Disturbed brain phospholipid and docosahexaenoic acid metabolism in calcium-independent phospholipase A(2)-VIA (iPLA(2)β)-knockout mice.
暂无分享,去创建一个
S. Rapoport | D. Greenstein | J. Turk | A. Taha | Kaizong Ma | M. Igarashi | M. Wohltmann | Lisa Chang | Hyung-wook Kim | Yewon Cheon | Hiren R Modi
[1] S. Rapoport,et al. Dietary n-6 PUFA deprivation downregulates arachidonate but upregulates docosahexaenoate metabolizing enzymes in rat brain. , 2011, Biochimica et biophysica acta.
[2] S. Rapoport,et al. Imaging decreased brain docosahexaenoic acid metabolism and signaling in iPLA2β (VIA)-deficient mice , 2010, Journal of Lipid Research.
[3] T. Hashimoto,et al. Phenotypic spectrum of patients with PLA2G6 mutation and PARK14-linked parkinsonism , 2010, Neurology.
[4] A. A. Farooqui,et al. Involvement of cytosolic phospholipase A(2), calcium independent phospholipase A(2) and plasmalogen selective phospholipase A(2) in neurodegenerative and neuropsychiatric conditions. , 2010, Current medicinal chemistry.
[5] J. Turk,et al. Endothelial cell prostaglandin I(2) and platelet-activating factor production are markedly attenuated in the calcium-independent phospholipase A(2)beta knockout mouse. , 2010, Biochemistry.
[6] L. Bubacco,et al. Molecular insights into the interaction between alpha-synuclein and docosahexaenoic acid. , 2009, Journal of molecular biology.
[7] Xianlin Han,et al. Genetic Ablation of Calcium-independent Phospholipase A2γ Leads to Alterations in Hippocampal Cardiolipin Content and Molecular Species Distribution, Mitochondrial Degeneration, Autophagy, and Cognitive Dysfunction* , 2009, The Journal of Biological Chemistry.
[8] S. Rapoport,et al. Intracellular- and extracellular-derived Ca(2+) influence phospholipase A(2)-mediated fatty acid release from brain phospholipids. , 2009, Biochimica et biophysica acta.
[9] S. Rapoport,et al. Dietary n-6 PUFA deprivation for 15 weeks reduces arachidonic acid concentrations while increasing n-3 PUFA concentrations in organs of post-weaning male rats. , 2009, Biochimica et biophysica acta.
[10] F. Calon,et al. Rapid beta-oxidation of eicosapentaenoic acid in mouse brain: an in situ study. , 2009, Prostaglandins, leukotrienes, and essential fatty acids.
[11] E. Bertini,et al. Neurodegeneration associated with genetic defects in phospholipase A2 , 2008, Neurology.
[12] S. Sakoda,et al. Neuroaxonal Dystrophy Caused by Group VIA Phospholipase A2 Deficiency in Mice: A Model of Human Neurodegenerative Disease , 2008, The Journal of Neuroscience.
[13] Xianlin Han,et al. Genetic Ablation of Calcium-independent Phospholipase A2γ Leads to Alterations in Mitochondrial Lipid Metabolism and Function Resulting in a Deficient Mitochondrial Bioenergetic Phenotype* , 2007, Journal of Biological Chemistry.
[14] N. Salem,et al. An n-3 fatty acid deficient diet affects mouse spatial learning in the Barnes circular maze. , 2007, Prostaglandins, leukotrienes, and essential fatty acids.
[15] G. Reiser,et al. Prostaglandin synthesis in rat brain astrocytes is under the control of the n‐3 docosahexaenoic acid, released by group VIB calcium‐independent phospholipase A2 , 2007, Journal of neurochemistry.
[16] S. Rapoport,et al. Altered brain lipid composition in cyclooxygenase-2 knockout mouse Published, JLR Papers in Press, January 3, 2007. , 2007, Journal of Lipid Research.
[17] S. Rapoport,et al. Dietary n-3 PUFA deprivation alters expression of enzymes of the arachidonic and docosahexaenoic acid cascades in rat frontal cortex , 2007, Molecular Psychiatry.
[18] N. Færgeman,et al. α‐Synuclein gene ablation increases docosahexaenoic acid incorporation and turnover in brain phospholipids , 2006, Journal of neurochemistry.
