Effects of Atorvastatin on Th polarization in patients with acute myocardial infarction
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Bin Li | Ying Liu | Jing Yuan | Xiang Cheng | Baojun Lu | Min Wang | Y. Liu | Jing Yuan | Yan Cheng | Xiang Cheng | Y. Liao | Jin-ying Zhang | Min Wang | Yan Cheng | Yu‐Hua Liao | Jinying Zhang | Hongxia Ge | Bao-jun Lu | Hong-xia Ge | Bin Li
[1] T. Toyo-Oka,et al. Inhibition of Hydroxymethylglutaryl-Coenzyme A Reductase Reduces Th1 Development and Promotes Th2 Development , 2003, Circulation research.
[2] Georg Ertl,et al. Impact of hydroxymethylglutaryl coenzyme a reductase inhibition on left ventricular remodeling after myocardial infarction: an experimental serial cardiac magnetic resonance imaging study. , 2002, Journal of the American College of Cardiology.
[3] Joerg Kallen,et al. Statins selectively inhibit leukocyte function antigen-1 by binding to a novel regulatory integrin site , 2001, Nature Medicine.
[4] Allen J. Taylor,et al. Effect of atorvastatin and pravastatin on serum C-reactive protein. , 2003, American heart journal.
[5] S. Grundy,et al. Effect of Hydroxymethyl Glutaryl Coenzyme A Reductase Inhibitor Therapy on High Sensitive C-Reactive Protein Levels , 2001, Circulation.
[6] L. Passos,et al. Anti-inflammatory effect of atorvastatin (80 mg) in unstable angina pectoris and non-Q-wave acute myocardial infarction. , 2003, The American journal of cardiology.
[7] M. Boothby,et al. Preferential role for NF-kappa B/Rel signaling in the type 1 but not type 2 T cell-dependent immune response in vivo. , 1999, Journal of immunology.
[8] J. Egido,et al. Atorvastatin reduces NF-kappaB activation and chemokine expression in vascular smooth muscle cells and mononuclear cells. , 1999, Atherosclerosis.
[9] A. Bøyum,et al. Isolation of mononuclear cells and granulocytes from human blood. , 1968 .
[10] D. Klinman,et al. Disease severity in patients with systemic lupus erythematosus correlates with an increased ratio of interleukin-10:interferon-gamma-secreting cells in the peripheral blood. , 1996, Arthritis and rheumatism.
[11] S. Constant,et al. Induction of Th1 and Th2 CD4+ T cell responses: the alternative approaches. , 1997, Annual review of immunology.
[12] P. Galuppo,et al. Improvement of Left Ventricular Remodeling and Function by Hydroxymethylglutaryl Coenzyme A Reductase Inhibition With Cerivastatin in Rats With Heart Failure After Myocardial Infarction , 2001, Circulation.
[13] M. Endres,et al. Treatment of Relapsing Paralysis in Experimental Encephalomyelitis by Targeting Th1 Cells through Atorvastatin , 2003, The Journal of experimental medicine.
[14] K. Dickstein,et al. Effect of combined statin and beta-blocker treatment on one-year morbidity and mortality after acute myocardial infarction associated with heart failure. , 2004, The American journal of cardiology.
[15] M. Entman,et al. The inflammatory response in myocardial infarction. , 2002, Cardiovascular research.
[16] S. Frøland,et al. Increased Expression of Interleukin-1 in Coronary Artery Disease With Downregulatory Effects of HMG-CoA Reductase Inhibitors , 2004, Circulation.
[17] J. Leor,et al. Cytotoxic T lymphocytes are activated following myocardial infarction and can recognize and kill healthy myocytes in vitro. , 2000, Journal of molecular and cellular cardiology.
[18] J. Egido,et al. Decreased circulating Fas ligand in patients with familial combined hyperlipidemia or carotid atherosclerosis: normalization by atorvastatin. , 2004, Journal of the American College of Cardiology.
[19] A. Bogers,et al. Peripheral blood dendritic cells in human end-stage heart failure and the early post-transplant period: evidence for systemic Th1 immune responses. , 2004, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.
[20] O. Dewald,et al. Inflammatory mechanisms in myocardial infarction. , 2003, Current drug targets. Inflammation and allergy.
[21] F. Mach,et al. Statins as a newly recognized type of immunomodulator , 2000, Nature Medicine.
[22] J. Rehman,et al. Experimental autoimmune myocarditis produced by adoptive transfer of splenocytes after myocardial infarction. , 1998, Circulation research.
[23] S. Youssef,et al. The HMG-CoA reductase inhibitor, atorvastatin, promotes a Th2 bias and reverses paralysis in central nervous system autoimmune disease , 2002, Nature.
[24] K. Mohler,et al. Analysis of cytokine mRNA expression in the central nervous system of mice with experimental autoimmune encephalomyelitis reveals that IL-10 mRNA expression correlates with recovery. , 1992, Journal of immunology.