The MAO Inhibitor Tranylcypromine Alters LPS- and Aβ-Mediated Neuroinflammatory Responses in Wild-type Mice and a Mouse Model of AD
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Hyongjun Jeon | H. Hoe | Jin-Hee Park | Young-Ho Lee | Ji-soo Lee | Y. Jeong | Yuxi Lin | S. Jeon | Hyunhee Park | Kyung-Min Han | Hyun-ju Lee | Yu Gyung Kim | Ji-Soo Lee | HyunHee Park | Hyun-ju Lee | Yuxi Lin
[1] Wei Jiang,et al. Antidepressants of different classes cause distinct behavioral and brain pro- and anti-inflammatory changes in mice submitted to an inflammatory model of depression. , 2020, Journal of affective disorders.
[2] H. Hoe,et al. Dasatinib regulates LPS-induced microglial and astrocytic neuroinflammatory responses by inhibiting AKT/STAT3 signaling , 2019, Journal of Neuroinflammation.
[3] Xingkai Zhang,et al. The effects of tranylcypromine on osteoclastogenesis in vitro and in vivo , 2019, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[4] C. Popoiu,et al. MONOAMINE OXIDASE IS A SOURCE OF OXIDATIVE STRESS IN OBESE PATIENTS WITH CHRONIC INFLAMMATION. , 2019, Canadian journal of physiology and pharmacology.
[5] C. Popoiu,et al. Monoamine Oxidase-Related Vascular Oxidative Stress in Diseases Associated with Inflammatory Burden , 2019, Oxidative medicine and cellular longevity.
[6] Ashwini M. Londhe,et al. Newly developed reversible MAO-B inhibitor circumvents the shortcomings of irreversible inhibitors in Alzheimer’s disease , 2019, Science Advances.
[7] J. Schütte,et al. LSD1 inhibition by tranylcypromine derivatives interferes with GFI1-mediated repression of PU.1 target genes and induces differentiation in AML , 2019, Leukemia.
[8] T. Deierborg,et al. Microglia in Neurological Diseases: A Road Map to Brain-Disease Dependent-Inflammatory Response , 2018, Front. Cell. Neurosci..
[9] H. Choi,et al. The small molecule CA140 inhibits the neuroinflammatory response in wild-type mice and a mouse model of AD , 2018, Journal of Neuroinflammation.
[10] Jeongyeon Kim,et al. Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice , 2018, Journal of Neuroinflammation.
[11] D. Muntean,et al. Monoamine oxidase inhibition improves vascular function and reduces oxidative stress in rats with lipopolysaccharide-induced inflammation. , 2018, General physiology and biophysics.
[12] K. Biswas,et al. Regulation of monoamine oxidase A (MAO-A) expression, activity, and function in IL-13–stimulated monocytes and A549 lung carcinoma cells , 2018, The Journal of Biological Chemistry.
[13] Yu-bo Zhou,et al. Tying up tranylcypromine: Novel selective histone lysine specific demethylase 1 (LSD1) inhibitors. , 2017, European journal of medicinal chemistry.
[14] D. Westaway,et al. Role of microglial amylin receptors in mediating beta amyloid (Aβ)-induced inflammation , 2017, Journal of Neuroinflammation.
[15] P. Schlattmann,et al. Tranylcypromine in mind (Part II): Review of clinical pharmacology and meta-analysis of controlled studies in depression , 2017, European Neuropsychopharmacology.
[16] L. Schneider,et al. Defeating Alzheimer's disease and other dementias: a priority for European science and society , 2016, The Lancet Neurology.
[17] F. Drago,et al. Neuroprotective effects of the monoamine oxidase inhibitor tranylcypromine and its amide derivatives against Aβ(1-42)-induced toxicity. , 2015, European journal of pharmacology.
[18] W. V. van Gool,et al. Systemic inflammation and microglial activation: systematic review of animal experiments , 2015, Journal of Neuroinflammation.
[19] O. Garaschuk,et al. Neuroinflammation in Alzheimer's disease , 2015, The Lancet Neurology.
[20] R. Cacabelos,et al. Epigenetic drug discovery for Alzheimer’s disease , 2014, Expert opinion on drug discovery.
[21] C. Cunningham,et al. Correction: A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1β, TNF-α and IL-6 Challenges in C57BL/6 Mice , 2013, PLoS ONE.
[22] C. Cunningham,et al. A Systematic Analysis of the Peripheral and CNS Effects of Systemic LPS, IL-1Β, TNF-α and IL-6 Challenges in C57BL/6 Mice , 2013, PloS one.
[23] H. Nakayama,et al. Resistance of the golden hamster to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-neurotoxicity is not only related with low levels of cerebral monoamine oxidase-B. , 2013, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.
[24] Yung-Hyun Choi,et al. Caffeine suppresses lipopolysaccharide-stimulated BV2 microglial cells by suppressing Akt-mediated NF-κB activation and ERK phosphorylation. , 2012, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[25] Xingshu Li,et al. Multitarget-directed benzylideneindanone derivatives: anti-β-amyloid (Aβ) aggregation, antioxidant, metal chelation, and monoamine oxidase B (MAO-B) inhibition properties against Alzheimer's disease. , 2012, Journal of medicinal chemistry.
