Regional brain cytochrome oxidase activity in β-amyloid precursor protein transgenic mice with the swedish mutation
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[1] M. Staufenbiel,et al. Spatial learning, exploration, anxiety, and motor coordination in female APP23 transgenic mice with the Swedish mutation , 2002, Brain Research.
[2] M. Staufenbiel,et al. Amyloid-Associated Neuron Loss and Gliogenesis in the Neocortex of Amyloid Precursor Protein Transgenic Mice , 2002, The Journal of Neuroscience.
[3] J. Land,et al. β‐Amyloid inhibits integrated mitochondrial respiration and key enzyme activities , 2001, Journal of neurochemistry.
[4] F. Gonzalez-Lima,et al. Reduced corpus callosum, fornix and hippocampus in PDAPP transgenic mouse model of Alzheimer's disease , 2001, Neuroreport.
[5] George Paxinos,et al. The Mouse Brain in Stereotaxic Coordinates , 2001 .
[6] P. Ince,et al. Mitochondrial enzyme-deficient hippocampal neurons and choroidal cells in AD , 2001, Neurology.
[7] A. Convit,et al. Hippocampal formation glucose metabolism and volume losses in MCI and AD , 2001, Neurobiology of Aging.
[8] F Gonzalez-Lima,et al. Energy Hypometabolism in Posterior Cingulate Cortex of Alzheimer's Patients: Superficial Laminar Cytochrome Oxidase Associated with Disease Duration , 2001, The Journal of Neuroscience.
[9] M. Staufenbiel,et al. Spontaneous Hemorrhagic Stroke in a Mouse Model of Cerebral Amyloid Angiopathy , 2001, The Journal of Neuroscience.
[10] J. Parks,et al. Neurotoxic Aβ peptides increase oxidative stress in vivo through NMDA‐receptor and nitric‐oxide‐synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitro , 2001, Journal of neurochemistry.
[11] G. V. Van Hoesen,et al. Selective pathological changes of the periaqueductal gray matter in Alzheimer's disease , 2000, Annals of neurology.
[12] G. Alexander,et al. The neurometabolic landscape of cognitive decline: in vivo studies with positron emission tomography in Alzheimer's disease. , 2000, International journal of psychophysiology : official journal of the International Organization of Psychophysiology.
[13] F. Leuven,et al. Single and multiple transgenic mice as models for Alzheimer’s disease , 2000, Progress in Neurobiology.
[14] Frederik Barkhof,et al. Unbiased whole-brain analysis of gray matter loss in Alzheimer's disease , 2000, Neuroscience Letters.
[15] H. Möller,et al. A selective defect of cytochrome c oxidase is present in brain of Alzheimer disease patients , 2000, Neurobiology of Aging.
[16] A. Nunomura,et al. Mitochondrial abnormalities in Alzheimer disease , 2000, Neurobiology of Aging.
[17] Keith A. Johnson,et al. Perfusion abnormalities in prodromal AD , 2000, Neurobiology of Aging.
[18] C. Czech,et al. Presenilins and Alzheimer’s disease: biological functions and pathogenic mechanisms , 2000, Progress in Neurobiology.
[19] T. Town,et al. Soluble β-amyloid peptides mediate vasoactivity via activation of a pro-inflammatory pathway , 2000, Neurobiology of Aging.
[20] J. C. Torre,et al. Critically attained threshold of cerebral hypoperfusion: the CATCH hypothesis of Alzheimer’s pathogenesis , 2000, Neurobiology of Aging.
[21] P. Adlard,et al. The cause of neuronal degeneration in Alzheimer's disease , 2000, Progress in Neurobiology.
[22] Chantal Mathis,et al. Early regional cerebral glucose hypometabolism in transgenic mice overexpressing the V717F β-amyloid precursor protein , 1999, Neuroscience Letters.
[23] P. Lantos,et al. β‐Amyloid deposition and neurofibrillary tangle formation in the olfactory bulb in ageing and Alzheimer’s disease , 1999 .
