Coming back: autophagy in cachexia
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[1] N. Tardif,et al. Autophagic-lysosomal pathway is the main proteolytic system modified in the skeletal muscle of esophageal cancer patients. , 2013, The American journal of clinical nutrition.
[2] L. Larsson,et al. The bone morphogenetic protein axis is a positive regulator of skeletal muscle mass , 2013, The Journal of cell biology.
[3] M. Sandri. Protein breakdown in muscle wasting: Role of autophagy-lysosome and ubiquitin-proteasome☆☆☆ , 2013, The international journal of biochemistry & cell biology.
[4] A. Schols,et al. Triggers and mechanisms of skeletal muscle wasting in chronic obstructive pulmonary disease. , 2013, The international journal of biochemistry & cell biology.
[5] B. Jasmin,et al. AMP-activated protein kinase at the nexus of therapeutic skeletal muscle plasticity in Duchenne muscular dystrophy. , 2013, Trends in molecular medicine.
[6] Stefano Piccolo,et al. BMP signaling controls muscle mass , 2013, Nature Genetics.
[7] A. Goldberg,et al. SIRT1 Protein, by Blocking the Activities of Transcription Factors FoxO1 and FoxO3, Inhibits Muscle Atrophy and Promotes Muscle Growth* , 2013, The Journal of Biological Chemistry.
[8] D. Marks,et al. Cancer‐ and endotoxin‐induced cachexia require intact glucocorticoid signaling in skeletal muscle , 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[9] A. Dingemans,et al. Nuclear transcription factor κ B activation and protein turnover adaptations in skeletal muscle of patients with progressive stages of lung cancer cachexia. , 2013, The American journal of clinical nutrition.
[10] M. Polkey,et al. MuRF-1 and Atrogin-1 Protein Expression and Quadriceps Fiber Size and Muscle Mass in Stable Patients with COPD , 2013, COPD.
[11] Y. Min,et al. Acute Exercise Induces FGF21 Expression in Mice and in Healthy Humans , 2013, PloS one.
[12] R. Tompkins,et al. Title efficacy of phosphodiesterase 5 inhibitor on distant burn-induced muscle autophagy, microcirculation, and survival rate. , 2013, American journal of physiology. Endocrinology and metabolism.
[13] M. Sandri,et al. Role of autophagy in COPD skeletal muscle dysfunction. , 2013, Journal of applied physiology.
[14] E. Blough,et al. Altered cardiac muscle mTOR regulation during the progression of cancer cachexia in the ApcMin/+ mouse , 2013, International journal of oncology.
[15] P. Costelli,et al. Autophagic degradation contributes to muscle wasting in cancer cachexia. , 2013, The American journal of pathology.
[16] J. Woodgett,et al. GSK-3α is a central regulator of age-related pathologies in mice. , 2013, The Journal of clinical investigation.
[17] F. López‐Soriano,et al. Mitochondrial and sarcoplasmic reticulum abnormalities in cancer cachexia: altered energetic efficiency? , 2013, Biochimica et biophysica acta.
[18] D. Klionsky,et al. SnapShot: Selective Autophagy , 2013, Cell.
[19] M. Sandri,et al. Cellular and molecular mechanisms of muscle atrophy , 2013, Disease Models & Mechanisms.
[20] Wen Dui,et al. MST1, a key player, in enhancing fast skeletal muscle atrophy , 2013, BMC Biology.
[21] G. Biolo,et al. Consensus definition of sarcopenia, cachexia and pre-cachexia: joint document elaborated by Special Interest Groups (SIG) "cachexia-anorexia in chronic wasting diseases" and "nutrition in geriatrics". , 2010, Clinical nutrition.
[22] P. Costelli,et al. Ca(2+)-dependent proteolysis in muscle wasting. , 2005, The international journal of biochemistry & cell biology.