Transcription factor Egr1 acts as an upstream regulator of β‐catenin signalling through up‐regulation of TCF4 and p300 expression during trans‐differentiation of endometrial carcinoma cells
暂无分享,去创建一个
M. Saegusa | I. Okayasu | J. Watanabe | M. Hamano | M. Hashimura | T. Kuwata | I Okayasu | M Saegusa | M Hashimura | T Kuwata | M Hamano | J Watanabe | M Kawaguchi | M. Kawaguchi | Takeshi Kuwata | Mieko Hamano | Jun Watanabe | Isao Okayasu
[1] Chien-Cheng Chen,et al. Egr‐1 is activated by 17β‐estradiol in MCF‐7 cells by mitogen‐activated protein kinase‐dependent phosphorylation of ELK‐1 , 2004, Journal of cellular biochemistry.
[2] B. Vogelstein,et al. An evaluation of six antibodies for immunohistochemistry of mutant p53 gene product in archival colorectal neoplasms , 1994, The Journal of pathology.
[3] Hideki Yamamoto,et al. Regulation of Wnt signaling by protein-protein interaction and post-translational modifications , 2006, Experimental & Molecular Medicine.
[4] D. Nathans,et al. A gene activated in mouse 3 T 3 cells by serum growth factors encodes a protein with “ zinc finger ” sequences ( transcription factors / serum response element ) , 2009 .
[5] H. Kuramoto,et al. Stimulatory effect of estrogen on the growth of endometrial cancer cells is regulated by cell-cycle regulators , 2007, The Journal of Steroid Biochemistry and Molecular Biology.
[6] M J Bissell,et al. Involvement of extracellular matrix constituents in breast cancer. , 1995, Seminars in cancer biology.
[7] R. Goodman,et al. CBP/p300 in cell growth, transformation, and development. , 2000, Genes & development.
[8] V. Rangnekar,et al. Suppression of PTEN Expression by NF-κB Prevents Apoptosis , 2004, Molecular and Cellular Biology.
[9] R. Nusse,et al. Wnt signaling: a common theme in animal development. , 1997, Genes & development.
[10] H R Herschman,et al. Cloning of tetradecanoyl phorbol ester-induced 'primary response' sequences and their expression in density-arrested Swiss 3T3 cells and a TPA non-proliferative variant. , 1987, Oncogene.
[11] F Borel,et al. Comparison of the DNA binding characteristics of the related zinc finger proteins WT1 and EGR1. , 1998, Biochemistry.
[12] R Grosschedl,et al. Functional interaction of beta-catenin with the transcription factor LEF-1. , 1996, Nature.
[13] M. Nishida. Ishikawa Cells: Opening of In Vitro Hormone Research on Endometrial Carcinoma , 2003 .
[14] Hans Clevers,et al. XTcf-3 Transcription Factor Mediates β-Catenin-Induced Axis Formation in Xenopus Embryos , 1996, Cell.
[15] D. Novosad,et al. Estrogen activates raf-1 kinase and induces expression of Egr-1 in MCF-7 breast cancer cells , 1998, Molecular and Cellular Biochemistry.
[16] M. Saegusa,et al. Changes in Expression of Estrogen Receptors α and β in Relation to Progesterone Receptor and pS2 Status in Normal and Malignant Endometrium , 2000, Japanese journal of cancer research : Gann.
[17] P. Lemaire,et al. Two mouse genes encoding potential transcription factors with identical DNA-binding domains are activated by growth factors in cultured cells. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[18] C. Creutzberg,et al. Nuclear β‐catenin is a molecular feature of type I endometrial carcinoma , 2003 .
[19] L. Lau,et al. A gene activated in mouse 3T3 cells by serum growth factors encodes a protein with "zinc finger" sequences. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[20] E. Adamson,et al. Coactivating factors p300 and CBP are transcriptionally crossregulated by Egr1 in prostate cells, leading to divergent responses. , 2004, Molecular cell.
[21] M. Saegusa,et al. β-Catenin simultaneously induces activation of the p53-p21WAF1 pathway and overexpression of cyclin D1 during squamous differentiation of endometrial carcinoma cells , 2004 .
[22] M. Saegusa,et al. Upregulation of TCF4 expression as a transcriptional target of β-catenin/p300 complexes during trans-differentiation of endometrial carcinoma cells , 2005, Laboratory Investigation.
[23] M. Saegusa,et al. Crosstalk between NF‐κB/p65 and β‐catenin/TCF4/p300 signalling pathways through alterations in GSK‐3β expression during trans‐differentiation of endometrial carcinoma cells , 2007, The Journal of pathology.
[24] D. Herbold,et al. The significance of squamous differentiation in endometrial carcinoma , 1988 .
[25] R. Kurman,et al. The behavior of endometrial hyperplasia. A long-term study of “untreated” hyperplasia in 170 patients , 1986 .
[26] M. Saegusa,et al. A functional role of Cdx2 in beta-catenin signaling during transdifferentiation in endometrial carcinomas. , 2007, Carcinogenesis.
[27] Michael Kühl,et al. Functional interaction of β-catenin with the transcription factor LEF-1 , 1996, Nature.
[28] J. Behrens,et al. Nucleo-cytoplasmic distribution of β-catenin is regulated by retention , 2006, Journal of Cell Science.
[29] M. Saegusa,et al. Up‐regulation of pS2 expression during the development of adenocarcinomas but not squamous cell carcinomas of the uterine cervix, independently of expression of c‐jun or oestrogen and progesterone receptors , 2000, The Journal of pathology.
[30] J. Herman,et al. Abnormalities of the APC/β-catenin pathway in endometrial cancer , 2002, Oncogene.
[31] J. V. Bokhman. Two pathogenetic types of endometrial carcinoma. , 1983, Gynecologic oncology.