RASSF6 is a novel member of the RASSF family of tumor suppressors

[1]  L. Hesson,et al.  The RASSF1A Tumor Suppressor Activates Bax via MOAP-1* , 2006, Journal of Biological Chemistry.

[2]  R. Bernards,et al.  Involvement of MINK, a Ste20 family kinase, in Ras oncogene-induced growth arrest in human ovarian surface epithelial cells. , 2005, Molecular cell.

[3]  P. Openshaw,et al.  BRONCHIOLITIS , 2005, The Lancet.

[4]  Kohzoh Imai,et al.  The Ras effector RASSF2 is a novel tumor-suppressor gene in human colorectal cancer. , 2005, Gastroenterology.

[5]  B. Neel,et al.  The tumor suppressor RASSF1A and MAP-1 link death receptor signaling to Bax conformational change and cell death. , 2005, Molecular cell.

[6]  F. Latif,et al.  Role of the Ras-association domain family 1 tumor suppressor gene in human cancers. , 2005, Cancer research.

[7]  M. You,et al.  Tumor susceptibility of Rassf1a knockout mice. , 2005, Cancer research.

[8]  L. Hesson,et al.  RASSF4/AD037 Is a Potential Ras Effector/Tumor Suppressor of the RASSF Family , 2004, Cancer Research.

[9]  S. Barik,et al.  Activation of cytokines and NF-kappa B in corneal epithelial cells infected by respiratory syncytial virus: potential relevance in ocular inflammation and respiratory infection , 2004, BMC Microbiology.

[10]  G. Clark,et al.  A Role for the RASSF1A Tumor Suppressor in the Regulation of Tubulin Polymerization and Genomic Stability , 2004, Cancer Research.

[11]  Bertil Johansson,et al.  Genome characteristics of primary carcinomas, local recurrences, carcinomatoses, and liver metastases from colorectal cancer patients , 2004, Molecular Cancer.

[12]  Dominic P. Kwiatkowski,et al.  Haplotype mapping of the bronchiolitis susceptibility locus near IL8 , 2004, Human Genetics.

[13]  Channing J Der,et al.  The dark side of Ras: regulation of apoptosis , 2003, Oncogene.

[14]  C. Larsson,et al.  The t(1;3) breakpoint-spanning genes LSAMP and NORE1 are involved in clear cell renal cell carcinomas. , 2003, Cancer cell.

[15]  G. Clark,et al.  RASSF2 Is a Novel K-Ras-specific Effector and Potential Tumor Suppressor* , 2003, Journal of Biological Chemistry.

[16]  G. Clark,et al.  The Pro-apoptotic Ras Effector Nore1 May Serve as a Ras-regulated Tumor Suppressor in the Lung* , 2003, Journal of Biological Chemistry.

[17]  M. Barbacid,et al.  RAS oncogenes: the first 30 years , 2003, Nature Reviews Cancer.

[18]  G. Clark,et al.  Rig is a novel Ras-related protein and potential neural tumor suppressor , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[19]  G. Pfeifer,et al.  Methylation of the RASSF1A Gene in Human Cancers , 2002, Biological chemistry.

[20]  J. Avruch,et al.  RASSF3 and NORE1: identification and cloning of two human homologues of the putative tumor suppressor gene RASSF1 , 2002, Oncogene.

[21]  R. Buchsbaum,et al.  Cell Signaling: Life or Death Decisions of Ras Proteins , 2002, Current Biology.

[22]  R. Xavier,et al.  Identification of a Novel Ras-Regulated Proapoptotic Pathway , 2002, Current Biology.

[23]  J. Minna,et al.  Epigenetic inactivation of RASSF1A in lung and breast cancers and malignant phenotype suppression. , 2001, Journal of the National Cancer Institute.

[24]  A. Bell,et al.  Ras Uses the Novel Tumor Suppressor RASSF1 as an Effector to Mediate Apoptosis* , 2000, The Journal of Biological Chemistry.

[25]  C. Der,et al.  Elucidation of Binding Determinants and Functional Consequences of Ras/Raf-Cysteine-rich Domain Interactions* , 2000, The Journal of Biological Chemistry.

[26]  Chun Xing Li,et al.  Epigenetic inactivation of a RAS association domain family protein from the lung tumour suppressor locus 3p21.3 , 2000, Nature Genetics.

[27]  C. Der,et al.  Understanding Ras: 'it ain't over 'til it's over'. , 2000, Trends in cell biology.

[28]  A. Balmain,et al.  Integration of positive and negative growth signals during ras pathway activation in vivo. , 2000, Current opinion in genetics & development.

[29]  G. Evan,et al.  Traps to catch unwary oncogenes. , 1998, Trends in genetics : TIG.

[30]  M. Malumbres,et al.  RAS pathways to cell cycle control and cell transformation. , 1998, Frontiers in bioscience : a journal and virtual library.

[31]  C. Y. Wang,et al.  Requirement of NF-kappaB activation to suppress p53-independent apoptosis induced by oncogenic Ras. , 1997, Science.

[32]  S. Lowe,et al.  Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a , 1997, Cell.

[33]  Chris P. Ponting,et al.  A novel family ofras-binding domains , 1996 .

[34]  J. Pessin,et al.  Interactions between Src Homology (SH) 2/SH3 Adapter Proteins and the Guanylnucleotide Exchange Factor SOS Are Differentially Regulated by Insulin and Epidermal Growth Factor* , 1996, The Journal of Biological Chemistry.

[35]  T. Kataoka,et al.  Post-translational Modification of H-Ras Is Required for Activation of, but Not for Association with, B-Raf (*) , 1996, The Journal of Biological Chemistry.

[36]  C. Der,et al.  Peptides containing a consensus Ras binding sequence from Raf-1 and theGTPase activating protein NF1 inhibit Ras function. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[37]  M. Wigler,et al.  Multiple ras functions can contribute to mammalian cell transformation , 1995, Cell.

[38]  M. Marshall,et al.  The effector interactions of p21ras. , 1993, Trends in biochemical sciences.

[39]  R. L. Johnson,et al.  Mammalian expression vectors for Ras family proteins: generation and use of expression constructs to analyze Ras family function. , 2001, Methods in enzymology.

[40]  J. Buss,et al.  Targeting proteins to membranes, using signal sequences for lipid modifications. , 2001, Methods in enzymology.

[41]  S. G. Coats,et al.  Targeting proteins to membranes using signal sequences for lipid modification. , 1995, Methods in enzymology.