Allogeneic hematopoietic stem cell transplantation recipients have defects of both switched and igm memory B cells.

[1]  A. Vanoli,et al.  Splenic function and IgM‐memory B cells in Crohn's disease patients treated with infliximab , 2008, Inflammatory bowel diseases.

[2]  Steffen Jung,et al.  Perivascular clusters of dendritic cells provide critical survival signals to B cells in bone marrow niches , 2008, Nature Immunology.

[3]  C. Mackay,et al.  T follicular helper (TFH) cells in normal and dysregulated immune responses. , 2008, Annual review of immunology.

[4]  N. Haynes.  Follicular associated T cells and their B-cell helper qualities. , 2008, Tissue antigens.

[5]  M. Kouba,et al.  Elevated numbers of immature/transitional CD21- B lymphocytes and deficiency of memory CD27+ B cells identify patients with active chronic graft-versus-host disease. , 2008, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

[6]  Eren,et al.  The EUROclass trial: defining subgroups in common variable immunodeficiency. , 2008, Blood.

[7]  M. Zachariah,et al.  Follicular shuttling of marginal zone B cells facilitates antigen transport , 2008, Nature Immunology.

[8]  Y. Li,et al.  Cytokine-Mediated Regulation of Human B Cell Differentiation into Ig-Secreting Cells: Predominant Role of IL-21 Produced by CXCR5+ T Follicular Helper Cells1 , 2007, The Journal of Immunology.

[9]  E. Shpall,et al.  Delayed immune reconstitution after cord blood transplantation is characterized by impaired thymopoiesis and late memory T-cell skewing. , 2007, Blood.

[10]  L. D'Orsogna,et al.  Circulating memory B-cell subpopulations are affected differently by HIV infection and antiretroviral therapy , 2007, AIDS.

[11]  S. Tangye,et al.  Human IgM+CD27+ B Cells: Memory B Cells or “Memory” B Cells?1 , 2007, The Journal of Immunology.

[12]  Adeeba Kamarulzaman,et al.  AIDS Res Hum Retroviruses , 2006 .

[13]  G. Murphy,et al.  Host-Derived Langerhans Cells Persist after MHC-Matched Allografting Independent of Donor T Cells and Critically Influence the Alloresponses Mediated by Donor Lymphocyte Infusions1 , 2006, The Journal of Immunology.

[14]  F. Sallusto,et al.  Understanding and making use of human memory B cells , 2006, Immunological reviews.

[15]  S. Pittaluga,et al.  Selective generation of functional somatically mutated IgM+CD27+, but not Ig isotype-switched, memory B cells in X-linked lymphoproliferative disease. , 2006, The Journal of clinical investigation.

[16]  S. Tangye,et al.  Expansion of Functionally Immature Transitional B Cells Is Associated with Human-Immunodeficient States Characterized by Impaired Humoral Immunity1 , 2006, The Journal of Immunology.

[17]  A. Tosti,et al.  Transferring functional immune responses to pathogens after haploidentical hematopoietic transplantation. , 2005, Blood.

[18]  R. Jacob,et al.  CD27+ B cells in human lymphatic organs: re‐evaluating the splenic marginal zone , 2005, Immunology.

[19]  C. Contag,et al.  Ex vivo expanded dendritic cells home to T-cell zones of lymphoid organs and survive in vivo after allogeneic bone marrow transplantation. , 2005, The American journal of pathology.

[20]  K. Agematsu,et al.  Regulation of Aged Humoral Immune Defense against Pneumococcal Bacteria by IgM Memory B Cell , 2005, The Journal of Immunology.

[21]  F. Locatelli,et al.  B lymphocyte reconstitution after hematopoietic stem cell transplantation: functional immaturity and slow recovery of memory CD27+ B cells. , 2005, Experimental hematology.

[22]  M. Lipsitch,et al.  CD4+ T cells mediate antibody-independent acquired immunity to pneumococcal colonization. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[23]  F. Locatelli,et al.  Antibody response to pneumococcal vaccine in children receiving bone marrow transplantation , 1995, Journal of Clinical Immunology.

[24]  L. Staudt,et al.  Human blood IgM "memory" B cells are circulating splenic marginal zone B cells harboring a prediversified immunoglobulin repertoire. , 2004, Blood.

[25]  U. Duffner,et al.  Host Dendritic Cells Alone Are Sufficient to Initiate Acute Graft-versus-Host Disease1 , 2004, The Journal of Immunology.

[26]  S. Kashiwagi,et al.  Increased frequency of CD27- (naive) B cells and their phenotypic alteration in HIV type 1-infected patients. , 2004, AIDS research and human retroviruses.

[27]  D. Foster,et al.  Cutting Edge: IL-21 Is a Switch Factor for the Production of IgG1 and IgG3 by Human B Cells1 , 2004, The Journal of Immunology.

[28]  L. Hougs,et al.  Human Memory B Cells Transferred by Allogenic Bone Marrow Transplantation Contribute Significantly to the Antibody Repertoire of the Recipient 1 , 2004, The Journal of Immunology.

[29]  R. Carsetti,et al.  Peripheral development of B cells in mouse and man , 2004, Immunological reviews.

