The Differential Effect of Treadmill Exercise Intensity on Hippocampal Soluble Aβ and Lipid Metabolism in APP/PS1 Mice
暂无分享,去创建一个
[1] G. Ricci. Social Aspects of Dementia Prevention from a Worldwide to National Perspective: A Review on the International Situation and the Example of Italy , 2019, Behavioural neurology.
[2] Diana C. Rostirolla,et al. Nuclear RXRα and RXRβ receptors exert distinct and opposite effects on RA-mediated neuroblastoma differentiation. , 2019, Biochimica et biophysica acta. Molecular cell research.
[3] YingChun Liu,et al. Effect of retinoid X receptor-α nuclear export inhibition on apoptosis of neurons in vivo and in vitro. , 2017, Molecular medicine reports.
[4] Alina Solomon,et al. Physical Activity and Alzheimer’s Disease: A Systematic Review , 2017, The journals of gerontology. Series A, Biological sciences and medical sciences.
[5] H. Moreno,et al. Role of Liver X Receptor in AD Pathophysiology , 2015, PloS one.
[6] R. Morishita,et al. The roles of lipid and glucose metabolism in modulation of β-amyloid, tau, and neurodegeneration in the pathogenesis of Alzheimer disease , 2015, Front. Aging Neurosci..
[7] X. Tong,et al. Treadmill exercise enhances synaptic plasticity, but does not alter β-amyloid deposition in hippocampi of aged APP/PS1 transgenic mice , 2015, Neuroscience.
[8] C. Cotman,et al. Moderate to high-intensity physical exercise in patients with Alzheimer’s disease , 2015, Alzheimer's & Dementia.
[9] Lars Bäckman,et al. A 2 year multidomain intervention of diet, exercise, cognitive training, and vascular risk monitoring versus control to prevent cognitive decline in at-risk elderly people (FINGER): a randomised controlled trial , 2015, The Lancet.
[10] M. Carrillo,et al. Summary of the evidence on modifiable risk factors for cognitive decline and dementia: A population-based perspective , 2015, Alzheimer's & Dementia.
[11] Minna Rusanen,et al. Leisure-time physical activity from mid- to late life, body mass index, and risk of dementia , 2015, Alzheimer's & Dementia.
[12] Juan Zhang,et al. Cholesterol metabolism and homeostasis in the brain , 2015, Protein & Cell.
[13] Kristine Yaffe,et al. Potential for primary prevention of Alzheimer's disease: an analysis of population-based data , 2014, The Lancet Neurology.
[14] B. Schreurs,et al. Dietary cholesterol increases ventricular volume and narrows cerebrovascular diameter in a rabbit model of Alzheimer’s disease , 2013, Neuroscience.
[15] J. Pozueta,et al. Synaptic changes in Alzheimer’s disease and its models , 2013, Neuroscience.
[16] Carl W. Cotman,et al. Exercise counteracts declining hippocampal function in aging and Alzheimer's disease , 2013, Neurobiology of Disease.
[17] R. Steiner,et al. Sterol metabolism disorders and neurodevelopment-an update. , 2013, Developmental disabilities research reviews.
[18] Michael T. Heneka,et al. PPARγ/RXRα-Induced and CD36-Mediated Microglial Amyloid-β Phagocytosis Results in Cognitive Improvement in Amyloid Precursor Protein/Presenilin 1 Mice , 2012, The Journal of Neuroscience.
[19] J. Vance. Dysregulation of cholesterol balance in the brain: contribution to neurodegenerative diseases , 2012, Disease Models & Mechanisms.
[20] S. Rivest,et al. Lipid metabolism and neuroinflammation in Alzheimer's disease: a role for liver X receptors. , 2012, Endocrine reviews.
[21] V. Pankratz,et al. Computer activities, physical exercise, aging, and mild cognitive impairment: a population-based study. , 2012, Mayo Clinic proceedings.
[22] D A Bennett,et al. Total daily physical activity and the risk of AD and cognitive decline in older adults , 2012, Neurology.
