Myocardial performance and free energy of ATP-hydrolysis in isolated rat hearts during graded hypoxia, reoxygenation and high Ke+-perfusion.

[1]  E. Braunwald,et al.  Myocardial High Energy Phosphate Stores in Acutely Induced Hypoxic Heart Failure , 1966, Circulation research.

[2]  P. Mathes,et al.  Functional compartmentation of ATP and creatine phosphate in heart muscle. , 1970, Journal of molecular and cellular cardiology.

[3]  F. Kavaler,et al.  Positive and Negative Inotropic Effects of Elevated Extracellular Potassium Level on Mammalian Ventricular Muscle , 1972, The Journal of general physiology.

[4]  O. H. Bing,et al.  PO2-modulated performance of cardiac muscle. , 1976, The American journal of physiology.

[5]  A. Katz,et al.  Mechanism of early "pump" failure of the ischemic heart: possible role of adenosine triphosphate depletion and inorganic phosphate accumulation. , 1977, The American journal of cardiology.

[6]  D. Ellis The effects of external cations and ouabain on the intracellular sodium activity of sheep heart Purkinje fibres , 1977, The Journal of physiology.

[7]  B. Chance,et al.  Heterogeneity of the Hypoxic State in Perfused Rat Heart , 1977, Circulation research.

[8]  M D Payet,et al.  Slow inward and outward currents of rat ventricular fibers under anoxia. , 1978, Journal de physiologie.

[9]  G. Peterson,et al.  A simplified method for analysis of inorganic phosphate in the presence of interfering substances. , 1978, Analytical biochemistry.

[10]  P. Poole‐Wilson,et al.  Hypoxia and calcium. , 1979, Journal of molecular and cellular cardiology.

[11]  D. Hearse Oxygen deprivation and early myocardial contractile failure: a reassessment of the possible role of adenosine triphosphate. , 1979, The American journal of cardiology.

[12]  H. Kammermeier,et al.  Relationship of phosphorylation potential and oxygen consumption in isolated perfused rat hearts. , 1980, Journal of molecular and cellular cardiology.

[13]  P. Poole‐Wilson,et al.  Tissue acidosis in myocardial hypoxia. , 1980, Journal of molecular and cellular cardiology.

[14]  H. Kammermeier,et al.  Rapid assay of adenine nucleotides or creatine compounds in extracts of cardiac tissue by paired-ion reverse-phase high-performance liquid chromatography. , 1980, Analytical biochemistry.

[15]  E. Ritman,et al.  Biphasic Changes in Maximum Relaxation Rate during Progressive Hypoxia in Isometric Kitten Papillary Muscle and Isovolumic Rabbit Ventricle , 1980, Circulation research.

[16]  Michiel J. Janse,et al.  Comparison of the Effect of the Regional Ischemia, Hypoxia, Hyperkalemia, and Acidosis on Intracellular and Extracellular Potentials and Metabolism in the Isolated Porcine Heart , 1980, Circulation research.

[17]  N. Paradise,et al.  Criteria for adequate oxygenation of isometric kitten papillary muscle. , 1981, The American journal of physiology.

[18]  A. Katz,et al.  Low concentrations of fatty acids can inhibit calcium efflux from sarcoplasmic reticulum vesicles. , 1981, Life sciences.

[19]  E Jüngling,et al.  Free energy change of ATP-hydrolysis: a causal factor of early hypoxic failure of the myocardium? , 1982, Journal of molecular and cellular cardiology.

[20]  H. Fozzard,et al.  Transmembrane Na+ and Ca2+ electrochemical gradients in cardiac muscle and their relationship to force development , 1982, The Journal of general physiology.

[21]  Kapel'ko Vi,et al.  Comparative evaluation of contraction and relaxation of isolated heart muscle with decreased calcium concentration in the perfusate, acidosis, and metabolic blockade. , 1982 .

[22]  D. Allen,et al.  Intracellular calcium concentration during hypoxia and metabolic inhibition in mammalian ventricular muscle. , 1983, The Journal of physiology.

[23]  R. Coronel,et al.  The change of the free energy of ATP hydrolysis during global ischemia and anoxia in the rat heart. Its possible role in the regulation of transsarcolemmal sodium and potassium gradients. , 1984, Journal of molecular and cellular cardiology.

[24]  D Durrer,et al.  Combined effects of hypoxia, hyperkalemia and acidosis on membrane action potential and excitability of guinea-pig ventricular muscle. , 1984, Journal of molecular and cellular cardiology.

[25]  D. Allen,et al.  Measurements of intracellular calcium concentration in heart muscle: the effects of inotropic interventions and hypoxia. , 1984, Journal of molecular and cellular cardiology.

[26]  D. Spray,et al.  Regulation of gap junctional conductance. , 1985, The American journal of physiology.

[27]  J. Reeves The Sarcolemmal Sodium-Calcium Exchange System , 1985 .

[28]  J. Kentish The effects of inorganic phosphate and creatine phosphate on force production in skinned muscles from rat ventricle. , 1986, The Journal of physiology.

[29]  A. Wilde,et al.  The Combined Effects of Hypoxia, High K+, and Acidosis on the Intracellular Sodium Activity and Resting Potential in Guinea Pig Papillary Muscle , 1986, Circulation research.

[30]  S. Sheu,et al.  Na+-Ca2+ exchange contributes to increase of cytosolic Ca2+ concentration during depolarization in heart muscle. , 1986, The American journal of physiology.

[31]  D. Allen,et al.  Is force production in the myocardium directly dependent upon the free energy change of ATP hydrolysis? , 1986, Journal of molecular and cellular cardiology.

[32]  T. Aw,et al.  Mitochondrial transmembrane ion distribution during anoxia. , 1987, The American journal of physiology.

[33]  H. Kammermeier Interrelationship between the free energy change of ATP-hydrolysis, cytosolic inorganic phosphate and cardiac performance during hypoxia and reoxygenation. , 1987, Biomedica biochimica acta.

[34]  H. Kammermeier High energy phosphate of the myocardium: concentration versus free energy change. , 1987, Basic research in cardiology.