Time of day, outcome, and response to thrombolytic therapy: the National Institute of Neurological Disorders and Stroke Recombinant Tissue Plasminogen Activator Stroke Trial experience.

[1]  À. Rovira,et al.  Progression of Symptomatic Intracranial Large Artery Atherosclerosis Is Associated With a Proinflammatory State and Impaired Fibrinolysis , 2008, Stroke.

[2]  Zsolt Garami,et al.  Site of Arterial Occlusion Identified by Transcranial Doppler Predicts the Response to Intravenous Thrombolysis for Stroke , 2007, Stroke.

[3]  C. Molina,et al.  Tandem Internal Carotid Artery/Middle Cerebral Artery Occlusion: An Independent Predictor of Poor Outcome After Systemic Thrombolysis , 2006, Stroke.

[4]  C. Molina,et al.  Hemodynamic changes in the basilar artery following stenting , 2004, Neurology.

[5]  E. Granieri,et al.  Circadian variability in hemorrhagic stroke. , 2002, JAMA.

[6]  W M O'Fallon,et al.  Circadian and seasonal occurrence of subarachnoid and intracerebral hemorrhage , 2001, Neurology.

[7]  J. Grotta,et al.  Early stroke treatment associated with better outcome: the ninds rt-pa stroke study , 2000, Neurology.

[8]  Ninds,et al.  Effect of Intravenous Recombinant Tissue Plasminogen Activator on Ischemic Stroke Lesion Size Measured by Computed Tomography , 2000, Stroke.

[9]  J. Broderick,et al.  Predicting prognosis after stroke , 2000, Neurology.

[10]  R. Sothern,et al.  Relation between circadian patterns in levels of circulating lipoprotein(a), fibrinogen, platelets, and related lipid variables in men. , 2000, American heart journal.

[11]  E. Abinader,et al.  Circadian fluctuations in the efficacy of thrombolysis with streptokinase , 1999, Postgraduate medical journal.

[12]  Circadian variation in ischemic stroke subtypes. , 1999, Stroke.

[13]  N L Geller,et al.  Use of a global test for multiple outcomes in stroke trials with application to the National Institute of Neurological Disorders and Stroke t-PA Stroke Trial. , 1996, Stroke.

[14]  S. Diamond,et al.  Computer simulation of systemic circulation and clot lysis dynamics during thrombolytic therapy that accounts for inner clot transport and reaction. , 1996, Circulation.

[15]  Y. Hirota,et al.  Circadian variations of onset of acute myocardial infarction and efficacy of thrombolytic therapy. , 1996, Journal of the American College of Cardiology.

[16]  Joseph P. Broderick,et al.  Tissue plasminogen activator for acute ischemic stroke. The National Institute of Neurological Disorders and Stroke rt-PA Stroke Study Group. , 1995 .

[17]  P. Kurnik.  Circadian variation in the efficacy of tissue-type plasminogen activator. , 1995, Circulation.

[18]  R. Manfredini,et al.  Acute intracerebral haemorrhage: circadian and circannual patterns of onset , 1994, Acta neurologica Scandinavica.

[19]  R. Manfredini,et al.  Chronobiological aspects of acute cerebrovascular diseases , 1993, Acta neurologica Scandinavica.

[20]  M. Fujita,et al.  Circadian variation in the success rate of intracoronary thrombolysis for acute myocardial infarction. , 1993, The American journal of cardiology.

[21]  Daniel B Hier,et al.  Circadian Rhythmicity of Stroke Onset: Intracerebral and Subarachnoid Hemorrhage , 1992, Stroke.

[22]  J. Marler,et al.  Circadian variation in onset of acute ischemic stroke. , 1990, Archives of neurology.

[23]  F. Violi,et al.  Circadian variation in the frequency of ischemic stroke. , 1990, Stroke.

[24]  Daniel B Hier,et al.  Morning increase in onset of ischemic stroke. , 1989, Stroke.

[25]  W. Chandler,et al.  Diurnal variation of tissue-type plasminogen activator and its rapid inhibitor (PAI-1). , 1989, Circulation.

[26]  E. Kruithof,et al.  Diurnal Variation of the Fibrinolytic System , 1988, Thrombosis and Haemostasis.

[27]  H. Schouten,et al.  Interobserver agreement for the assessment of handicap in stroke patients. , 1988, Stroke.

[28]  J. Alpert,et al.  Circadian variation in thrombolytic response to recombinant tissue-type plasminogen activator in acute myocardial infarction , 1988 .

[29]  S. Willich,et al.  Concurrent morning increase in platelet aggregability and the risk of myocardial infarction and sudden cardiac death. , 1987, The New England journal of medicine.

[30]  J. Gris,et al.  Circadian changes in anticoagulant effect of heparin infused at a constant rate. , 1985, British medical journal.

[31]  R. Mangiafico,et al.  Daily modifications of plasma fibrinogen platelets aggregation, Howell's time, PTT, TT, and antithrombin II in normal subjects and in patients with vascular disease. , 1982, Chronobiologia.

[32]  J. Marshall.  Diurnal Variation in Occurrence of Strokes , 1977, Stroke.

[33]  B. Jennett,et al.  ASSESSMENT OF OUTCOME AFTER SEVERE BRAIN DAMAGE A Practical Scale , 1975, The Lancet.

[34]  T. Deegan,et al.  Circadian variations of plasma catecholamine, cortisol and immunoreactive insulin concentrations in supine subjects. , 1974, Clinica chimica acta; international journal of clinical chemistry.

[35]  A. Ehrly,et al.  Circadian rhythm of human blood viscosity. , 1973, Biorheology.

[36]  S. Epstein,et al.  Impairment of the Diurnal Fibrinolytic Response in Man: EFFECTS OF AGING, TYPE IV HYPERLIPOPROTEINEMIA, AND CORONARY ARTERY DISEASE , 1973, Circulation research.

[37]  T F Gallagher,et al.  Twenty-four hour pattern of the episodic secretion of cortisol in normal subjects. , 1971, The Journal of clinical endocrinology and metabolism.

[38]  Foss Mv.  MANAGEMENT OF VIRUS HEPATITIS. , 1964 .