Roles of 3,4-methylenedioxymethamphetamine (MDMA)-induced alteration of connexin43 and intracellular Ca(2+) oscillation in its cardiotoxicity.
暂无分享,去创建一个
Rong-shuai Wang | Liang Liu | L. Ren | G. Qu | Qian Liu | Luo Zhuo | Quan Liu | Ting-yi Sun | Yan Liu
[1] Mario Delmar,et al. Cardiac connexins, mutations and arrhythmias , 2012, Current opinion in cardiology.
[2] R. Gourdie,et al. Enhanced PKCε mediated phosphorylation of connexin43 at serine 368 by a carboxyl-terminal mimetic peptide is dependent on injury , 2011, Channels.
[3] R. Gourdie,et al. A Peptide Mimetic of the Connexin43 Carboxyl Terminus Reduces Gap Junction Remodeling and Induced Arrhythmia Following Ventricular Injury , 2011, Circulation research.
[4] S. Sadayappan,et al. In vivo and in vitro cardiac responses to beta-adrenergic stimulation in volume-overload heart failure. , 2010, Journal of molecular and cellular cardiology.
[5] S. Bello,et al. Myocardial expression of TNF-alpha, IL-1beta, IL-6, IL-8, IL-10 and MCP-1 after a single MDMA dose administered in a rat model. , 2010, Current pharmaceutical biotechnology.
[6] S. Bello,et al. MDMA toxicity and pathological consequences: a review about experimental data and autopsy findings. , 2010, Current pharmaceutical biotechnology.
[7] J. Haefliger,et al. An angiotensin II- and NF-kappaB-dependent mechanism increases connexin 43 in murine arteries targeted by renin-dependent hypertension. , 2010, Cardiovascular research.
[8] M. Nasr-Esfahani,et al. Toxicity of ecstasy (MDMA) towards embryonic stem cell-derived cardiac and neural cells. , 2010, Toxicology in vitro : an international journal published in association with BIBRA.
[9] L. Gandía,et al. The effects of 3,4-methylenedioxymethamphetamine (MDMA) on nicotinic receptors: intracellular calcium increase, calpain/caspase 3 activation, and functional upregulation. , 2010, Toxicology and applied pharmacology.
[10] P. Lampe,et al. Connexin43 phosphorylation: structural changes and biological effects. , 2009, The Biochemical journal.
[11] F. Carvalho,et al. Metabolites of MDMA Induce Oxidative Stress and Contractile Dysfunction in Adult Rat Left Ventricular Myocytes , 2009, Cardiovascular Toxicology.
[12] Seungho Wang,et al. Effects of 18‐glycyrrhetinic acid on serine 368 phosphorylation of connexin43 in rat neonatal cardiomyocytes , 2008, Cell biology international.
[13] M. Neri,et al. Cardiac oxidative stress determination and myocardial morphology after a single ecstasy (MDMA) administration in a rat model , 2008, International Journal of Legal Medicine.
[14] Liang Liu,et al. Aconitine alters connexin43 phosphorylation status and [Ca2+] oscillation patterns in cultured ventricular myocytes of neonatal rats. , 2007, Toxicology in vitro : an international journal published in association with BIBRA.
[15] J. Saffitz,et al. Protein kinase Cε mediates salutary effects on electrical coupling induced by ischemic preconditioning , 2007 .
[16] Mark von Zastrow,et al. Microtubule Plus-End-Tracking Proteins Target Gap Junctions Directly from the Cell Interior to Adherens Junctions , 2007, Cell.
[17] P. Lampe,et al. Selectivity of Connexin 43 Channels Is Regulated Through Protein Kinase C–Dependent Phosphorylation , 2006, Circulation research.
[18] F. Schifano,et al. Ecstasy (MDMA, MDA, MDEA, MBDB) consumption, seizures, related offences, prices, dosage levels and deaths in the UK (1994–2003) , 2006, Journal of psychopharmacology.
[19] R. Böhm,et al. Hyperdynamer Kreislaufstillstand nach Ingestion von Ecstasy , 2005 .
[20] G. Morley,et al. Cardiac-Specific Loss of N-Cadherin Leads to Alteration in Connexins With Conduction Slowing and Arrhythmogenesis , 2005, Circulation research.
[21] S. Elliott. MDMA and MDA concentrations in antemortem and postmortem specimens in fatalities following hospital admission. , 2005, Journal of analytical toxicology.
[22] D. Laird. Connexin phosphorylation as a regulatory event linked to gap junction internalization and degradation. , 2005, Biochimica et biophysica acta.
[23] Xin Xu,et al. Connexin43 Associated with an N-cadherin-containing Multiprotein Complex Is Required for Gap Junction Formation in NIH3T3 Cells* , 2005, Journal of Biological Chemistry.
[24] M. Rosen,et al. Gap junctional remodeling by hypoxia in cultured neonatal rat ventricular myocytes. , 2005, Cardiovascular research.
[25] P. Lampe,et al. The effects of connexin phosphorylation on gap junctional communication. , 2004, The international journal of biochemistry & cell biology.
[26] Fabrizio Schifano,et al. A bitter pill. Overview of ecstasy (MDMA, MDA) related fatalities , 2004, Psychopharmacology.
[27] E. Fernandes,et al. Metabolism is required for the expression of ecstasy-induced cardiotoxicity in vitro. , 2004, Chemical research in toxicology.
[28] R. de la Torre,et al. Human pharmacology of MDMA: pharmacokinetics, metabolism, and disposition. , 2004, Therapeutic drug monitoring.
