The association between ambient UVB dose and ANCA-associated vasculitis relapse and onset

[1]  M. Little,et al.  Environmental risk factors associated with ANCA associated vasculitis: A systematic mapping review. , 2020, Autoimmunity reviews.

[2]  H. Campbell,et al.  Genetically-predicted vitamin D status, ambient UVB during the pandemic and COVID-19 risk in UK Biobank: Mendelian Randomisation study , 2020, medRxiv.

[3]  S. Fisher,et al.  The role of vitamin D in increasing circulating T regulatory cell numbers and modulating T regulatory cell phenotypes in patients with inflammatory disease or in healthy volunteers: A systematic review , 2019, PloS one.

[4]  C. O'Morain,et al.  Sunshine is an Important Determinant of Vitamin D Status Even Among High‐dose Supplement Users: Secondary Analysis of a Randomized Controlled Trial in Crohn's Disease Patients , 2019, Photochemistry and photobiology.

[5]  John Fox,et al.  Visualizing Fit and Lack of Fit in Complex Regression Models with Predictor Effect Plots and Partial Residuals , 2018 .

[6]  D. O’Sullivan,et al.  Data linkage in medical science using the resource description framework: the AVERT model , 2018, HRB open research.

[7]  L. Gesualdo,et al.  Proteinase-3 and myeloperoxidase serotype in relation to demographic factors and geographic distribution in anti-neutrophil cytoplasmic antibody-associated glomerulonephritis , 2018, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[8]  A. Voskuyl,et al.  Relation between duration of the prodromal phase and renal damage in ANCA-associated vasculitis , 2017, BMC Nephrology.

[9]  C. Kallenberg,et al.  University of Groningen Revised 2017 international consensus on testing of ANCAs in granulomatosis with polyangiitis and microscopic polyangiitis Bossuyt, , 2017 .

[10]  H. McNulty,et al.  Ambient UVB Dose and Sun Enjoyment Are Important Predictors of Vitamin D Status in an Older Population. , 2017, The Journal of nutrition.

[11]  A. Salama,et al.  Epigenetic Modifications in ANCA-Associated Vasculitis: Potential for Insights into Disease Pathogenesis and Prediction of Outcome? , 2017, Journal of the American Society of Nephrology : JASN.

[12]  P. van Paassen,et al.  Seasonal Influence on the Risk of Relapse at a Rise of Antineutrophil Cytoplasmic Antibodies in Vasculitis Patients with Renal Involvement , 2017, The Journal of Rheumatology.

[13]  O. Zimba.  338. VITAMIN D DEFICIENCY IN PATIENTS WITH GRANULOMATOSIS WITH POLYANGIITIS , 2017 .

[14]  P. Sørensen,et al.  Vitamin D supplementation reduces relapse rate in relapsing-remitting multiple sclerosis patients treated with natalizumab. , 2016, Multiple sclerosis and related disorders.

[15]  H. Direskeneli,et al.  AB0530 25-Hydroxyvitamin D3 Deficiency: Prevalance and Its Impact on Disease Activity in Small-Medium Vessel Vasculitis , 2016 .

[16]  H. Campbell,et al.  The contributions of adjusted ambient ultraviolet B radiation at place of residence and other determinants to serum 25‐hydroxyvitamin D concentrations , 2016, The British journal of dermatology.

[17]  W. Shadish,et al.  CONSORT extension for reporting N-of-1 trials (CENT) 2015: Explanation and elaboration , 2015, BMJ : British Medical Journal.

[18]  N. Basu,et al.  Classification, epidemiology and clinical subgrouping of antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis. , 2015, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[19]  S. Mackie,et al.  HLA allele variation as a potential explanation for the geographical distribution of granulomatosis with polyangiitis. , 2015, Rheumatology.

[20]  M. Segelmark,et al.  A Population-based Study Showing Better Renal Prognosis for Proteinase 3 Antineutrophil Cytoplasmic Antibody (ANCA)–associated Nephritis Versus Myeloperoxidase ANCA–associated Nephritis , 2014, The Journal of Rheumatology.

