Neuronal calcium-binding proteins and schizophrenia
暂无分享,去创建一个
[1] C. Andressen,et al. Calcium-binding proteins: selective markers of nerve cells , 1993, Cell and Tissue Research.
[2] P. Somogyi,et al. Relationship of neuronal vulnerability and calcium binding protein immunoreactivity in ischemia , 1990, Experimental Brain Research.
[3] I. Brown,et al. Analysis of putative high-mobility-group (HMG) proteins in neuronal and glial nuclei from rabbit brain , 1981, Neurochemical Research.
[4] A. Guidotti,et al. Calmodulin in brain of schizophrenics , 1980, Neurochemical Research.
[5] I. Módy,et al. Calbindin-D28K (CaBP) levels and calcium currents in acutely dissociated epileptic neurons , 2004, Experimental Brain Research.
[6] Gavin P. Reynolds,et al. A selective decrease in the relative density of parvalbumin-immunoreactive neurons in the hippocampus in schizophrenia , 2002, Schizophrenia Research.
[7] C. Beasley,et al. Neurochemical correlates of cortical GABAergic deficits in schizophrenia: selective losses of calcium binding protein immunoreactivity , 2001, Brain Research Bulletin.
[8] C. Beasley,et al. GABAergic neuronal subtypes in the human frontal cortex — development and deficits in schizophrenia , 2001, Journal of Chemical Neuroanatomy.
[9] P. Goldman-Rakic,et al. Segregation of serotonin 5‐HT2A and 5‐HT3 receptors in inhibitory circuits of the primate cerebral cortex , 2000, The Journal of comparative neurology.
[10] C. Beasley,et al. No alterations in the density of parvalbumin or calretinin immunoreactive neurons in the anterior cingulate cortex (BA 24b/c) in schizophrenia or bipolar disorder , 2000, Schizophrenia Research.
[11] J. Newcomer,et al. NMDA receptor hypofunction model of schizophrenia. , 1999, Journal of psychiatric research.
[12] J. Pierri,et al. Altered GABA neurotransmission and prefrontal cortical dysfunction in schizophrenia , 1999, Biological Psychiatry.
[13] F. Benes,et al. Evidence for altered trisynaptic circuitry in schizophrenic hippocampus , 1999, Biological Psychiatry.
[14] N. Kuroki,et al. Calbindin immunoreactivity in the hippocampal formation and neocortex of schizophrenics , 1999, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[15] Paul J. Harrison. The neuropathology of schizophrenia , 2008 .
[16] D. Lewis. Chandelier cells: shedding light on altered cortical circuitry in schizophrenia , 1998, Molecular Psychiatry.
[17] F. Terro,et al. Mild kainate toxicity produces selective motoneuron death with marked activation of CA2+-permeable AMPA/kainate receptors , 1998, Brain Research.
[18] F. Gage,et al. Nicotinic Receptor-Induced Apoptotic Cell Death of Hippocampal Progenitor Cells , 1998, The Journal of Neuroscience.
[19] F. Benes,et al. A reduction of nonpyramidal cells in sector CA2 of schizophrenics and manic depressives , 1998, Biological Psychiatry.
[20] P. Gaspar,et al. Subpopulations of cortical GABAergic interneurons differ by their expression of D1 and D2 dopamine receptor subtypes. , 1998, Brain research. Molecular brain research.
[21] T. Woo,et al. A subclass of prefrontal gamma-aminobutyric acid axon terminals are selectively altered in schizophrenia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[22] B. Bogerts,et al. Schizophrenia and anteroventral thalamic nucleus: selective decrease of parvalbumin-immunoreactive thalamocortical projection neurons , 1998, Psychiatry Research: Neuroimaging.
[23] D. Weinberger,et al. A candidate molecule approach to defining developmental pathology in schizophrenia. , 1998, Schizophrenia bulletin.
[24] P. Kalus,et al. Altered distribution of parvalbumin-immunoreactive local circuit neurons in the anterior cingulate cortex of schizophrenic patients , 1997, Psychiatry Research: Neuroimaging.
[25] T. Woo,et al. Schizophrenia and the parvalbumin-containing class of cortical local circuit neurons. , 1997, The American journal of psychiatry.
[26] C. Beasley,et al. Parvalbumin-immunoreactive neurons are reduced in the prefrontal cortex of schizophrenics , 1997, Schizophrenia Research.
[27] H. Thoenen,et al. Vulnerability of Midbrain Dopaminergic Neurons in Calbindin‐D28k‐deficient Mice: Lack of Evidence for a Neuroprotective Role of Endogenous Calbindin in MPTPtreated and Weaver Mice , 1997, The European journal of neuroscience.