[29] M. C. Brañes,et al. Plasma membrane channels formed by connexins: their regulation and functions. , 2003, Physiological reviews.
[30] M. Colado,et al. The Pharmacology and Clinical Pharmacology of 3,4-Methylenedioxymethamphetamine (MDMA, “Ecstasy”) , 2003, Pharmacological Reviews.
[31] K. Varner,et al. Changes in cardiovascular responsiveness and cardiotoxicity elicited during binge administration of Ecstasy. , 2002, The Journal of pharmacology and experimental therapeutics.
[32] F. Fornai,et al. Ecstasy during loud noise exposure induces dramatic ultrastructural changes in the heart. , 2002, Pharmacology & toxicology.
[33] M. Landry. MDMA: A Review of Epidemiological Data , 2002, Journal of psychoactive drugs.
[34] P A Insel,et al. Protein kinase C isozymes and the regulation of diverse cell responses. , 2000, American journal of physiology. Lung cellular and molecular physiology.
[35] P. O'cain,et al. Cardiovascular and sympathetic responses and reflex changes elicited by MDMA , 2000, Physiology & Behavior.
[36] P. Lampe,et al. Phosphorylation of Connexin43 on Serine368 by Protein Kinase C Regulates Gap Junctional Communication , 2000, The Journal of cell biology.
[37] E. Hertzberg,et al. Use of antibodies in the analysis of connexin 43 turnover and phosphorylation. , 2000, Methods.
[38] R. de la Torre,et al. Cardiovascular and neuroendocrine effects and pharmacokinetics of 3, 4-methylenedioxymethamphetamine in humans. , 1999, The Journal of pharmacology and experimental therapeutics.
[39] G. Beitia,et al. 3,4-Methylenedioxymethamphetamine (ecstasy)-induced hepatotoxicity: effect on cytosolic calcium signals in isolated hepatocytes. , 1999, Liver.
[40] A. Maio,et al. Downregulation of connexin 43 gene expression in rat heart during inflammation. The role of tumour necrosis factor. , 1999, Cytokine.
[41] S. Dhein. Gap junction channels in the cardiovascular system: pharmacological and physiological modulation. , 1998, Trends in pharmacological sciences.
[42] P. Parker,et al. The extended protein kinase C superfamily. , 1998, The Biochemical journal.
[43] A. Nairn,et al. Phosphorylation of connexin43 and the regulation of neonatal rat cardiac myocyte gap junctions. , 1997, Journal of molecular and cellular cardiology.
[44] A. Forrest,et al. Pathology of deaths associated with "ecstasy" and "eve" misuse. , 1996, Journal of clinical pathology.
[45] G. Goodwin,et al. Review of the pharmacology and clinical pharmacology of 3,4-methylenedioxymethamphetamine (MDMA or “Ecstasy”) , 1995, Psychopharmacology.
[46] K. Willecke,et al. Specific permeability and selective formation of gap junction channels in connexin-transfected HeLa cells , 1995, The Journal of cell biology.
[47] S. Dawling,et al. Toxicity and deaths from 3,4-methylenedioxymethamphetamine ("ecstasy") , 1992, The Lancet.
[48] E. Azmitia,et al. MDMA (ecstasy) effects on cultured serotonergic neurons: evidence for Ca2+-dependent toxicity linked to release , 1990, Brain Research.
[49] B. Yamamoto,et al. Acute and subchronic effects of methylenedioxymethamphetamine [(±)MDMA] on locomotion and serotonin syndrome behavior in the rat , 1989, Pharmacology Biochemistry and Behavior.
[50] R. Riemersma,et al. "Ecstasy" and sudden cardiac death. , 1988, The American journal of forensic medicine and pathology.
[51] M. Geyer,et al. Stimulant and hallucinogenic behavioral profiles of 3,4-methylenedioxymethamphetamine and N-ethyl-3,4-methylenedioxyamphetamine in rats. , 1988, The Journal of pharmacology and experimental therapeutics.
[52] R. Bost,et al. 'Eve' and 'Ecstasy'. A report of five deaths associated with the use of MDEA and MDMA. , 1987, JAMA.
[53] B. Horecker,et al. Binding of protein kinase C to neutrophil membranes in the presence of Ca2+ and its activation by a Ca2+-requiring proteinase. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[54] R. Rothman,et al. Neural and cardiac toxicities associated with 3,4-methylenedioxymethamphetamine (MDMA). , 2009, International review of neurobiology.
[55] N. Tribulova,et al. Myocardial gap junctions: targets for novel approaches in the prevention of life-threatening cardiac arrhythmias. , 2008, Physiological research.
[56] S. Beebe,et al. 3,4-Methylenedioxymethamphetamine activates nuclear factor-κB, increases intracellular calcium, and modulates gene transcription in rat heart cells , 2007, Cardiovascular Toxicology.
[57] C. Sterk,et al. Ecstasy and gateway drugs: initiating the use of ecstasy and other drugs. , 2007, Annals of epidemiology.
[58] Robert D. Goldman,et al. Cells: a laboratory manual , 1997 .
[59] R Kemler,et al. N-cadherin in adult rat cardiomyocytes in culture. II. Spatio-temporal appearance of proteins involved in cell-cell contact and communication. Formation of two distinct N-cadherin/catenin complexes. , 1996, Journal of cell science.
[60] W. Slikker,et al. Behavioral and neurochemical effects of orally administered MDMA in the rodent and nonhuman primate. , 1989, Neurotoxicology.