDK4 and CDK2 activities and up-regulation of the CDK inhibitors p27(Kip) and p21(Cip). , 2003, Biochemical pharmacology.
[28] M. Karin,et al. AP-1 as a regulator of cell life and death , 2002, Nature Cell Biology.
[29] S. Lowe,et al. Dissecting p53 tumor suppressor functions in vivo. , 2002, Cancer cell.
[30] K. Chai,et al. In vitro anti-proliferative effect of 1,2,3,4,6-penta-O-galloyl-beta-D-glucose on human hepatocellular carcinoma cell line, SK-HEP-1 cells. , 2001, Cancer letters.
[31] R. Fine,et al. Formation of nuclear Bax/p53 complexes is associated with chemotherapy induced apoptosis , 2000, Oncogene.
[32] E. Wagner,et al. The Mammalian UV Response c-Jun Induction Is Required for Exit from p53-Imposed Growth Arrest , 2000, Cell.
[33] K. Golka,et al. Cultures of exfoliated epithelial cells from different locations of the human urinary tract and the renal tubular system , 2000, Archives of Toxicology.
[34] D. Green,et al. p53 Induces Apoptosis by Caspase Activation through Mitochondrial Cytochrome c Release* , 2000, The Journal of Biological Chemistry.
[35] S. Lin-Shiau,et al. Induction of apoptosis by penta-O-galloyl-beta-D-glucose through activation of caspase-3 in human leukemia HL-60 cells. , 1999, European journal of pharmacology.
[36] K. Kinzler,et al. A model for p53-induced apoptosis , 1997, Nature.
[37] N. Mulder,et al. Modulation of cis-diamminedichloroplatinum(II) resistance: a review. , 1992, British Journal of Cancer.
[38] G. G. Gross,et al. Biosynthesis of gallotannins: formation of polygalloylglucoses by enzymatic acylation of 1,2,3,4,6-penta-O-galloylglucose. , 1990, Archives of biochemistry and biophysics.
[39] M. John,et al. Prevention of cisplatin induced nephrotoxicity by terpenes isolated from Ganoderma lucidum occurring in Southern Parts of India. , 2011, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.
[40] G. Kroemer. [Mitochondrial control of apoptosis]. , 2001, Bulletin de l'Academie nationale de medecine.
[41] H. Lorenz,et al. A rapid and simple method for the isolation of apoptotic DNA fragments. , 1994, Nucleic acids research.
[42] M. Maines. Effect of cis-platinum on heme, drug, and steroid metabolism pathways: possible involvement in nephrotoxicity and infertility. , 1990, Critical reviews in toxicology.
[43] T. F. Patton,et al. Pharmacokinetics of intact cisplatin in plasma. Infusion versus bolus dosing , 1982 .