[28] P. Goldman-Rakic,et al. Functional and anatomical aspects of prefrontal pathology in schizophrenia. , 1997, Schizophrenia bulletin.
[29] L. Colom,et al. Expression of calbindin-D28K in motoneuron hybrid cells after retroviral infection with calbindin-D28K cDNA prevents amyotrophic lateral sclerosis IgG-mediated cytotoxicity. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[30] Marco R. Celio,et al. Guidebook to the calcium-binding proteins , 1996 .
[31] S. Sesack,et al. Axon terminals immunolabeled for dopamine or tyrosine hydroxylase synapse on GABA‐immunoreactive dendrites in rat and monkey cortex , 1995, The Journal of comparative neurology.
[32] F. Benes. Altered glutamatergic and GABAergic mechanisms in the cingulate cortex of the schizophrenic brain. , 1995, Archives of general psychiatry.
[33] J. Olney,et al. Glutamate receptor dysfunction and schizophrenia. , 1995, Archives of general psychiatry.
[34] S. Daviss,et al. Local circuit neurons of the prefrontal cortex in schizophrenia: selective increase in the density of calbindin-immunoreactive neurons , 1995, Psychiatry Research.
[35] P. Goldman-Rakic,et al. Abnormally high neuronal density in the schizophrenic cortex. A morphometric analysis of prefrontal area 9 and occipital area 17. , 1995, Archives of general psychiatry.
[36] C. Marcuccilli,et al. Regulation of excitatory transmission at hippocampal synapses by calbindin D28k. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[37] D. Dr. Calcium regulation of apoptosis. , 1995 .
[38] K. A. Jones,et al. Cortical neurons containing calretinin are selectively resistant to calcium overload and excitotoxicity in vitro , 1994, Neuroscience.
[39] V. Möckel,et al. Vulnerability to excitotoxic stimuli of cultured rat hippocampal neurons containing the calcium-binding proteins calretinin and calbindin D28K , 1994, Brain Research.
[40] W. Janssen,et al. Distribution and synaptic localization of immunocytochemically identified NMDA receptor subunit proteins in sensory-motor and visual cortices of monkey and human , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[41] Françoise Condé,et al. Local circuit neurons immunoreactive for calretinin, calbindin D‐28k or parvalbumin in monkey prefronatal cortex: Distribution and morphology , 1994, The Journal of comparative neurology.
[42] C. Heizmann,et al. Parvalbumin and calbindin D‐28k in the human motor system and in motor neuron disease , 1993, Neuropathology and applied neurobiology.
[43] J. Lund,et al. Local circuit neurons of developing and mature macaque prefrontal cortex: Golgi and immunocytochemical characteristics , 1993, The Journal of comparative neurology.
[44] B. Komm,et al. Stable expression of the calbindin-D28K complementary DNA interferes with the apoptotic pathway in lymphocytes. , 1992, Molecular endocrinology.
[45] K. Baimbridge,et al. Calcium-binding proteins in the nervous system , 1992, Trends in Neurosciences.
[46] C. Heizmann,et al. Changes in Ca2+-binding proteins in human neurodegenerative disorders , 1992, Trends in Neurosciences.
[47] F. Benes,et al. Deficits in small interneurons in prefrontal and cingulate cortices of schizophrenic and schizoaffective patients. , 1991, Archives of general psychiatry.
[48] M. Mattson,et al. Evidence for calcium-reducing and excitoprotective roles for the calcium-binding protein calbindin-1328k in cultured hippocampal neurons , 1991, Neuron.
[49] J. Mazière,et al. Could the interaction of neuroleptics with calmodulin be an "explanation" of the psychotropic effects? , 1991, L'Encephale.
[50] S. Christakos,et al. Specific reduction of calcium-binding protein (28-kilodalton calbindin-D) gene expression in aging and neurodegenerative diseases. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[51] H. Scharfman,et al. Protection of dentate hilar cells from prolonged stimulation by intracellular calcium chelation. , 1989, Science.
[52] R. S. Sloviter. Calcium‐binding protein (calbindin‐D28k) and parvalbumin immunocytochemistry: Localization in the rat hippocampus with specific reference to the selective vulnerability of hippocampal neurons to seizure activity , 1989, The Journal of comparative neurology.
[53] S. Christakos,et al. Vitamin D-dependent calcium binding proteins: chemistry, distribution, functional considerations, and molecular biology. , 1989, Endocrine reviews.
[54] C. Klee,et al. Advances in second messenger and phosphoprotein research , 1988 .
[55] F. Benes,et al. Quantitative cytoarchitectural studies of the cerebral cortex of schizophrenics. , 1986, Archives of general psychiatry.