Improved guinea-pig ventricular cell model incorporating a diadic space, IKr and IKs, and length- and tension-dependent processes.
暂无分享,去创建一个
D. Noble | D. Noble | P. Kohl | P. Noble | A. Varghese
[1] J. Koch-weser,et al. THE INFLUENCE OF THE INTERVAL BETWEEN BEATS ON MYOCARDIAL CONTRACTILITY. , 1963, Pharmacological reviews.
[2] B. Katzung,et al. Time- and voltage-dependent interactions of antiarrhythmic drugs with cardiac sodium channels. , 1977, Biochimica et biophysica acta.
[3] D. Noble,et al. Excitation-contraction coupling and extracellular calcium transients in rabbit atrium: reconstruction of basic cellular mechanisms , 1987, Proceedings of the Royal Society of London. Series B. Biological Sciences.
[4] D. Noble,et al. A model of the single atrial cell: relation between calcium current and calcium release , 1990, Proceedings of the Royal Society of London. B. Biological Sciences.
[5] M. Sanguinetti,et al. Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents , 1990, The Journal of general physiology.
[6] D. Noble,et al. The Role of Sodium ‐ Calcium Exchange during the Cardiac Action Potential a , 1991, Annals of the New York Academy of Sciences.
[7] P. Gage,et al. A persistent sodium current in rat ventricular myocytes. , 1992, The Journal of physiology.
[8] R L Winslow,et al. Generation and propagation of ectopic beats induced by spatially localized Na–K pump inhibition in atrial network models , 1993, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[9] Y. Lai,et al. Generation and propagation of normal and abnormal pacemaker activity in network models of cardiac sinus node and atrium , 1995 .
[10] M. Boyett,et al. The role of the Na(+)‐Ca2+ exchanger in the rate‐dependent increase in contraction in guinea‐pig ventricular myocytes. , 1995, The Journal of physiology.
[11] D A Terrar,et al. Separation of the components of the delayed rectifier potassium current using selective blockers of IKr and IKs in guinea‐pig isolated ventricular myocytes , 1996, Experimental physiology.
[12] G. Gintant,et al. Two components of delayed rectifier current in canine atrium and ventricle. Does IKs play a role in the reverse rate dependence of class III agents? , 1996, Circulation research.
[13] D A Terrar,et al. The deactivation kinetics of the delayed rectifier components IKr and IKs in guinea‐pig isolated ventricular myocytes , 1996, Experimental physiology.
[14] M. Cannell,et al. Ca2+ influx during the cardiac action potential in guinea pig ventricular myocytes. , 1996, Circulation research.
[15] G. Langer,et al. Calcium concentration and movement in the diadic cleft space of the cardiac ventricular cell. , 1996, Biophysical journal.
[16] K. Linz,et al. Modulation of L-type calcium current by internal potassium in guinea pig ventricular myocytes. , 1997, Cardiovascular research.
[17] D. Noble,et al. Modeling of internal pH, ion concentration, and bioenergetic changes during myocardial ischemia. , 1997, Advances in experimental medicine and biology.
[18] P Kohl,et al. Cellular mechanisms of cardiac mechano-electric feedback in a mathematical model. , 1998, The Canadian journal of cardiology.
[19] D Noble,et al. Modelling of sodium-overload arrhythmias and their suppression. , 1998, The Canadian journal of cardiology.