[24] S. Rapoport,et al. Functional Brain Imaging in the Resting State and during Activation in Alzheimer's Disease: Implications for Disease Mechanisms Involving Oxidative Phosphorylation , 1999, Annals of the New York Academy of Sciences.
[25] M. Frotscher,et al. Cerebral Amyloid Induces Aberrant Axonal Sprouting and Ectopic Terminal Formation in Amyloid Precursor Protein Transgenic Mice , 1999, The Journal of Neuroscience.
[26] M. Wong-Riley,et al. Expression and regulation of NMDA receptor subunit R1 and neuronal nitric oxide synthase in cortical neuronal cultures: Correlation with cytochrome oxidase , 1999, Journal of neurocytology.
[27] D. Selkoe,et al. Translating cell biology into therapeutic advances in Alzheimer's disease , 1999, Nature.
[28] Chris I. De Zeeuw,et al. GATA-3 Is Involved in the Development of Serotonergic Neurons in the Caudal Raphe Nuclei , 1999, The Journal of Neuroscience.
[29] B. Sommer,et al. Association of microglia with amyloid plaques in brains of APP23 transgenic mice. , 1999, The American journal of pathology.
[30] D. Dickson. Microglia in Alzheimer's disease and transgenic models. How close the fit? , 1999, The American journal of pathology.
[31] Jae-Young Koh,et al. Histochemically Reactive Zinc in Plaques of the Swedish Mutant β-Amyloid Precursor Protein Transgenic Mice , 1999, The Journal of Neuroscience.
[32] Sarah Tomlin,et al. Microtechnology: Laying it on thick , 1999, Nature.
[33] M. Esiri,et al. Mitochondrial enzyme expression in the hippocampus in relation to Alzheimer-type pathology , 1999, Acta Neuropathologica.
[34] Veerle Baekelandt,et al. Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain* , 1999, The Journal of Biological Chemistry.
[35] S. Kish,et al. Decreased Brain Protein Levels of Cytochrome Oxidase Subunits in Alzheimer's Disease and in Hereditary Spinocerebella Ataxia Disorders , 1999, Journal of neurochemistry.
[36] D. Borchelt,et al. SOD1 rescues cerebral endothelial dysfunction in mice overexpressing amyloid precursor protein , 1999, Nature Neuroscience.
[37] D. Cassarino,et al. An evaluation of the role of mitochondria in neurodegenerative diseases: mitochondrial mutations and oxidative pathology, protective nuclear responses, and cell death in neurodegeneration , 1999, Brain Research Reviews.
[38] T. Rosahl,et al. Transgenic animals relevant to Alzheimer’s disease , 1999, Neuropharmacology.
[39] B. Sommer,et al. Neuron loss in APP transgenic mice , 1998, Nature.
[40] C. Jack,et al. Rate of medial temporal lobe atrophy in typical aging and Alzheimer's disease , 1998, Neurology.
[41] D. Sirinathsinghji. Transgenic mouse models of Alzheimer's disease. , 1998, Biochemical Society transactions.
[42] R. Lalonde,et al. Regional brain variations of cytochrome oxidase activity and motor coordination in Lurcher mutant mice , 1998, Experimental Brain Research.
[43] G. Alexander,et al. Regional glucose metabolic abnormalities are not the result of atrophy in Alzheimer's disease , 1998, Neurology.
[44] M. Tabaton,et al. Amyloid‐β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative Stress , 1998, Journal of neurochemistry.
[45] B. Sommer,et al. Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[46] R. Davis,et al. Cybrids in Alzheimer's disease: A cellular model of the disease? , 1997, Neurology.
[47] Rudolph E Tanzi,et al. Presenilins and Alzheimer's disease , 1997, Current Opinion in Neurobiology.
[48] B. Hyman,et al. APPSW Transgenic Mice Develop Age‐related Aβ Deposits and Neuropil Abnormalities, but no Neuronal Loss in CA1 , 1997, Journal of neuropathology and experimental neurology.