[21]  D. Bates,et al.  Fitting Linear Mixed-Effects Models Using lme4 , 2014, 1406.5823.

[22]  R. Lucas,et al.  Vitamin D deficiency and risk for rheumatic diseases: an update , 2013, Current opinion in rheumatology.

[23]  Yichun Hu,et al.  Classification of antineutrophil cytoplasmic autoantibody vasculitides: the role of antineutrophil cytoplasmic autoantibody specificity for myeloperoxidase or proteinase 3 in disease recognition and prognosis. , 2012, Arthritis and rheumatism.

[24]  D. Talwar,et al.  The relation between acute changes in the systemic inflammatory response and plasma 25-hydroxyvitamin D concentrations after elective knee arthroplasty. , 2011, The American journal of clinical nutrition.

[25]  G. Ebers,et al.  Relationship of UV exposure to prevalence of multiple sclerosis in England , 2011, Neurology.

[26]  JoAnn E. Manson,et al.  The 2011 Report on Dietary Reference Intakes for Calcium and Vitamin D from the Institute of Medicine: What Clinicians Need to Know , 2010, The Journal of clinical endocrinology and metabolism.

[27]  Sanford Weisberg,et al.  An R Companion to Applied Regression , 2010 .

[28]  T. Dwyer,et al.  Higher 25‐hydroxyvitamin D is associated with lower relapse risk in multiple sclerosis , 2010, Annals of neurology.

[29]  A. Ponsonby,et al.  Antineutrophil cytoplasmic antibody-associated vasculitides: could geographic patterns be explained by ambient ultraviolet radiation? , 2009, Arthritis and rheumatism.

[30]  C. Kallenberg,et al.  Retinoid X receptor beta polymorphisms do not explain functional differences in vitamins D and A response in Antineutrophil cytoplasmic antibody associated vasculitis patients , 2009, Autoimmunity.

[31]  L. Stamp,et al.  Wegener’s granulomatosis in New Zealand: evidence for a latitude‐dependent incidence gradient , 2007, Internal medicine journal.

[32]  C. Kallenberg.  Antineutrophil cytoplasmic autoantibody-associated small-vessel vasculitis , 2007, Current opinion in rheumatology.

[33]  L. Adorini,et al.  1,25-Dihydroxyvitamin D3 Selectively Modulates Tolerogenic Properties in Myeloid but Not Plasmacytoid Dendritic Cells1 , 2007, The Journal of Immunology.

[34]  D. Scott,et al.  Development and validation of a consensus methodology for the classification of the ANCA-associated vasculitides and polyarteritis nodosa for epidemiological studies , 2006, Annals of the rheumatic diseases.

[35]  W. Koldingsnes,et al.  Retrocalcaneal bursitis in polymyalgia rheumatica , 2001, Annals of the rheumatic diseases.

[36]  D. Scott,et al.  Geoepidemiology of systemic vasculitis: comparison of the incidence in two regions of Europe , 2001, Annals of the rheumatic diseases.

[37]  J. Slusser,et al.  Annual and Interannual Behavior of Solar Ultraviolet Irradiance Revealed by Broadband Measurements¶ , 2000, Photochemistry and photobiology.

[38]  N. Jablonski,et al.  The evolution of human skin coloration. , 2000, Journal of human evolution.

[39]  M. Maclure.  The case-crossover design: a method for studying transient effects on the risk of acute events. , 1991, American journal of epidemiology.

[40]  D. O’Sullivan,et al.  Data linkage in medical science using the resource description framework: the AVERT model [version 1; peer review: 1 approved, 1 approved with reservations] , 2020 .

[41]  P. Merkel,et al.  2012 revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides. , 2013, Arthritis and rheumatism.

[42]  J. Sterne,et al.  Essential Medical Statistics , 2003 .

[43]  Hilde van der Togt,et al.  Publisher's Note , 2003, J. Netw. Comput. Appl..