Selenium effects on prostate cell growth.
暂无分享,去创建一个
[1] H. Ganther,et al. In vitro and in vivo studies of methylseleninic acid: evidence that a monomethylated selenium metabolite is critical for cancer chemoprevention. , 2000, Cancer research.
[2] D. Kalvakolanu,et al. The interferon-beta and tamoxifen combination induces apoptosis using thioredoxin reductase. , 2000, Biochimica et biophysica acta.
[3] H. Ganther,et al. Selenium modulation of cell proliferation and cell cycle biomarkers in normal and premalignant cells of the rat mammary gland. , 2000, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[4] H. Ganther,et al. Selenium metabolism, selenoproteins and mechanisms of cancer prevention: complexities with thioredoxin reductase. , 1999, Carcinogenesis.
[5] S. Byers,et al. Exogenous Expression of β-Catenin Regulates Contact Inhibition, Anchorage-Independent Growth, Anoikis, and Radiation-Induced Cell Cycle Arrest , 1999, Journal of Cell Biology.
[6] A G Yakovlev,et al. Role of Poly(ADP-ribose) Polymerase (PARP) Cleavage in Apoptosis , 1999, The Journal of Biological Chemistry.
[7] N. Taniguchi,et al. Functional expression of rat thioredoxin reductase: selenocysteine insertion sequence element is essential for the active enzyme. , 1999, The Biochemical journal.
[8] C. Ong,et al. Sodium selenite‐induced oxidative stress and apoptosis in human hepatoma HepG2 cells , 1999, International journal of cancer.
[9] P. Russell,et al. Cdc25 inhibited in vivo and in vitro by checkpoint kinases Cds1 and Chk1. , 1999, Molecular biology of the cell.
[10] S. Snyder,et al. Poly (ADP-ribose) polymerase, nitric oxide and cell death. , 1999, Trends in pharmacological sciences.
[11] V. Soldatenkov,et al. Detection of heterogeneity of apoptotic fragments of poly (ADP‐ribose) polymerase in MDA‐MB‐468 breast cancer cells: Two‐dimensional gel analysis , 1999, Electrophoresis.
[12] G. Powis,et al. Cloning, sequencing and functional expression of a novel human thioredoxin reductase , 1999, FEBS letters.
[13] G. Bijur,et al. Ascorbic acid‐dehydroascorbate induces cell cycle arrest at G2/M DNA damage checkpoint during oxidative stress , 1999, Environmental and molecular mutagenesis.
[14] K. Neldner,et al. Selenium compounds have disparate abilities to impose oxidative stress and induce apoptosis. , 1999, Free radical biology & medicine.
[15] M. Böcher,et al. mRNA stability and selenocysteine insertion sequence efficiency rank gastrointestinal glutathione peroxidase high in the hierarchy of selenoproteins. , 1999, European journal of biochemistry.
[16] S. Byers,et al. Exogenous Expression of b -Catenin Regulates Contact Inhibition, Anchorage-independent Growth, Anoikis, and Radiation-induced Cell Cycle Arrest , 1999 .
[17] C. Ip,et al. Lessons from basic research in selenium and cancer prevention. , 1998, The Journal of nutrition.
[18] R. Jagus,et al. Thioredoxin Reductase Mediates Cell Death Effects of the Combination of Beta Interferon and Retinoic Acid , 1998, Molecular and Cellular Biology.
[19] E. Rimm,et al. Study of prediagnostic selenium level in toenails and the risk of advanced prostate cancer. , 1998, Journal of the National Cancer Institute.
[20] W. Ansorge,et al. The cdc25B phosphatase is essential for the G2/M phase transition in human cells. , 1998, Journal of cell science.
[21] A. Yakovlev,et al. Irreversible binding of poly(ADP)ribose polymerase cleavage product to DNA ends revealed by atomic force microscopy: possible role in apoptosis. , 1998, Cancer research.
[22] J. Pietenpol,et al. Negative regulation of Wee1 expression and Cdc2 phosphorylation during p53-mediated growth arrest and apoptosis. , 1998, Cancer research.
[23] L. Li,et al. Extended survivability of prostate cancer cells in the absence of trophic factors: increased proliferation, evasion of apoptosis, and the role of apoptosis proteins. , 1998, Cancer research.
