Glutathione influences the proliferation as well as the extent of mitochondrial activation in rat splenocytes.

[1]  M. Smyth.  Glutathione modulates activation-dependent proliferation of human peripheral blood lymphocyte populations without regulating their activated function. , 1991, Journal of immunology.

[2]  P. Rabinovitch,et al.  Proliferative capacity of human peripheral blood lymphocytes sorted on the basis of glutathione content , 1990, Journal of cellular physiology.

[3]  S. Arkins,et al.  The proliferative response of rat T cells to calcium ionophores increases with age. , 1990, Cellular immunology.

[4]  W. Dröge,et al.  Interleukin-2 mRNA expression, lymphokine production and DNA synthesis in glutathione-depleted T cells. , 1990, Cellular immunology.

[5]  M. Ratinaud,et al.  A new method for testing cell ageing using two mitochondria specific fluorescent probes , 1990, Mechanisms of Ageing and Development.

[6]  P. Zelarney,et al.  Lymphocyte proliferation in glutathione-depleted lymphocytes: direct relationship between glutathione availability and the proliferative response. , 1989, Immunopharmacology.

[7]  D. Lawrence,et al.  Cell cycle progression of glutathione-depleted human peripheral blood mononuclear cells is inhibited at S phase. , 1989, Journal of immunology.

[8]  S. Liang,et al.  Glutathione regulates interleukin-2 activity on cytotoxic T-cells. , 1989, The Journal of biological chemistry.

[9]  M. Ratinaud,et al.  In situ flow cytometric analysis of nonyl acridine orange-stained mitochondria from splenocytes. , 1988, Cytometry.

[10]  T. Ishii,et al.  Regulation of glutathione levels in mouse spleen lymphocytes by transport of cysteine , 1987, Journal of cellular physiology.

[11]  D. Lawrence,et al.  Modulation of intracellular glutathione concentrations alters lymphocyte activation and proliferation. , 1987, Experimental cell research.

[12]  S. Andreoli,et al.  Role of glutathione in protecting endothelial cells against hydrogen peroxide oxidant injury. , 1986, The Journal of laboratory and clinical medicine.

[13]  X. Ronot,et al.  Mitochondrial uptake of rhodamine 123 by rabbit articular chondrocytes. , 1986, Cytometry.

[14]  D. J. Reed.  Regulation of reductive processes by glutathione. , 1986, Biochemical pharmacology.

[15]  J. J. Nadakavukaren,et al.  Increased rhodamine 123 uptake by carcinoma cells. , 1985, Cancer research.

[16]  M. Edgren,et al.  Glutathione requirement for the rejoining of radiation-induced DNA breaks in misonidazole-treated cells. , 1985, International journal of radiation biology and related studies in physics, chemistry, and medicine.

[17]  A. Meister.  Methods for the selective modification of glutathione metabolism and study of glutathione transport. , 1985, Methods in enzymology.

[18]  L. Harker,et al.  Glutathione redox cycle protects cultured endothelial cells against lysis by extracellularly generated hydrogen peroxide. , 1984, The Journal of clinical investigation.

[19]  M. Varnes,et al.  The role of thiols in cellular response to radiation and drugs. , 1983, Radiation research.

[20]  T. W. James,et al.  Proliferation of mitochondria during the cell cycle of the human cell line (HL-60) , 1981, The Journal of cell biology.

[21]  D. Chaplin,et al.  Inhibition of lectin-induced lymphocyte activation by diamide and other sulfhydryl reagents. , 1978, Cellular immunology.

[22]  L. Bratteby,et al.  Labelling of red blood cells in vitro with small amounts of di-iso-propyl-flurophosphonate (DF32P). , 1968, Scandinavian journal of clinical and laboratory investigation.