Effects of antipsychotic medication on functional connectivity in major depressive disorder with psychotic features

[1]  A. Lahti,et al.  Hippocampal Hyperconnectivity to the Visual Cortex Predicts Treatment Response. , 2023, Schizophrenia bulletin.

[2]  B. Mulsant,et al.  Predictors of relapse of psychotic depression: Findings from the STOP-PD II randomized clinical trial. , 2022, Journal of psychiatric research.

[3]  B. Mulsant,et al.  Factor analysis of the CORE measure of psychomotor disturbance in psychotic depression: Findings from the STOP-PD II study , 2022, Psychiatry Research.

[4]  J. P. Hamilton,et al.  Altered resting-state functional connectome in major depressive disorder: a mega-analysis from the PsyMRI consortium , 2021, Translational Psychiatry.

[5]  C. Pantelis,et al.  Functional Connectivity in Antipsychotic-Treated and Antipsychotic-Naive Patients With First-Episode Psychosis and Low Risk of Self-harm or Aggression , 2021, JAMA psychiatry.

[6]  C. Pantelis,et al.  Differentiating the effect of antipsychotic medication and illness on brain volume reductions in first-episode psychosis: A Longitudinal, Randomised, Triple-blind, Placebo-controlled MRI Study , 2021, Neuropsychopharmacology.

[7]  W. Cahn,et al.  Functional connectome differences in individuals with hallucinations across the psychosis continuum , 2021, Scientific reports.

[8]  D. Pizzagalli,et al.  Bioenergetics and Abnormal Functional Connectivity in Psychotic Disorders , 2020, Molecular Psychiatry.

[9]  Michael F. Green,et al.  Early Visual Processing Is Associated With Social Cognitive Performance in Recent-Onset Schizophrenia , 2020, Frontiers in Psychiatry.

[10]  B. Mulsant,et al.  Effects of Antipsychotic Medication on Brain Structure in Patients With Major Depressive Disorder and Psychotic Features: Neuroimaging Findings in the Context of a Randomized Placebo-Controlled Clinical Trial. , 2020, JAMA psychiatry.

[11]  Daniel S. Margulies,et al.  Topographic organization of the human subcortex unveiled with functional connectivity gradients , 2020, Nature Neuroscience.

[12]  Evan M. Gordon,et al.  Defining Individual-Specific Functional Neuroanatomy for Precision Psychiatry , 2019, Biological Psychiatry.

[13]  B. Mulsant,et al.  Effect of Continuing Olanzapine vs Placebo on Relapse Among Patients With Psychotic Depression in Remission: The STOP-PD II Randomized Clinical Trial. , 2019, JAMA.

[14]  Long-Biao Cui,et al.  Effect of second-generation antipsychotics on brain network topology in first-episode schizophrenia: A longitudinal rs-fMRI study , 2019, Schizophrenia Research.

[15]  P. Brambilla,et al.  Effects of olanzapine during cognitive and emotional processing in schizophrenia: A review of functional magnetic resonance imaging findings , 2019, Human psychopharmacology.

[16]  J. Qiu,et al.  Reduced default mode network functional connectivity in patients with recurrent major depressive disorder , 2019, Proceedings of the National Academy of Sciences.

[17]  F. Barkhof,et al.  Exploring resting state connectivity in patients with psychotic depression , 2019, PloS one.

[18]  Michael W. Cole,et al.  Mapping the human brain's cortical-subcortical functional network organization , 2018, NeuroImage.

[19]  E. Vasar,et al.  Profiling of lipidomics before and after antipsychotic treatment in first-episode psychosis , 2019, European Archives of Psychiatry and Clinical Neuroscience.

[20]  Matthew F. Glasser,et al.  Ciftify: A framework for surface-based analysis of legacy MR acquisitions , 2018, NeuroImage.

[21]  Y. Trakadis,et al.  Biomarkers for major depressive and bipolar disorders using metabolomics: A systematic review , 2018, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.

[22]  M. Mimura,et al.  Glutamatergic neurometabolite levels in major depressive disorder: a systematic review and meta-analysis of proton magnetic resonance spectroscopy studies , 2018, Molecular Psychiatry.

[23]  B. Mulsant,et al.  Resting state functional connectivity in patients with remitted psychotic depression: A multi-centre STOP-PD study , 2018, EBioMedicine.