[23] L. Yágüez,et al. The effects on cognitive functions of a movement‐based intervention in patients with Alzheimer's type dementia: a pilot study , 2011, International journal of geriatric psychiatry.
[24] J. Viña,et al. Cholesterol and amyloid-β: evidence for a cross-talk between astrocytes and neuronal cells. , 2011, Journal of Alzheimer's disease : JAD.
[25] N. Fitz,et al. The role of ATP-binding cassette transporter A1 in Alzheimer's disease and neurodegeneration. , 2010, Biochimica et biophysica acta.
[26] K. Yaffe,et al. Physical Activity Over the Life Course and Its Association with Cognitive Performance and Impairment in Old Age , 2010, Journal of the American Geriatrics Society.
[27] A. Tall,et al. Role of HDL, ABCA1, and ABCG1 transporters in cholesterol efflux and immune responses. , 2010, Arteriosclerosis, thrombosis, and vascular biology.
[28] J. Buxbaum,et al. APOE genotype results in differential effects on the peripheral clearance of amyloid-beta42 in APOE knock-in and knock-out mice. , 2010, Journal of Alzheimer's disease : JAD.
[29] Katie Hamm,et al. apoE isoform-specific disruption of amyloid beta peptide clearance from mouse brain. , 2008, The Journal of clinical investigation.
[30] S. Andersson,et al. Neuronal expression and subcellular localization of cholesterol 24‐hydroxylase in the mouse brain , 2008, The Journal of comparative neurology.
[31] D. Holtzman,et al. Overexpression of ABCA1 reduces amyloid deposition in the PDAPP mouse model of Alzheimer disease. , 2008, The Journal of clinical investigation.
[32] J. Repa,et al. Liver X Receptor Activation Enhances Cholesterol Loss from the Brain, Decreases Neuroinflammation, and Increases Survival of the NPC1 Mouse , 2007, The Journal of Neuroscience.
[33] I. Lefterov,et al. Role of LXR and ABCA1 in the pathogenesis of Alzheimer's disease - implications for a new therapeutic approach. , 2007, Current Alzheimer research.
[34] K. Fukuchi,et al. Lack of LDL receptor aggravates learning deficits and amyloid deposits in Alzheimer transgenic mice , 2006, Neurobiology of Aging.
[35] J. Dietschy,et al. Thematic review series: Brain Lipids. Cholesterol metabolism in the central nervous system during early development and in the mature animal Published, JLR Papers in Press, May 16, 2004. DOI 10.1194/jlr.R400004-JLR200 , 2004, Journal of Lipid Research.
[36] K. Weisgraber,et al. Cholesterol Published, JLR Papers in Press, January 23, 2003. DOI 10.1194/jlr.R300007-JLR200 , 2003, Journal of Lipid Research.
[37] K. Fassbender,et al. Effects of statins on human cerebral cholesterol metabolism and secretion of Alzheimer amyloid peptide , 2002, Neurology.
[38] J. Leverenz,et al. Diet-induced hypercholesterolemia enhances brain A&bgr; accumulation in transgenic mice , 2002, Neuroreport.
[39] Yonghe Li,et al. Differential Functions of Members of the Low Density Lipoprotein Receptor Family Suggested by Their Distinct Endocytosis Rates* , 2001, The Journal of Biological Chemistry.
[40] Rong Wang,et al. Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model , 2000, Neurobiology of Disease.
[41] Y. Rolland,et al. Feasibility [corrected] of regular physical exercise for patients with moderate to severe Alzheimer disease. , 2000, The journal of nutrition, health & aging.
[42] W. Klein,et al. Cholesterol Modulates -Secretase Cleavage of Amyloid Precursor Protein (*) , 1996, The Journal of Biological Chemistry.
[43] J. Dallongeville,et al. Modulation of plasma triglyceride levels by apoE phenotype: a meta-analysis. , 1992, Journal of lipid research.