Calcium responses in human fibroblasts: A diagnostic molecular profile for Alzheimer's disease
暂无分享,去创建一个
Daniel L. Alkon | Giuliano Binetti | Marco Racchi | Stefano Govoni | D. Alkon | G. Binetti | M. Racchi | S. Govoni | N. Hirashima | Fiorenzo Battaini | Naohide Hirashima | René Etcheberrigaray | Stefania Bergamaschi | R. Etcheberrigaray | S. Bergamaschi | F. Battaini
[1] S. Matsuyama,et al. Tetraethylammonium-induced calcium concentration changes in skin fibroblasts from patients with Alzheimer disease. , 1995, Dementia.
[2] D. Alkon,et al. Alzheimer and beta-amyloid-treated fibroblasts demonstrate a decrease in a memory-associated GTP-binding protein, Cp20. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[3] G. Gibson,et al. Increased Inositol 1,4,5‐Trisphosphate Accumulation Correlates with an Up‐Regulation of Bradykinin Receptors in Alzheimer's Disease , 1995, Journal of neurochemistry.
[4] M. Takeda,et al. Cell-Cycle-Dependent Abnormal Calcium Response in Fibroblasts from Patients with Familial Alzheimer's Disease , 1995 .
[5] D. Alkon,et al. Use of Cultured Fibroblasts in Elucidating the Pathophysiology and Diagnosis of Alzheimer's Disease a , 1994, Annals of the New York Academy of Sciences.
[6] D. Alkon,et al. Molecular Mechanisms of Memory and the Pathophysiology of Alzheimer's Disease , 1994, Annals of the New York Academy of Sciences.
[7] D. Alkon,et al. Characterization of a GTP-binding protein implicated in both memory storage and interorganelle vesicle transport. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[8] D L Alkon,et al. Soluble beta-amyloid induction of Alzheimer's phenotype for human fibroblast K+ channels. , 1994, Science.
[9] K. Oka,et al. Internal Ca2+ mobilization is altered in fibroblasts from patients with Alzheimer disease. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[10] W. Wetsel,et al. Reduced Protein-Kinase-C Immunoreactivity In Fibroblasts From Patients With Alzheimers-Disease , 1994 .
[11] G. Binetti,et al. Cytosol protein kinase C downregulation in fibroblasts from Alzheimer's disease patients , 1993, Neurology.
[12] R. Mullins,et al. Serum- and bradykinin-induced calcium transients in familial Alzheimer's fibroblasts , 1993, Neurobiology of Aging.
[13] D. Alkon,et al. Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[14] E. Villacres,et al. Intracellular calcium response is reduced in CD4+ lymphocytes in Alzheimer's disease and in older persons with down's syndrome , 1993, Neurobiology of Aging.
[15] R. Scott. Extraneuronal Manifestations of Alzheimer's Disease , 1993, Journal of the American Geriatrics Society.
[16] E. Mignot,et al. Platelet cytosolic free-calcium concentration is increased in aging and Alzheimer's disease , 1993, Biological Psychiatry.
[17] D. Alkon,et al. Classical conditioning and protein kinase C activation regulate the same single potassium channel in Hermissenda crassicornis photoreceptors. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[18] F. Maxfield,et al. Resting [Ca2+]i and [Ca2+]i transients are similar in fibroblasts from normal and Alzheimer's donors , 1992, Neurobiology of Aging.
[19] D. Kyle,et al. Recent developments in the understanding of bradykinin receptors. , 1992, Life sciences.
[20] J. Hall,et al. Bradykinin receptors: pharmacological properties and biological roles. , 1992, Pharmacology & therapeutics.
[21] H. Thaler,et al. Inositol phosphates and intracellular calcium after bradykinin stimulation in fibroblasts from young, normal aged and Alzheimer donors , 1991, Neurobiology of Aging.
[22] A. Adunsky,et al. Increased cytosolic free calcium in lymphocytes of Alzheimer patients , 1991, Journal of Neuroimmunology.
[23] L. Toral-Barza,et al. Interactions between inositol phosphates and cytosolic free calcium following bradykinin stimulation in cultured human skin fibroblasts. , 1991, Biochimica et biophysica acta.
[24] D L Alkon,et al. Isolation of a G protein that is modified by learning and reduces potassium currents in Hermissenda. , 1990, Science.
[25] D L Alkon,et al. Memory storage and neural systems. , 1989, Scientific American.
[26] J. Goldman,et al. Altered response of fibroblasts from aged and Alzheimer donors to drugs that elevate cytosolic free calcium , 1988, Neurobiology of Aging.
[27] Daniel L. Alkon,et al. Memory Traces in the Brain , 1987 .
[28] J. Blass,et al. Diminished mitogen-induced calcium uptake by lymphocytes from alzheimer patients , 1987, Biological Psychiatry.
[29] J. Goldman,et al. Cytosolic free calcium and cell spreading decrease in fibroblasts from aged and Alzheimer donors. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[30] M. Schork,et al. Statistics With Applications to the Biological and Health Sciences. , 1971 .
[31] L. Nee,et al. A family with histologically confirmed Alzheimer's disease. , 1983, Archives of neurology.