[24]  C. Pantelis,et al.  Staged treatment and acceptability guidelines in early psychosis study (STAGES): A randomized placebo controlled trial of intensive psychosocial treatment plus or minus antipsychotic medication for first‐episode psychosis with low‐risk of self‐harm or aggression. Study protocol and baseline characte , 2018, Early intervention in psychiatry.

[25]  Satrajit S. Ghosh,et al.  FMRIPrep: a robust preprocessing pipeline for functional MRI , 2018, bioRxiv.

[26]  Evan M. Gordon,et al.  Precision Functional Mapping of Individual Human Brains , 2017, Neuron.

[27]  Jesper Andersson,et al.  A multi-modal parcellation of human cerebral cortex , 2016, Nature.

[28]  B T Thomas Yeo,et al.  The modular and integrative functional architecture of the human brain , 2015, Proceedings of the National Academy of Sciences.

[29]  Costin Tanase,et al.  A multimodal analysis of antipsychotic effects on brain structure and function in first-episode schizophrenia. , 2015, JAMA psychiatry.

[30]  A. Reiss,et al.  Decreased Hypothalamic Functional Connectivity with Subgenual Cortex in Psychotic Major Depression , 2015, Neuropsychopharmacology.

[31]  Steven C.R. Williams,et al.  Reduced Cortical Volume and Elevated Astrocyte Density in Rats Chronically Treated With Antipsychotic Drugs—Linking Magnetic Resonance Imaging Findings to Cellular Pathology , 2014, Biological Psychiatry.

[32]  A. Malhotra,et al.  White Matter Changes Associated with Antipsychotic Treatment in First-Episode Psychosis , 2014, Neuropsychopharmacology.

[33]  J. Quevedo,et al.  In vitro effect of antipsychotics on brain energy metabolism parameters in the brain of rats , 2013, Acta Neuropsychiatrica.

[34]  Hannah R. Snyder Major depressive disorder is associated with broad impairments on neuropsychological measures of executive function: a meta-analysis and review. , 2013, Psychological bulletin.

[35]  Daniel C Javitt,et al.  Early sensory contributions to contextual encoding deficits in schizophrenia. , 2011, Archives of general psychiatry.

[36]  Shitij Kapur,et al.  Effect of Chronic Antipsychotic Treatment on Brain Structure: A Serial Magnetic Resonance Imaging Study with Ex Vivo and Postmortem Confirmation , 2011, Biological Psychiatry.

[37]  Ronald Pierson,et al.  Long-term antipsychotic treatment and brain volumes: a longitudinal study of first-episode schizophrenia. , 2011, Archives of general psychiatry.

[38]  S. Rombouts,et al.  Frontiers in Systems Neuroscience Systems Neuroscience , 2022 .

[39]  B. Mulsant,et al.  A double-blind randomized controlled trial of olanzapine plus sertraline vs olanzapine plus placebo for psychotic depression: the study of pharmacotherapy of psychotic depression (STOP-PD). , 2009, Archives of general psychiatry.

[40]  C. Pantelis,et al.  A 1H-MRS investigation of the medial temporal lobe in antipsychotic-naïve and early-treated first episode psychosis , 2008, Schizophrenia Research.

[41]  Allan R. Sampson,et al.  Effect of Chronic Antipsychotic Exposure on Astrocyte and Oligodendrocyte Numbers in Macaque Monkeys , 2008, Biological Psychiatry.

[42]  A. Sampson,et al.  Effect of Chronic Exposure to Antipsychotic Medication on Cell Numbers in the Parietal Cortex of Macaque Monkeys , 2007, Neuropsychopharmacology.

[43]  Hans O Kalkman,et al.  The role of the phosphatidylinositide 3-kinase-protein kinase B pathway in schizophrenia. , 2006, Pharmacology & therapeutics.

[44]  K. Hugdahl,et al.  Brain activation measured with fMRI during a mental arithmetic task in schizophrenia and major depression. , 2004, The American journal of psychiatry.

[45]  N. Minshew,et al.  Prefrontal membrane phospholipid metabolism of child and adolescent offspring at risk for schizophrenia or schizoaffective disorder: an in vivo 31P MRS study , 2003, Molecular Psychiatry.

[46]  Paul Greengard,et al.  Evidence for decreased DARPP-32 in the prefrontal cortex of patients with schizophrenia. , 2002, Archives of general psychiatry.