[19] Edward A Dennis,et al. The phospholipase A2 superfamily and its group numbering system. , 2006, Biochimica et biophysica acta.
[20] Rachel Levy,et al. PLA2G6 mutation underlies infantile neuroaxonal dystrophy. , 2006, American journal of human genetics.
[21] F. Bosetti,et al. Cyclooxygenase‐1 and ‐2 enzymes differentially regulate the brain upstream NF‐κB pathway and downstream enzymes involved in prostaglandin biosynthesis , 2006, Journal of neurochemistry.
[22] G. Cole,et al. Docosahexaenoic Acid Protects from Amyloid and Dendritic Pathology in an Alzheimer's Disease Mouse Model , 2006, Nutrition and health.
[23] Colin A. Johnson,et al. PLA2G6, encoding a phospholipase A2, is mutated in neurodegenerative disorders with high brain iron , 2006, Nature Genetics.
[24] Kaizong Ma,et al. Resting and arecoline‐stimulated brain metabolism and signaling involving arachidonic acid are altered in the cyclooxygenase‐2 knockout mouse , 2006, Journal of neurochemistry.
[25] S. Zhang,et al. Effects of Stable Suppression of Group VIA Phospholipase A2 Expression on Phospholipid Content and Composition, Insulin Secretion, and Proliferation of INS-1 Insulinoma Cells* , 2006, Journal of Biological Chemistry.
[26] S. Rapoport,et al. One generation of n-3 polyunsaturated fatty acid deprivation increases depression and aggression test scores in rats Published, JLR Papers in Press, October 6, 2005. , 2006, Journal of Lipid Research.
[27] A. A. Farooqui,et al. Distribution of calcium-independent phospholipase A2 (iPLA2) in monkey brain , 2005, Journal of neurocytology.
[28] E. Dennis,et al. Distinguishing phospholipase A2 types in biological samples by employing group-specific assays in the presence of inhibitors. , 2005, Prostaglandins & other lipid mediators.
[29] S. Rapoport,et al. Rapid High-Energy Microwave Fixation is Required to Determine the Anandamide (N-arachidonoylethanolamine) Concentration of Rat Brain , 2005, Neurochemical Research.
[30] M. Creer,et al. Calcium-independent phospholipase A2 is regulated by a novel protein kinase C in human coronary artery endothelial cells. , 2005, American journal of physiology. Cell physiology.
[31] Robert Langenbach,et al. Prostaglandin E2 and microsomal prostaglandin E synthase‐2 expression are decreased in the cyclooxygenase‐2‐deficient mouse brain despite compensatory induction of cyclooxygenase‐1 and Ca2+‐dependent phospholipase A2 , 2004, Journal of neurochemistry.
[32] J. Bell,et al. Half‐lives of docosahexaenoic acid in rat brain phospholipids are prolonged by 15 weeks of nutritional deprivation of n‐3 polyunsaturated fatty acids , 2004, Journal of neurochemistry.
[33] G. Reiser,et al. Role of Ca2+-independent phospholipase A2 and n−3 polyunsaturated fatty acid docosahexaenoic acid in prostanoid production in brain: perspectives for protection in neuroinflammation , 2004, International Journal of Developmental Neuroscience.
[34] David J. Miller,et al. Male Mice That Do Not Express Group VIA Phospholipase A2 Produce Spermatozoa with Impaired Motility and Have Greatly Reduced Fertility* , 2004, Journal of Biological Chemistry.
[35] Takashi Morihara,et al. Docosahexaenoic Acid Protects from Dendritic Pathology in an Alzheimer's Disease Mouse Model , 2004, Neuron.
[36] L. Horrocks,et al. Docosahexaenoic acid in the diet: its importance in maintenance and restoration of neural membrane function. , 2004, Prostaglandins, leukotrienes, and essential fatty acids.
[37] F. Bosetti,et al. The expression of brain cyclooxygenase‐2 is down‐regulated in the cytosolic phospholipase A2 knockout mouse , 2003, Journal of neurochemistry.
[38] G. Reiser,et al. Docosahexaenoic acid and arachidonic acid release in rat brain astrocytes is mediated by two separate isoforms of phospholipase A2 and is differently regulated by cyclic AMP and Ca2+ , 2003, British journal of pharmacology.
[39] C. Serhan,et al. Novel Docosatrienes and 17S-Resolvins Generated from Docosahexaenoic Acid in Murine Brain, Human Blood, and Glial Cells , 2003, The Journal of Biological Chemistry.