[30]  W. Saliba,et al.  Pneumococcal Sepsis due to Functional Hyposplenism in a Bone Marrow Transplant Patient , 2004, European Journal of Clinical Microbiology and Infectious Diseases.

[31]  Thomas Boehm,et al.  Human Immunoglobulin M Memory B Cells Controlling Streptococcus pneumoniae Infections Are Generated in the Spleen , 2003, The Journal of experimental medicine.

[32]  B. Blazar,et al.  Abnormal T cell-dependent B-cell responses in SCID mice receiving allogeneic bone marrow in utero. Severe combined immune deficiency. , 2002, Blood.

[33]  R. Gress,et al.  Reconstitution of thymic function after stem cell transplantation in humans , 2002, Current opinion in hematology.

[34]  P. Ljungman,et al.  Early and late invasive pneumococcal infection following stem cell transplantation: a European Bone Marrow Transplantation survey , 2002, British journal of haematology.

[35]  R. Handgretinger,et al.  Immune recovery and immunotherapy after stem cell transplantation in children , 2001, Bone Marrow Transplantation.

[36]  C. Dykewicz.  Summary of the Guidelines for Preventing Opportunistic Infections among Hematopoietic Stem Cell Transplant Recipients. , 2001, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

[37]  S. Kvaløy,et al.  Humoral immunity to viral and bacterial antigens in lymphoma patients 4–10 years after high-dose therapy with ABMT. Serological responses to revaccinations according to EBMT guidelines , 2001, Bone Marrow Transplantation.

[38]  P. Ruutu,et al.  Opsonophagocytic activity against Streptococcus pneumoniae type 19F in allogeneic BMT recipients before and after vaccination with pneumococcal polysaccharide vaccine , 2001, Bone Marrow Transplantation.

[39]  S. Singhal,et al.  Chronic graft versus host disease is associated with long-term risk for pneumococcal infections in recipients of bone marrow transplants. , 2000, Blood.

[40]  P. Ljungman,et al.  Serum immunoglobulin levels in relation to levels of specific antibodies in allogeneic and autologous bone marrow transplant recipients. , 2000, Transplantation.

[41]  M. Picardi,et al.  Spleen sizing by ultrasound scan and risk of pneumococcal infection in patients with chronic GVHD: preliminary observations , 1999, Bone Marrow Transplantation.

[42]  J. V. D. van de Winkel,et al.  Crucial role of FcgammaRIIa (CD32) in assessment of functional anti-Streptococcus pneumoniae antibody activity in human sera. , 1999, The Journal of infectious diseases.

[43]  R. V. van Lier,et al.  Reconstitution of the T-cell compartment after bone marrow transplantation: restoration of the repertoire by thymic emigrants. , 1998, Blood.

[44]  K. Rajewsky,et al.  Human Immunoglobulin (Ig)M+IgD+ Peripheral Blood B Cells Expressing the CD27 Cell Surface Antigen Carry Somatically Mutated Variable Region Genes: CD27 as a General Marker for Somatically Mutated (Memory) B Cells , 1998, The Journal of experimental medicine.

[45]  R. Storb,et al.  Reconstitution of membrane IgD- (mIgD-) B cells after marrow transplantation lags behind the reconstitution of mIgD+ B cells. , 1997, Blood.

[46]  A. Glas,et al.  Immunoglobulin heavy chain variable region gene usage in bone marrow transplant recipients: lack of somatic mutation indicates a maturational arrest. , 1996, Blood.

[47]  M. French,et al.  Severity of infections in IgA deficiency: correlation with decreased serum antibodies to pneumococcal polysaccharides and decreased serum IgG2 and/or IgG4 , 1995, Clinical and experimental immunology.

[48]  N. Russell,et al.  Functional hyposplenism following allogeneic bone marrow transplantation. , 1995, Journal of clinical pathology.

[49]  M. Ohbayashi,et al.  White pulp reconstitution after human bone marrow transplantation. , 1993, The American journal of pathology.

[50]  G. Sale,et al.  Abnormal CD4:CD8 ratios and delayed germinal center reconstitution in lymph nodes of human graft recipients with graft-versus-host disease (GVHD): an immunohistological study. , 1992, Experimental hematology.

[51]  J. Storek,et al.  Reconstitution of B cell immunity following bone marrow transplantation. , 1992, Bone marrow transplantation.

[52]  R. Geha,et al.  Lymphokine profile in bone marrow transplant recipients. , 1991, Blood.

[53]  A. Newland,et al.  IgG subclass levels and immune reconstitution after T cell‐depleted allogeneic bone marrow transplantation , 1990, Clinical and experimental immunology.

[54]  M. Martelli,et al.  Acquisition of Ig isotype diversity after bone marrow transplantation in adults. A recapitulation of normal B cell ontogeny. , 1988, Journal of immunology.

[55]  H. Ochs,et al.  Recovery of antibody production in human allogeneic marrow graft recipients: influence of time posttransplantation, the presence or absence of chronic graft-versus-host disease, and antithymocyte globulin treatment. , 1981, Blood.

[56]  S. Feig,et al.  Pneumococcal infections after human bone-marrow transplantation. , 1979, Annals of internal medicine.