The role of shear stress in the generation of rupture-prone vulnerable plaques
暂无分享,去创建一个
P. Serruys | CJ Slager | J. Wentzel | F. Gijsen | A. V. D. Wal | A. V. D. Steen | J. Schuurbiers | CJ Slager | JJ Wentzel | FJH Gijsen | JCH Schuurbiers | AC van der Wal | AFW van der Steen | PW Serruys | J.C.H. Schuurbiers
[1] François Mach,et al. Inflammation and Atherosclerosis , 2004, Herz.
[2] D. Green,et al. Effect of exercise training on endothelium‐derived nitric oxide function in humans , 2004, The Journal of physiology.
[3] G. Getz,et al. Site Specificity of Atherosclerosis: Site-Selective Responses to Atherosclerotic Modulators , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[4] A. Iskandrian,et al. Major risk factors for cardiovascular disease: debunking the "only 50%" myth. , 2003, JAMA.
[5] A. Wahle,et al. Effect of Endothelial Shear Stress on the Progression of Coronary Artery Disease, Vascular Remodeling, and In-Stent Restenosis in Humans: In Vivo 6-Month Follow-Up Study , 2003, Circulation.
[6] P. Serruys,et al. Extension of Increased Atherosclerotic Wall Thickness Into High Shear Stress Regions Is Associated With Loss of Compensatory Remodeling , 2003, Circulation.
[7] E. Falk,et al. Expansive Remodeling Is a Response of the Plaque-Related Vessel Wall in Aortic Roots of ApoE-Deficient Mice: An Experiment of Nature , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[8] C. Kunsch,et al. Laminar Flow Induction of Antioxidant Response Element-mediated Genes in Endothelial Cells , 2003, The Journal of Biological Chemistry.
[9] Gabriel Acevedo-Bolton,et al. Intracardiac fluid forces are an essential epigenetic factor for embryonic cardiogenesis , 2003, Nature.
[10] S. Nakatani,et al. Effect of disease eccentricity on compensatory remodeling of coronary arteries: evidence from intravascular ultrasound before interventions. , 2002, International journal of cardiology.
[11] Renu Virmani,et al. Morphological Predictors of Arterial Remodeling in Coronary Atherosclerosis , 2002, Circulation.
[12] J. Keaney,et al. Are ACE inhibitors a "magic bullet" against oxidative stress? , 2001, Circulation.
[13] R M Nerem,et al. Endothelial cellular response to altered shear stress. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[14] P. Libby,et al. Endothelial function and coronary artery disease , 2001, Current opinion in lipidology.
[15] M. Davies,et al. Relation between coronary artery remodelling (compensatory dilatation) and stenosis in human native coronary arteries , 2001, Heart.
[16] B. Lévy,et al. Adaptive common carotid arteries remodeling after unilateral internal carotid artery occlusion in adult patients. , 2001, Cardiovascular research.
[17] A. Tedgui,et al. Anti-Inflammatory Mechanisms in the Vascular Wall , 2001, Circulation research.
[18] H. Niederegger,et al. Network of Vascular-Associated Dendritic Cells in Intima of Healthy Young Individuals , 2001, Arteriosclerosis, thrombosis, and vascular biology.
[19] Renu Virmani,et al. Healed Plaque Ruptures and Sudden Coronary Death: Evidence That Subclinical Rupture Has a Role in Plaque Progression , 2001, Circulation.
[20] H. C. Stary. Lipid and macrophage accumulations in arteries of children and the development of atherosclerosis. , 2000, The American journal of clinical nutrition.
[21] P. Doriot,et al. In‐vivo measurements of wall shear stress in human coronary arteries , 2000, Coronary artery disease.
[22] P. Serruys,et al. True 3-dimensional reconstruction of coronary arteries in patients by fusion of angiography and IVUS (ANGUS) and its quantitative validation. , 2000, Circulation.
[23] O. Tricot,et al. Relation between endothelial cell apoptosis and blood flow direction in human atherosclerotic plaques. , 2000, Circulation.
[24] R. Virmani,et al. Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[25] S E Nissen,et al. Extent and direction of arterial remodeling in stable versus unstable coronary syndromes : an intravascular ultrasound study. , 2000, Circulation.
[26] S. Alper,et al. Hemodynamic shear stress and its role in atherosclerosis. , 1999, JAMA.
[27] T Suzuki,et al. Process of progression of coronary artery lesions from mild or moderate stenosis to moderate or severe stenosis: A study based on four serial coronary arteriograms per year. , 1999, Circulation.
[28] P J de Feyter,et al. Angioscopic complex lesions are predominantly compensatory enlarged: an angioscopy and intracoronary ultrasound study. , 1999, Cardiovascular research.