[40] S. Rapoport,et al. Brain lipid metabolism in the cPLA2 knockout mouse Published, JLR Papers in Press, October 1, 2002. DOI 10.1194/jlr.M200298-JLR200 , 2003, Journal of Lipid Research.
[41] S. Rapoport,et al. Recent studies on interactions between n-3 and n-6 polyunsaturated fatty acids in brain and other tissues , 2002, Current opinion in lipidology.
[42] Thomas D. Schmittgen,et al. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.
[43] Stanley I. Rapoport,et al. In vivo fatty acid incorporation into brain phosholipids in relation to plasma availability, signal transduction and membrane remodeling , 2001, Journal of Molecular Neuroscience.
[44] R. Gross,et al. Identification of the Calmodulin-binding Domain of Recombinant Calcium-independent Phospholipase A2β , 2001, The Journal of Biological Chemistry.
[45] T. Perlmann,et al. Docosahexaenoic acid, a ligand for the retinoid X receptor in mouse brain. , 2000, Science.
[46] S. Rapoport,et al. Nutritional Deprivation of α‐Linolenic Acid Decreases but Does Not Abolish Turnover and Availability of Unacylated Docosahexaenoic Acid and Docosahexaenoyl‐CoA in Rat Brain , 2000, Journal of neurochemistry.
[47] E. Dennis,et al. The expanding superfamily of phospholipase A(2) enzymes: classification and characterization. , 2000, Biochimica et biophysica acta.
[48] E. Dennis,et al. Group-specific assays that distinguish between the four major types of mammalian phospholipase A2. , 1999, Analytical biochemistry.
[49] J. Bell,et al. Dynamics of Docosahexaenoic Acid Metabolism in the Central Nervous System: Lack of Effect of Chronic Lithium Treatment , 1999, Neurochemical Research.
[50] M. Murakami,et al. Functional Coupling Between Various Phospholipase A2s and Cyclooxygenases in Immediate and Delayed Prostanoid Biosynthetic Pathways* , 1999, The Journal of Biological Chemistry.
[51] Hee-Yong Kim,et al. Mobilization of arachidonate and docosahexaenoate by stimulation of the 5-HT2A receptor in rat C6 glioma cells , 1997, Brain Research.
[52] S. Rapoport,et al. Relation Between Free Fatty Acid and Acyl-CoA Concentrations in Rat Brain Following Decapitation , 1997, Neurochemical Research.
[53] S. Rapoport,et al. Evidence for the Involvement of Docosahexaenoic Acid in Cholinergic Stimulated Signal Transduction at the Synapse , 1997, Neurochemical Research.
[54] C. Bruehl,et al. Polyunsaturated fatty acids modulate sodium and calcium currents in CA1 neurons. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[55] S. Rapoport,et al. Isolation and quantitation of long-chain acyl-coenzyme A esters in brain tissue by solid-phase extraction. , 1994, Analytical biochemistry.
[56] S. Rapoport,et al. Brain Arachidonic Acid Incorporation and Precursor Pool Specific Activity During Intravenous Infusion of Unesterified [3H]Arachidonate in the Anesthetized Rat , 1994, Journal of neurochemistry.
[57] E. Dennis,et al. Ca(2+)-independent cytosolic phospholipase A2 from macrophage-like P388D1 cells. Isolation and characterization. , 1994, The Journal of biological chemistry.
[58] S. Rapoport,et al. A quantitative method for measuring regional in vivo fatty-acid incorporation into and turnover within brain phospholipids: review and critical analysis , 1992, Brain Research Reviews.
[59] T Nariai,et al. Arecoline‐Stimulated Brain Incorporation of Intravenously Administered Fatty Acids in Unanesthetized Rats , 1991, Journal of neurochemistry.
[60] R. Smith,et al. Evaluation of direct saponification method for determination of cholesterol in meats. , 1986, Journal - Association of Official Analytical Chemists.
[61] M. M. Bradford. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.
[62] J. Folch,et al. A simple method for the isolation and purification of total lipides from animal tissues. , 1957, The Journal of biological chemistry.
[63] J. Turk,et al. The molecular biology of the group VIA Ca2+-independent phospholipase A2. , 2001, Progress in nucleic acid research and molecular biology.