Calcitonin may be a useful therapeutic agent for osteoclastogenesis syndromes involving premature eruption of the tooth.

[1]  C. Gardner.  Morphological analysis of osteoclastogenesis induced by RANKL in mouse bone marrow cell cultures , 2007, Cell biology international.

[2]  R. Chiusaroli,et al.  Calpain Is Required for Normal Osteoclast Function and Is Down-regulated by Calcitonin* , 2006, Journal of Biological Chemistry.

[3]  T. Chambers,et al.  Murine osteoclast formation and function: differential regulation by humoral agents. , 2006, Endocrinology.

[4]  Kozo Nakamura,et al.  Role of RANKL in physiological and pathological bone resorption and therapeutics targeting the RANKL–RANK signaling system , 2005, Immunological reviews.

[5]  Hiroaki Nakamura,et al.  Distribution of actin filaments, non-muscle myosin, M-Ras, and extracellular signal-regulated kinase (ERK) in osteoclasts after calcitonin administration. , 2005, Archives of histology and cytology.

[6]  Hiroshi Takayanagi,et al.  Mechanistic insight into osteoclast differentiation in osteoimmunology , 2005, Journal of Molecular Medicine.

[7]  A. Galanos,et al.  Effect of intermittent administration of 200 IU intranasal salmon calcitonin and low doses of 1α(OH) vitamin D3 on bone mineral density of the lumbar spine and hip region and biochemical bone markers in women with postmenopausal osteoporosis: a pilot study , 2005, Clinical Rheumatology.

[8]  S. Yao,et al.  In vivo expression of RANKL in the rat dental follicle as determined by laser capture microdissection. , 2004, Archives of oral biology.

[9]  J. Heinrich,et al.  CSF-1, RANKL and OPG regulate osteoclastogenesis during murine tooth eruption. , 2004, Archives of oral biology.

[10]  M. Pogrel.  Calcitonin therapy for central giant cell granuloma. , 2003, Journal of oral and maxillofacial surgery : official journal of the American Association of Oral and Maxillofacial Surgeons.

[11]  Yongwon Choi,et al.  The Role of RANKL (TRANCE/TNFSF11), a Tumor Necrosis Factor Family Member, in Skeletal Development: Effects of Gene Knockout and Transgenic Rescue , 2003, Connective tissue research.

[12]  G. Wise,et al.  Cellular, Molecular, and Genetic Determinants of Tooth Eruption on Behalf Of: International and American Associations for Dental Research , 2002 .

[13]  S. Khosla,et al.  Minireview: the OPG/RANKL/RANK system. , 2001, Endocrinology.

[14]  G. Wise,et al.  Osteoprotegerin and Osteoclast Differentiation Factor in Tooth Eruption , 2000, Journal of dental research.

[15]  T. Martin,et al.  Osteoprotegerin produced by osteoblasts is an important regulator in osteoclast development and function. , 2000, Endocrinology.

[16]  S. Takeshita,et al.  Identification and Characterization of the New Osteoclast Progenitor with Macrophage Phenotypes Being Able to Differentiate into Mature Osteoclasts , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[17]  J. Reginster,et al.  Evidence-based guidelines for the prevention and treatment of glucocorticoid-induced osteoporosis: a consensus document of the Belgian Bone Club , 2005, Osteoporosis International.

[18]  N. Udagawa,et al.  Cells of Bone , 2002 .

[19]  John P. Bilezikian,et al.  Principles of Bone Biology , 1996 .