Early regression and late progression in coronary artery lesions in the first 3 months following coronary angioplasty

In order to determine the changes in stenotic lesions following coronary angioplasty, detailed quantitative angiographie measurements were performed in 254 patients (292 lesions) immediately post-angioplasty and then at one of three predetermined follow-up times, at 30, 60 or 90 days. The absolute changes in mm of the minimal lumen diameter were compared for the three groups, and a relatively high follow-up rate of 88% was achieved. In the groups of patients followed-up at 30 and 60 days, the response was variable with 6% of the lesions showing a significant improvement in both groups and 1% and 12% respectively, showing a deterioration. At 90 days no lesions were seen to improve with 23% deteriorating.

[1]  B. Waller,et al.  Morphologic evidence of accelerated left main coronary artery stenosis: a late complication of percutaneous transluminal balloon angioplasty of the proximal left anterior descending coronary artery. , 1987, Journal of the American College of Cardiology.

[2]  P. Serruys,et al.  Emergency coronary angioplasty in refractory unstable angina. , 1985, The New England journal of medicine.

[3]  T. Takaro,et al.  Observer Agreement in Evaluating Coronary Angiograms , 1975, Circulation.

[4]  E J Topol,et al.  Variability of quantitative digital subtraction coronary angiography before and after percutaneous transluminal coronary angioplasty. , 1987, The American journal of cardiology.

[5]  S. M. Collins,et al.  Changes in cross-sectional area of the coronary lumen in the six months after angioplasty: a quantitative analysis of the variable response to percutaneous transluminal angioplasty. , 1986, Circulation.

[6]  R E Vlietstra,et al.  Restenosis after percutaneous transluminal coronary angioplasty (PTCA): a report from the PTCA Registry of the National Heart, Lung, and Blood Institute. , 1984, The American journal of cardiology.

[7]  L. Elveback,et al.  The unpredictable progression of symptomatic coronary artery disease: a serial clinical-angiographic analysis. , 1981, Mayo Clinic proceedings.

[8]  A E Becker,et al.  Transluminal coronary angioplasty and early restenosis. Fibrocellular occlusion after wall laceration. , 1983, British heart journal.

[9]  J. Kramer,et al.  Progression and regression of coronary atherosclerosis: relation to risk factors. , 1983, American heart journal.

[10]  C Vallbracht,et al.  Recurrence rate after successful coronary angioplasty. , 1985, European heart journal.

[11]  V. Fuster,et al.  Balloon angioplasty. Natural history of the pathophysiological response to injury in a pig model. , 1985, Circulation research.

[12]  J. H. C. Reiber,et al.  State of the Art in Quantitative Coronary Arteriography , 1986, Developments in Cardiovascular Medicine.

[13]  C. J. Kooijman,et al.  Quantitative angiography of the left anterior descending coronary artery: correlations with pressure gradient and results of exercise thallium scintigraphy. , 1985, Circulation.

[14]  S Levine,et al.  Coronary angioplasty: clinical and angiographic follow-up. , 1985, The American journal of cardiology.

[15]  B Meier,et al.  Restenosis after successful coronary angioplasty in patients with single-vessel disease. , 1986, Circulation.

[16]  N. Ratliff,et al.  Intimal proliferation of smooth muscle cells as an explanation for recurrent coronary artery stenosis after percutaneous transluminal coronary angioplasty. , 1985, Journal of the American College of Cardiology.

[17]  Johan H. C. Reiber,et al.  Approaches towards standardization in acquisition and quantitation of arterial dimensions from cineangiograms , 1986 .

[18]  C. J. Slager,et al.  Quantitative Coronary and Left Ventricular Cineangiography: Methodology and Clinical Applications , 1986 .

[19]  W. O’Neill,et al.  Value of exercise radionuclide ventriculography and thallium-201 scintigraphy in evaluating successful coronary angioplasty: comparison with coronary flow reserve, translesional gradient and percent diameter stenosis. , 1987, European heart journal.

[20]  B. McManus,et al.  Status of the major epicardial coronary arteries 80 to 150 days after percutaneous transluminal coronary angioplasty. Analysis of 3 necropsy patients. , 1983, The American journal of cardiology.

[21]  H. Blackburn.  Electrocardiographic classification for population comparisons. The Minnesota code. , 1969, Journal of electrocardiology.

[22]  J. Douglas,et al.  Coronary artery spasm at the site of angioplasty in the first 2 months after successful percutaneous transluminal coronary angioplasty. , 1983, Journal of the American College of Cardiology.

[23]  I. Hjermann,et al.  [The Minnesota code]. , 1970, Nordisk medicin.

[24]  J. Kramer,et al.  Progression of Coronary Atherosclerosis , 1981, Circulation.

[25]  T. Ulbright,et al.  Morphologic observations after percutaneous transluminal balloon angioplasty of early and late aortocoronary saphenous vein bypass grafts. , 1984, Journal of the American College of Cardiology.

[26]  J. Murray,et al.  Variability in the Analysis of Coronary Arteriograms , 1977, Circulation.

[27]  R. Dinsmore,et al.  Interobserver Variability in Coronary Angiography , 1976, Circulation.

[28]  J. J. Gerbrands,et al.  Assessment of short-, medium-, and long-term variations in arterial dimensions from computer-assisted quantitation of coronary cineangiograms. , 1985, Circulation.

[29]  A. Bruschke,et al.  The Anatomic Evolution of Coronary Artery Disease Demonstrated by Coronary Arteriography in 256 Nonoperated Patients , 1981, Circulation.

[30]  E L Bolson,et al.  A new digital electronic caliper for measurement of coronary arterial stenosis: comparison with visual estimates and computer-assisted measurements. , 1984, The American journal of cardiology.