Effects of K+ channel openers on vasculature
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[1] I. Ghiu,et al. K ATP channels of primary human coronary artery endothelial cells consist of a heteromultimeric complex of Kir6.1, Kir6.2, and SUR2B subunits. , 2004, Journal of molecular and cellular cardiology.
[2] S. Heinemann,et al. Reactive oxygen species impair Slo1 BK channel function by altering cysteine-mediated calcium sensing , 2004, Nature Structural &Molecular Biology.
[3] M. Wheeler,et al. Episodic coronary artery vasospasm and hypertension develop in the absence of Sur2 K(ATP) channels. , 2002, The Journal of clinical investigation.
[4] T. Shibasaki,et al. Mouse model of Prinzmetal angina by disruption of the inward rectifier Kir6.1 , 2002, Nature Medicine.
[5] A. Braun,et al. A Catalytically Inactive Mutant of Type I cGMP-dependent Protein Kinase Prevents Enhancement of Large Conductance, Calcium-sensitive K+ Channels by Sodium Nitroprusside and cGMP* , 2001, The Journal of Biological Chemistry.
[6] S. Seino,et al. Functional Roles of Cardiac and Vascular ATP-Sensitive Potassium Channels Clarified by Kir6.2-Knockout Mice , 2001, Circulation research.
[7] K. Muraki,et al. Effects of KRN2391 on ionic currents in rabbit femoral arterial myocytes , 2001, British journal of pharmacology.
[8] K. Muraki,et al. Nifedipine and nisoldipine modulate membrane potential of vascular endothelium via a myo-endothelial pathway. , 2000, Life sciences.
[9] S. Lohmann,et al. Cyclic GMP-dependent Protein Kinase Signaling Pathway Inhibits RhoA-induced Ca2+ Sensitization of Contraction in Vascular Smooth Muscle* , 2000, The Journal of Biological Chemistry.
[10] J. Petersson,et al. Vasodilator effects of KRN2391, levcromakalim and 3-morpholino-sydnonimin in human pial and omental arteries , 2000, Naunyn-Schmiedeberg's Archives of Pharmacology.
[11] J. Daut,et al. ATP‐sensitive potassium channels in capillaries isolated from guinea‐pig heart , 2000, The Journal of physiology.
[12] T. Murai,et al. Levcromakalim causes indirect endothelial hyperpolarization via a myo‐endothelial pathway , 1999, British journal of pharmacology.
[13] A. Szewczyk,et al. Mitochondrial ATP‐Dependent Potassium Channels: Viable Candidate Effectors of Ischemic Preconditioning a , 1999, Annals of the New York Academy of Sciences.
[14] K. Muraki,et al. Ca2+ spark as a regulator of ion channel activity. , 1999, Japanese journal of pharmacology.
[15] Hikaru Suzuki,et al. Endothelium‐dependent hyperpolarization and intercellular electrical coupling in guinea‐pig mesenteric arterioles , 1999, The Journal of physiology.
[16] F. Hofmann,et al. Regulation of stably expressed and native BK channels from human myometrium by cGMP- and cAMP-dependent protein kinase , 1998, Pflügers Archiv.
[17] A. Bonev,et al. Frequency modulation of Ca2+sparks is involved in regulation of arterial diameter by cyclic nucleotides. , 1998, American journal of physiology. Cell physiology.
[18] N. Standen,et al. ATP-sensitive and inwardly rectifying potassium channels in smooth muscle. , 1997, Physiological reviews.
[19] Y. Horio,et al. Sulphonylurea receptor 2B and Kir6.1 form a sulphonylurea‐sensitive but ATP‐insensitive K+ channel. , 1997, The Journal of physiology.
[20] Y. Horio,et al. A Novel Sulfonylurea Receptor Forms with BIR (Kir6.2) a Smooth Muscle Type ATP-sensitive K+ Channel* , 1996, The Journal of Biological Chemistry.
[21] T. Bolton,et al. Two types of ATP‐sensitive potassium channels in rat portal vein smooth muscle cells , 1996, British journal of pharmacology.
[22] J. Inazawa,et al. Reconstitution of IKATP: An Inward Rectifier Subunit Plus the Sulfonylurea Receptor , 1995, Science.
[23] N. Ogawa. Pharmacological properties of KRN2391, a novel vasodilator of the nitrate-potassium channel opener hybrid type. , 1994, General pharmacology.
[24] Y. Okada,et al. Antihypertensive and vasorelaxant effects on KRN2391 in spontaneously hypertensive rats. , 1994, General pharmacology.
[25] T. Ishikawa,et al. Regulation of Ca2+ channels by cAMP and cGMP in vascular smooth muscle cells. , 1993, Circulation research.
[26] M. Shigekawa,et al. Maxi K+ channels are stimulated by cyclic guanosine monophosphate-dependent protein kinase in canine coronary artery smooth muscle cells , 1993, Pflügers Archiv.
[27] K. Sanders,et al. Cromakalim and lemakalim activate Ca2+-dependent K+ channels in canine colon , 1992, Pflügers Archiv.
[28] T. Yanagisawa,et al. An analysis of the nitrate‐like and K channel opening actions of KRN2391 in canine coronary arterial smooth muscle , 1991, British journal of pharmacology.
[29] N. Standen,et al. Hyperpolarizing vasodilators activate ATP-sensitive K+ channels in arterial smooth muscle. , 1989, Science.
[30] D. Escande,et al. The potassium channel opener cromakalim (BRL 34915) activates ATP-dependent K+ channels in isolated cardiac myocytes. , 1988, Biochemical and biophysical research communications.
[31] Y. Tawada,et al. Regulation of the plasma membrane Ca2+ pump by cyclic nucleotides in cultured vascular smooth muscle cells. , 1988, The Journal of biological chemistry.
[32] C. Hales,et al. THE SULPHONYLUREA RECEPTOR MAY BE AN ATP-SENSITIVE POTASSIUM CHANNEL , 1985, The Lancet.
[33] T. Itoh,et al. Effects of 2-nicotinamidoethyl nitrate (nicorandil; SG-75) and its derivative on smooth muscle cells of the canine mesenteric artery. , 1984, The Journal of pharmacology and experimental therapeutics.
[34] A. Noma,et al. ATP-regulated K+ channels in cardiac muscle , 1983, Nature.
[35] K. Muraki,et al. Effects of KRN4884, a novel K+ channel opener, on ionic currents in rabbit femoral arterial myocytes. , 2003, Journal of pharmacological sciences.
[36] Yi Cui,et al. The molecular composition of K(ATP) channels in human pulmonary artery smooth muscle cells and their modulation by growth. , 2002, American journal of respiratory cell and molecular biology.
[37] M. Gollasch. Potassium channels in the coronary circulation , 2001 .