Transcripts from the Epstein-Barr virus BamHI A fragment are detectable in all three forms of virus latency

[1]  Philip Smith,et al.  Expression of a family of complementary-strand transcripts in Epstein-Barr virus-infected cells. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[2]  Q Y Yao,et al.  Epstein-Barr virus latent gene transcription in nasopharyngeal carcinoma cells: coexpression of EBNA1, LMP1, and LMP2 transcripts , 1992, Journal of virology.

[3]  A. Davies,et al.  ThreePathways ofEpstein-Barr Virus GeneActivation from EBNAl-Positive Latency inB Lymphocytes , 1992 .

[4]  T. Tursz,et al.  Consistent Transcription oftheEpstein-Barr VirusLMP2 GeneinNasopharyngeal Carcinoma , 1992 .

[5]  H. Sato,et al.  Novel transcription from the Epstein-Barr virus terminal EcoRI fragment, DIJhet, in a nasopharyngeal carcinoma , 1990, Journal of virology.

[6]  J. Strominger,et al.  Promoter switching in Epstein-Barr virus during the initial stages of infection of B lymphocytes. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[7]  J. Strominger,et al.  Mutually exclusive use of viral promoters in Epstein-Barr virus latently infected lymphocytes. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[8]  T. Tursz,et al.  Epstein-Barr virus gene expression in nasopharyngeal carcinoma. , 1988, The Journal of general virology.

[9]  L. Young,et al.  Differences in B cell growth phenotype reflect novel patterns of Epstein‐Barr virus latent gene expression in Burkitt's lymphoma cells. , 1987, The EMBO journal.

[10]  I. Andó,et al.  EB virus induction is associated with B-cell maturation. , 1986, Immunology.

[11]  E. Lennette,et al.  Replication of Epstein-Barr virus within the epithelial cells of oral "hairy" leukoplakia, an AIDS-associated lesion. , 1985, The New England journal of medicine.