[26] Phenelzine (Monoamine Oxidase Inhibitor) Increases Production of Nitric Oxide and Proinflammatory Cytokines via the NF-κB Pathway in Lipopolysaccharide-Activated Microglia Cells , 2012, Neurochemical Research.
[27] M. Youdim,et al. From antioxidant chelators to site-activated multi-target chelators targeting hypoxia inducing factor, beta-amyloid, acetylcholinesterase and monoamine oxidase A/B. , 2012, Mini reviews in medicinal chemistry.
[28] S. Rose-John,et al. IL-6 Trans-Signaling Modulates TLR4-Dependent Inflammatory Responses via STAT3 , 2011, The Journal of Immunology.
[29] D. Selkoe. Alzheimer's disease. , 2011, Cold Spring Harbor perspectives in biology.
[30] E. Obuchowicz,et al. Moclobemide exerts anti-inflammatory effect in lipopolysaccharide-activated primary mixed glial cell culture , 2010, Naunyn-Schmiedeberg's Archives of Pharmacology.
[31] M. Youdim,et al. Site-activated chelators targeting acetylcholinesterase and monoamine oxidase for Alzheimer's therapy. , 2010, ACS chemical biology.
[32] J. Belluzzi,et al. Age influences the effects of nicotine and monoamine oxidase inhibition on mood-related behaviors in rats , 2010, Psychopharmacology.
[33] J. Changeux,et al. Regional changes in the cholinergic system in mice lacking monoamine oxidase A , 2009, Brain Research Bulletin.
[34] B. Gorzalka,et al. Differential effects of the antidepressants tranylcypromine and fluoxetine on limbic cannabinoid receptor binding and endocannabinoid contents , 2008, Journal of Neural Transmission.
[35] David M. Gooden,et al. Facile synthesis of substituted trans-2-arylcyclopropylamine inhibitors of the human histone demethylase LSD1 and monoamine oxidases A and B. , 2008, Bioorganic & medicinal chemistry letters.
[36] W. Yeh,et al. LPS/TLR4 signal transduction pathway. , 2008, Cytokine.
[37] D. Bylund,et al. Juvenile rats in the forced-swim test model the human response to antidepressant treatment for pediatric depression , 2008, Psychopharmacology.
[38] J. Belluzzi,et al. Monoamine Oxidase Inhibitors Allow Locomotor and Rewarding Responses to Nicotine , 2006, Neuropsychopharmacology.
[39] M. Youdim,et al. Monoamine oxidase: isoforms and inhibitors in Parkinson's disease and depressive illness , 2006, British journal of pharmacology.
[40] Y. Akao,et al. Involvement of type A monoamine oxidase in neurodegeneration: regulation of mitochondrial signaling leading to cell death or neuroprotection. , 2006, Journal of neural transmission. Supplementum.
[41] J. Koenigsknecht-Talboo,et al. Microglial Phagocytosis Induced by Fibrillar β-Amyloid and IgGs Are Differentially Regulated by Proinflammatory Cytokines , 2005, The Journal of Neuroscience.
[42] R. El Bekay,et al. A new role for monoamine oxidases in the modulation of macrophage‐inducible nitric oxide synthase gene expression , 2004, Journal of leukocyte biology.
[43] Rolf Rossaint,et al. Activation of STAT3 by IL-6 and IL-10 in Primary Human Macrophages Is Differentially Modulated by Suppressor of Cytokine Signaling 3 1 , 2003, The Journal of Immunology.
[44] A. Parini,et al. Monoamine oxidase B induces ERK-dependent cell mitogenesis by hydrogen peroxide generation. , 2000, Biochemical and biophysical research communications.
[45] S. L. Yates,et al. Amyloid β and Amylin Fibrils Induce Increases in Proinflammatory Cytokine and Chemokine Production by THP‐1 Cells and Murine Microglia , 2000, Journal of neurochemistry.
[46] E. Nowakowska,et al. [Inhibitory monoamine oxidases of the new generation]. , 1997, Polski merkuriusz lekarski : organ Polskiego Towarzystwa Lekarskiego.
[47] J. Cummings. Lewy Body Diseases with Dementia - Pathophysiology and Treatment , 1995, Brain and Cognition.
[48] D. McManus,et al. Chronic administration of the antidepressant phenelzine and its N-acetyl analogue: effects on GABAergic function. , 1994, Journal of neural transmission. Supplementum.
[49] M. Youdim,et al. Selective MAO-A and B inhibitors, radical scavengers and nitric oxide synthase inhibitors in Parkinson's disease. , 1994, Life sciences.
[50] S. Belliard,et al. Monoamine oxidase inhibitors, cognitive functions and neurodegenerative diseases. , 1994, Journal of neural transmission. Supplementum.
[51] K. Kato,et al. Transfection of CD14 into 70Z/3 cells dramatically enhances the sensitivity to complexes of lipopolysaccharide (LPS) and LPS binding protein , 1992, The Journal of experimental medicine.
[52] S. Ross,et al. Inhibition of monoamine oxidase in monoaminergic neurones in the rat brain by irreversible inhibitors. , 1986, Biochemical pharmacology.
[53] J. Mann,et al. Studies of selective and reversible monoamine oxidase inhibitors. , 1984, The Journal of clinical psychiatry.