[29] B Hillen,et al. Relation of arterial geometry to luminal narrowing and histologic markers for plaque vulnerability: the remodeling paradox. , 1998, Journal of the American College of Cardiology.
[30] C von Birgelen,et al. Atherosclerotic coronary lesions with inadequate compensatory enlargement have smaller plaque and vessel volumes: observations with three dimensional intravascular ultrasound in vivo , 1998, Heart.
[31] T. V. van Leeuwen,et al. Compensatory enlargement in coronary and femoral arteries is related to neither the extent of plaque-free vessel wall nor lesion eccentricity. A postmortem study. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[32] P. Serruys,et al. Evaluation of endothelial shear stress and 3D geometry as factors determining the development of atherosclerosis and remodeling in human coronary arteries in vivo. Combining 3D reconstruction from angiography and IVUS (ANGUS) with computational fluid dynamics. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[33] K. Pritchard,et al. Suppression of angiotensin-converting enzyme expression and activity by shear stress. , 1997, Circulation research.
[34] M. Nehls,et al. Shear stress inhibits apoptosis of human endothelial cells , 1996, FEBS letters.
[35] R M Nerem,et al. Shear stress modulates expression of Cu/Zn superoxide dismutase in human aortic endothelial cells. , 1996, Circulation research.
[36] P. Boutouyrie,et al. Remodeling of the radial artery in response to a chronic increase in shear stress. , 1996, Hypertension.
[37] S. Izumo,et al. Control of endothelial cell gene expression by flow. , 1995, Journal of biomechanics.
[38] W D Wagner,et al. A definition of advanced types of atherosclerotic lesions and a histological classification of atherosclerosis. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association. , 1995, Arteriosclerosis, thrombosis, and vascular biology.
[39] V. Fuster,et al. Coronary plaque disruption. , 1995, Circulation.
[40] W D Wagner,et al. A definition of initial, fatty streak, and intermediate lesions of atherosclerosis. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[41] A. Becker,et al. Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. , 1994, Circulation.
[42] R. Kamm,et al. Distribution of Circumferential Stress in Ruptured and Stable Atherosclerotic Lesions A Structural Analysis With Histopathological Correlation , 1993, Circulation.
[43] M. Ferguson,et al. Carotid Artery Intraplaque Hemorrhage and Stenotic Velocity , 1993, Stroke.
[44] R. Busse,et al. Role of Endothelium‐Derived Bradykinin in the Control of Vascular Tone , 1992, Journal of cardiovascular pharmacology.
[45] D H Blankenhorn,et al. A definition of the intima of human arteries and of its atherosclerosis-prone regions. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association. , 1992, Circulation.
[46] K. Bürrig,et al. The endothelium of advanced arteriosclerotic plaques in humans. , 1991, Arteriosclerosis and thrombosis : a journal of vascular biology.
[47] G. V. R. Born,et al. INFLUENCE OF PLAQUE CONFIGURATION AND STRESS DISTRIBUTION ON FISSURING OF CORONARY ATHEROSCLEROTIC PLAQUES , 1989, The Lancet.
[48] G. Hansson,et al. Detection of activated T lymphocytes in the human atherosclerotic plaque. , 1989, The American journal of pathology.
[49] B L Langille,et al. Adaptations of carotid arteries of young and mature rabbits to reduced carotid blood flow. , 1989, The American journal of physiology.
[50] V. Fuster,et al. Angiographic progression of coronary artery disease and the development of myocardial infarction. , 1988, Journal of the American College of Cardiology.
[51] C. Zarins,et al. Compensatory enlargement of human atherosclerotic coronary arteries. , 1987, The New England journal of medicine.
[52] S Glagov,et al. Shear stress regulation of artery lumen diameter in experimental atherogenesis. , 1987, Journal of vascular surgery.
[53] M J Davies,et al. Thrombosis and acute coronary-artery lesions in sudden cardiac ischemic death. , 1984, The New England journal of medicine.
[54] T Togawa,et al. Adaptive regulation of wall shear stress to flow change in the canine carotid artery. , 1980, The American journal of physiology.
[55] F J Walburn,et al. A constitutive equation for whole human blood. , 1976, Biorheology.
[56] C. Velican,et al. Coronary arteries in children up to the age of ten years II. Intimal thickening and its role in atherosclerotic involvement. , 1976, Medecine interne.
[57] Cocks Dp,et al. Letter: A state-registered abortionists? , 1975 .
[58] G. Mann,et al. Atherosclerosis in the Masai. , 1972, American journal of epidemiology.
[59] G. Mann,et al. Physical fitness and immunity to heart-disease in Masai. , 1965, Lancet.