Effects of Castanospermine on Inflammatory Response in a Rat Model of Experimental Severe Acute Pancreatitis.
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Liang Zhao | Weixing Wang | Yu-pu Hong | Wenyi Guo | Chen Chen | F. Mei | Ming-wei Xiang
[1] J. Iredale,et al. Kynurenine–3–monooxygenase inhibition prevents multiple organ failure in rodent models of acute pancreatitis , 2015, Nature Medicine.
[2] Kailiang Zhao,et al. Effects of thymosin β4 on a rat model of severe acute pancreatitis. , 2015, Experimental and therapeutic medicine.
[3] Guojian Yin,et al. Rosmarinic Acid Attenuates Sodium Taurocholate-Induced Acute Pancreatitis in Rats by Inhibiting Nuclear Factor-κB Activation. , 2015, The American journal of Chinese medicine.
[4] Ping Xu,et al. Central role of neutrophil in the pathogenesis of severe acute pancreatitis , 2015, Journal of cellular and molecular medicine.
[5] Wen-hong Deng,et al. Relationship between expression of NADPH oxidase 2 and invasion and prognosis of human gastric cancer. , 2015, World journal of gastroenterology.
[6] W. Mehal,et al. Inflammasomes in pancreatic physiology and disease. , 2015, American journal of physiology. Gastrointestinal and liver physiology.
[7] J. Werner,et al. Inflammatory profiling of early experimental necrotizing pancreatitis. , 2015, Life sciences.
[8] H. Lee,et al. Interleukin-6 is associated with obesity, central fat distribution, and disease severity in patients with acute pancreatitis. , 2015, Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.].
[9] C. Chen,et al. Inhibition of glycogen synthase kinase-3β attenuates acute kidney injury in sodium taurocholate‑induced severe acute pancreatitis in rats. , 2014, Molecular medicine reports.
[10] J. Tasu,et al. High plasma levels of the pro-inflammatory cytokine IL-22 and the anti-inflammatory cytokines IL-10 and IL-1ra in acute pancreatitis. , 2014, Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.].
[11] Hong-yun Li,et al. Thalidomide alleviates acute pancreatitis-associated lung injury via down-regulation of NFκB induced TNF-α. , 2014, Pathology, research and practice.
[12] A. Dabrowski,et al. Increased expression of the intercellular adhesion molecule-1 (ICAM-1) on peripheral blood neutrophils in acute pancreatitis. , 2014, Advances in medical sciences.
[13] A. Saluja,et al. New insights into the pathogenesis of pancreatitis , 2013, Current opinion in gastroenterology.
[14] S. Radakovic,et al. Tumor Necrosis Factor-&agr; Levels Early in Severe Acute Pancreatitis: Is There Predictive Value Regarding Severity and Outcome? , 2013, Journal of clinical gastroenterology.
[15] A. Yoshimura,et al. IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality. , 2013, The Journal of clinical investigation.
[16] G. Poropat,et al. The Role of IL-6, 8, and 10, sTNFr, CRP, and Pancreatic Elastase in the Prediction of Systemic Complications in Patients with Acute Pancreatitis , 2013, Gastroenterology research and practice.
[17] Ping Chen,et al. siRNA-based targeting of fractalkine overexpression suppresses inflammation development in a severe acute pancreatitis rat model. , 2012, International journal of molecular medicine.
[18] A. Işık,et al. Effects of infliximab on bacterial translocation in experimental acute necrotizing pancreatitis , 2012, The Indian journal of medical research.
[19] M. Büchler,et al. Multidrug strategies are effective in the treatment of severe experimental pancreatitis. , 2012, Surgery.
[20] J. Izbicki,et al. The role of vascular adhesion molecules PECAM-1 (CD 31), VCAM-1 (CD 106), E-selectin (CD62E) and P-selectin (CD62P) in severe porcine pancreatitis. , 2009, Histology and histopathology.
[21] A. Ougolkov,et al. Inhibition of glycogen synthase kinase-3. , 2008, Methods in molecular biology.
[22] S. Chooklin,et al. Implication of interleukin 18 and intercellular adhesion molecule (ICAM)-1 in acute pancreatitis. , 2008, Hepato-Gastroenterology.
[23] M. Corsi,et al. Serum Adhesion Molecules in Acute Pancreatitis: Time Course and Early Assessment of Disease Severity , 2008, Pancreas.
[24] P. Hegyi,et al. The role of NF-κB activation in the pathogenesis of acute pancreatitis , 2007, Gut.
[25] M. den Dulk,et al. Combined donor leucocyte administration and immunosuppressive drug treatment for survival of rat heart allografts. , 2004, Transplant immunology.
[26] B. Engelhardt,et al. Glycosylation processing inhibition by castanospermine prevents experimental autoimmune encephalomyelitis by interference with IL-2 receptor signal transduction , 2002, Journal of Neuroimmunology.
[27] S. Pandol,et al. Localized pancreatic NF-kappaB activation and inflammatory response in taurocholate-induced pancreatitis. , 2001, American journal of physiology. Gastrointestinal and liver physiology.
[28] W. Cowden,et al. Castanospermine, an oligosaccharide-processing inhibitor, reduces membrane expression of adhesion molecules and prolongs heart allograft survival in rats. , 1996, Transplantation proceedings.
[29] W. Cowden,et al. Castanospermine, an oligosaccharide processing inhibitor, reduces membrane expression of adhesion molecules and prolongs heart allograft survival in rats. , 1997, Transplant Immunology.
[30] W. Cowden,et al. Inhibition of adjuvant arthritis in the rat by phosphosugars and the α‐glucosidase inhibitor castanospermine , 1992, Immunology and cell biology.
[31] D W Rattner,et al. A better model of acute pancreatitis for evaluating therapy. , 1992, Annals of surgery.
[32] A. Elbein. Glycosidase inhibitors: inhibitors of N‐linked oligosaccharide processing , 1991, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[33] W. Cowden,et al. Inhibition of experimental allergic encephalomyelitis by the α-glucosidase inhibitor castanospermine , 1989, Journal of the Neurological Sciences.
[34] J. Clardy,et al. Castanospermine, A 1,6,7,8-tetrahydroxyoctahydroindolizine alkaloid, from seeds of Castanospermum australe , 1981 .