[24] R. Kramer,et al. E-cadherin Regulates Anchorage-independent Growth and Survival in Oral Squamous Cell Carcinoma Cells* , 1998, The Journal of Biological Chemistry.
[25] W. Peters,et al. Effects of dietary anticarcinogens on rat gastrointestinal glutathione peroxidase activity. , 1998, Oncology reports.
[26] A. Boulares,et al. Transient Poly(ADP-ribosyl)ation of Nuclear Proteins and Role of Poly(ADP-ribose) Polymerase in the Early Stages of Apoptosis* , 1998, The Journal of Biological Chemistry.
[27] D. Morgan,et al. Nuclear Localization of Cyclin B1 Controls Mitotic Entry After DNA Damage , 1998, The Journal of cell biology.
[28] B. Turnbull,et al. Decreased incidence of prostate cancer with selenium supplementation: results of a double-blind cancer prevention trial. , 1998, British journal of urology.
[29] L. Clark,et al. Inhibitory effect of selenomethionine on the growth of three selected human tumor cell lines. , 1998, Cancer letters.
[30] P. Filipcik,et al. Nuclear all-trans retinoic acid receptors: in vitro effects of selenium. , 1998, Biological trace element research.
[31] N. Kyprianou,et al. Transient tyrosine phosphorylation of p34cdc2 is an early event in radiation‐induced apoptosis of prostate cancer cells , 1997, The Prostate.
[32] D. Medina,et al. Inhibition of cdk2 kinase activity by methylselenocysteine in synchronized mouse mammary epithelial tumor cells. , 1997, Carcinogenesis.
[33] E. Cohen-Jonathan,et al. Preferential cytoplasmic localization of p34cdc2 in recurrent human squamous cell carcinoma after radiotherapy. , 1997, Radiation research.
[34] Bruce W. Turnbull,et al. Effects of Selenium Supplementation for Cancer Prevention in Patients With Carcinoma of the Skin: A Randomized Controlled Trial , 1996 .
[35] D. Medina,et al. Organic and inorganic selenium compounds inhibit mouse mammary cell growth in vitro by different cellular pathways. , 1996, Cancer letters.
[36] R. Woutersen,et al. CANCER BIOLOGY: Effects of dietary β-carotene and selenium on initiation and promotion of pancreatic carcinogenesis in azaserine-treated rats , 1996 .
[37] R. Woutersen,et al. Effects of dietary beta-carotene and selenium on initiation and promotion of pancreatic carcinogenesis in azaserine-treated rats. , 1996, Carcinogenesis.
[38] R. Muschel,et al. Effects of ionizing radiation on cell cycle progression , 1995, Radiation and environmental biophysics.
[39] W. Telford,et al. Quantification of apoptotic events in pure and heterogeneous populations of cells using the flow cytometer. , 1995, Methods in cell biology.
[40] W. Bursch,et al. Kinetics of apoptosis and secondary necrosis in cultured rat thymocytes and S.49 mouse lymphoma and CEM human leukemia cells. , 1994, Biochemistry and cell biology = Biochimie et biologie cellulaire.
[41] K. Wong,et al. Thanatogen expression during involution of the rat ventral prostate after castration. , 1994, Journal of andrology.
[42] L. Yan,et al. Generation of reactive oxygen species from the reaction of selenium compounds with thiols and mammary tumor cells. , 1993, Biochemical pharmacology.
[43] L. Kauppinen,et al. Effects of selenomethionine on cell growth and on S-adenosylmethionine metabolism in cultured malignant cells. , 1990, The Biochemical journal.
[44] G. Owens,et al. Cell cycle versus density dependence of smooth muscle alpha actin expression in cultured rat aortic smooth muscle cells , 1988, The Journal of cell biology.
[45] T. F. Parkinson,et al. Elemental content of vegetables and millet grown in potted soil amended with lignite fly ash , 1981, Archives of environmental contamination and toxicology.
[46] Y. Oshika,et al. P-glycoprotein-mediated acquired multidrug resistance of human lung cancer cells in vivo. , 1996, British Journal of Cancer.