[49] S. Younkin,et al. Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic Mice , 1996, Science.
[50] D. Borchelt,et al. Inherited Neurodegenerative Diseases and Transgenic Models , 1996, Brain pathology.
[51] L. Mucke,et al. Comparison of Neurodegenerative Pathology in Transgenic Mice Overexpressing V717F β-Amyloid Precursor Protein and Alzheimer’s Disease , 1996, The Journal of Neuroscience.
[52] D. Premkumar,et al. Molecular aspects of inflammatory and immune responses in Alzheimer's disease , 1996, Neurobiology of Aging.
[53] D. Borchelt,et al. Age-related CNS disorder and early death in transgenic FVB/N mice overexpressing Alzheimer amyloid precursor proteins , 1995, Neuron.
[54] P. Chagnon,et al. Distribution of brain cytochrome oxidase activity in various neurodegenerative diseases. , 1995, Neuroreport.
[55] J. Parks,et al. Cytochrome C Oxidase in Alzheimer's Disease Brain , 1995, Neurology.
[56] L. Mucke,et al. Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein , 1995, Nature.
[57] Dirk Jones,et al. Quantitative mapping of cytovhrome oxidase activity in the central auditory system of the gerbil: a study with calibrated activity standards and metal-intensified histochemistry , 1994, Brain Research.
[58] T. Iwatsubo,et al. Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43) , 1994, Neuron.
[59] H. Reichmann,et al. Electron transport chain defects in Alzheimer's disease. , 1994, Neurology.
[60] H. T. ter Laak,et al. The Olfactory Bulb in Alzheimer Disease: A Morphologic Study of Neuron Loss, Tangles, and Senile Plaques in Relation to Olfaction , 1994, Alzheimer disease and associated disorders.
[61] P. Greengard,et al. Altered processing of Alzheimer amyloid precursor protein in response to neuronal degeneration. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[62] M. Wong-Riley,et al. Mitochondrial and nuclear gene expression for cytochrome oxidase subunits are disproportionately regulated by functional activity in neurons , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[63] J. Nobrega,et al. Brain Cytochrome Oxidase in Alzheimer's Disease , 1992, Journal of neurochemistry.
[64] G Blomqvist,et al. Brain metabolism in Alzheimer's dementia: studies of 11C-deoxyglucose accumulation, CSF monoamine metabolites and neuropsychological test performance in patients and healthy subjects. , 1991, Journal of neurology, neurosurgery, and psychiatry.
[65] C. Filley,et al. Cytochrome oxidase deficiency in Alzheimer's disease , 1990, Neurology.
[66] M. Wong-Riley. Cytochrome oxidase: an endogenous metabolic marker for neuronal activity , 1989, Trends in Neurosciences.
[67] R. Terry,et al. Senile dementia of the Alzheimer type , 1983, Annals of neurology.
[68] Michael W. Miller,et al. Cortical connections between rat cingulate cortex and visual, motor, and postsubicular cortices , 1983, The Journal of comparative neurology.
[69] M. Wong-Riley. Changes in the visual system of monocularly sutured or enucleated cats demonstrable with cytochrome oxidase histochemistry , 1979, Brain Research.
[70] O. Lindvall,et al. Subcortical projections to the prefrontal cortex in the rat as revealed by the horseradish peroxidase technique , 1978, Neuroscience.
[71] H. Hess,et al. Ultramicrospectrophotometric determination of cytochrome oxidase for quantitative histochemistry. , 1953, The Journal of biological chemistry.
[72] H. Braak,et al. The autonomic higher order processing nuclei of the lower brain stem are among the early targets of the Alzheimer's disease-related cytoskeletal pathology , 2001, Acta Neuropathologica.
[73] M. Beal,et al. Mitochondria and free radicals in neurodegenerative diseases , 1997 .
[74] G. Paxinos. The Rat nervous system , 1985 .