An inactivated Pseudomonas aeruginosa medicament inhibits airway allergic inflammation and improves epithelial functions
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Hui-jun Liu | X. Qin | Yurong Tan | Lili Wang | Huihui Yang
[1] B. Rubin,et al. Reduced surface toll-like receptor-4 expression and absent interferon-γ–inducible protein-10 induction in cystic fibrosis airway cells , 2011, Experimental lung research.
[2] F. Gantner,et al. TLR agonist mediated suppression of allergic responses is associated with increased innate inflammation in the airways. , 2011, Pulmonary pharmacology & therapeutics.
[3] P. Burgel,et al. Pseudomonas aeruginosa induces vascular endothelial growth factor synthesis in airway epithelium in vitro and in vivo , 2011, European Respiratory Journal.
[4] Jin-woo Park,et al. A20 Attenuates Allergic Airway Inflammation in Mice1 , 2009, The Journal of Immunology.
[5] Hong-shan Wang,et al. Cell-Autonomous Role for NF-κB in Immature Bone Marrow B Cells1 , 2009, The Journal of Immunology.
[6] Zizhen Kang,et al. The Critical Role of Epithelial-Derived Act1 in IL-17- and IL-25-Mediated Pulmonary Inflammation1 , 2009, The Journal of Immunology.
[7] A. Leonardi,et al. The Adaptor Protein CIKS/Act1 Is Essential for IL-25-Mediated Allergic Airway Inflammation1 , 2009, The Journal of Immunology.
[8] A. Ashkar,et al. FimH Adhesin of Type 1 Fimbriae Is a Potent Inducer of Innate Antimicrobial Responses Which Requires TLR4 and Type 1 Interferon Signalling , 2008, PLoS pathogens.
[9] R. Bals,et al. Microbial Patterns Signaling via Toll-Like Receptors 2 and 5 Contribute to Epithelial Repair, Growth and Survival , 2008, PloS one.
[10] I. Adcock,et al. Role of TLR2, TLR4, and MyD88 in murine ozone-induced airway hyperresponsiveness and neutrophilia. , 2007, Journal of applied physiology.
[11] A. Chang,et al. Reduced toll‐like receptor 4 and substance P gene expression is associated with airway bacterial colonization in children , 2007, Pediatric pulmonology.
[12] T. Yoshikawa,et al. The role of toll-like receptor 4 in airway inflammation induced by diesel exhaust particles , 2006, Archives of Toxicology.
[13] Hongzhen He,et al. Toll-like receptor 4 or 2 agonists decrease allergic inflammation. , 2005, American journal of respiratory cell and molecular biology.
[14] J. Pearce,et al. Toll-like receptor 4 in normal and inflamed lungs and other organs of pig, dog and cattle. , 2004, Histology and histopathology.
[15] E. C. Snow,et al. Act1, a negative regulator in CD40- and BAFF-mediated B cell survival. , 2004, Immunity.
[16] Zi-qiang Luo,et al. Temporal and spatial distribution of VIP, CGRP and their receptors in the development of airway hyperresponsiveness in the lungs. , 2004, Sheng li xue bao : [Acta physiologica Sinica].
[17] R. Ulevitch,et al. MD-2 and TLR4 N-Linked Glycosylations Are Important for a Functional Lipopolysaccharide Receptor* , 2002, The Journal of Biological Chemistry.