Early postnatal effects of prenatal exposure to glucocorticoids on testosterone metabolism and biogenic monoamines in discrete neuroendocrine regions of the rat brain.
暂无分享,去创建一个
[1] N. N. Dygalo,et al. Effect of corticosteroid imbalance on the catecholamine system of the fetal rat brain , 1991, Bulletin of Experimental Biology and Medicine.
[2] S. Matthews. Antenatal glucocorticoids and the developing brain: mechanisms of action. , 2001, Seminars in neonatology : SN.
[3] S. Matthews,et al. Maternal glucocorticoid treatment programs HPA regulation in adult offspring: sex-specific effects. , 2001, American journal of physiology. Endocrinology and metabolism.
[4] S. Matthews. Antenatal Glucocorticoids and Programming of the Developing CNS , 2000, Pediatric Research.
[5] A. Reznikov,et al. Prenatal stress and glucocorticoid effects on the developing gender-related brainProceedings of Xth International Congress on Hormonal Steroids, Quebec, Canada, 17–21 June 1998. , 1999, The Journal of Steroid Biochemistry and Molecular Biology.
[6] M. Joëls,et al. Brain corticosteroid receptor balance in health and disease. , 1998, Endocrine reviews.
[7] M. Takigawa,et al. Prenatal dexamethasone exposure alters brain monoamine metabolism and adrenocortical response in rat offspring. , 1997, American journal of physiology. Regulatory, integrative and comparative physiology.
[8] J. Seckl,et al. Dexamethasone in the last week of pregnancy attenuates hippocampal glucocorticoid receptor gene expression and elevates blood pressure in the adult offspring in the rat. , 1996, Neuroendocrinology.
[9] A. Reznikov,et al. Early postnatal changes in sexual dimorphism of catecholamine and indoleamine content in the brain of prenatally stressed rats , 1996, Neuroscience.
[10] R. Gorski,et al. A revised critical period for the sexual differentiation of the sexually dimorphic nucleus of the preoptic area in the rat. , 1995, Neuroendocrinology.
[11] D M Sheehan,et al. Prenatal dexamethasone or stress but not ACTH or corticosterone alter sexual behavior in male rats. , 1995, Neurotoxicology and teratology.
[12] A. Reznikov,et al. Catecholamines in steroid-dependent brain development , 1995, The Journal of Steroid Biochemistry and Molecular Biology.
[13] T. Slotkin,et al. Glucocorticoids and the development of neuronal function: effects of prenatal dexamethasone exposure on central noradrenergic activity. , 1992, Biology of the neonate.
[14] N. N. Dygalo,et al. [The catecholamine system in rat fetal brain with a disrupted corticosteroid balance]. , 1991, Biulleten' eksperimental'noi biologii i meditsiny.
[15] M. J. Baum,et al. Sex difference in whole-body androgen content in rats on fetal days 18 and 19 without evidence that androgen passes from males to females. , 1991, Biology of reproduction.
[16] G. Dörner. Hormone-dependent brain development and neuroendocrine prophylaxis. , 2009, Experimental and clinical endocrinology.
[17] F. Naftolin,et al. Estrogen formation in the developing rat brain: sex differences in aromatase activity during early post-natal life , 1985, Psychoneuroendocrinology.
[18] F. Naftolin,et al. Sexual differentiation of the central nervous system. , 1981, Science.
[19] G. Dörner,et al. Homosexual behaviour in prenatally stressed male rats after castration and oestrogen treatment in adulthood. , 1980, Endokrinologie.
[20] J. Weisz,et al. Plasma testosterone and progesterone titers of pregnant rats, their male and female fetuses, and neonatal offspring. , 1980, Endocrinology.
[21] A. Krstulović,et al. Rapid analysis of tryptophan metabolites using reversed-phase high-performance liquid chromatography with fluorometric detection. , 1979, Journal of chromatography.
[22] D. Jacobowitz,et al. Method for the rapid determination of norepinephrine, dopamine, and serotonin in the same brain region , 1978, Pharmacology Biochemistry and Behavior.
[23] G. Dörner. Hormones and brain differentiation , 1976 .
[24] I. Ward,et al. Prenatal Stress Feminizes and Demasculinizes the Behavior of Males , 1972, Science.
[25] B. Brodie,et al. Application of steady state kinetics to the estimation of synthesis rate and turnover time of tissue catecholamines. , 1966, The Journal of pharmacology and experimental therapeutics.