The role of neutrophils in myocardial ischemia-reperfusion injury.
暂无分享,去创建一个
J. Vinten-johansen | Zhi-Qing Zhao | J E Jordan | Z Q Zhao | J Vinten-Johansen | J. Jordan | Z. Zhao
[1] F. Sellke,et al. Anti-C5a monoclonal antibody reduces cardiopulmonary bypass and cardioplegia-induced coronary endothelial dysfunction. , 1998, The Journal of thoracic and cardiovascular surgery.
[2] A. M. Lefer,et al. Synergism between platelets and neutrophils in provoking cardiac dysfunction after ischemia and reperfusion: role of selectins. , 1998, Circulation.
[3] E R McVeigh,et al. Magnitude and time course of microvascular obstruction and tissue injury after acute myocardial infarction. , 1998, Circulation.
[4] J. Fortenberry,et al. Modifying risk for extracorporeal circulation: trial of four antiinflammatory strategies. , 1998, The Annals of thoracic surgery.
[5] A. M. Lefer,et al. Beneficial effects of N,N,N-trimethylsphingosine following ischemia and reperfusion in the isolated perfused rat heart. , 1998, Cardiovascular research.
[6] R. Wallace,et al. Heparin and nonanticoagulant heparin preserve regional myocardial contractility after ischemia-reperfusion injury: role of nitric oxide. , 1998, The Journal of thoracic and cardiovascular surgery.
[7] J. T. Cope,et al. Sodium nitroprusside exacerbates myocardial ischemia-reperfusion injury. , 1997, The Annals of thoracic surgery.
[8] R. Cummings,et al. Role of PSGL-1 binding to selectins in leukocyte recruitment. , 1997, The Journal of clinical investigation.
[9] M. Hori,et al. Amelioration of ischemia- and reperfusion-induced myocardial injury by 17beta-estradiol: role of nitric oxide and calcium-activated potassium channels. , 1997, Circulation.
[10] Hiroki Sato,et al. Monoclonal antibody to ICAM‐1 preserves postischemic blood flow and reduces infarct size after ischemia‐reperfusion in rabbit , 1997, Journal of leukocyte biology.
[11] R. Cummings,et al. Perspectives series: cell adhesion in vascular biology. Role of PSGL-1 binding to selectins in leukocyte recruitment. , 1997, The Journal of clinical investigation.
[12] A. M. Lefer,et al. Regulation of P-selectin expression in human endothelial cells by nitric oxide. , 1997, The American journal of physiology.
[13] K. Hirata,et al. Nitric oxide inhibits neutrophil adhesion to cytokine-activated cardiac myocytes. , 1997, The American journal of physiology.
[14] G. Folkerts,et al. Peroxynitrite: a two-faced metabolite of nitric oxide. , 1997, Life sciences.
[15] J. Hammon,et al. Basal nitric oxide expresses endogenous cardioprotection during reperfusion by inhibition of neutrophil-mediated damage after surgical revascularization. , 1997, The Journal of thoracic and cardiovascular surgery.
[16] H. Sato,et al. Adenosine A2 receptor activation attenuates reperfusion injury by inhibiting neutrophil accumulation, superoxide generation and coronary endothelial adherence. , 1997, The Journal of pharmacology and experimental therapeutics.
[17] E. Verrier,et al. Endothelial cell injury in cardiovascular surgery: ischemia-reperfusion. , 1996, The Annals of thoracic surgery.
[18] A. M. Lefer,et al. S-nitrosylated tissue-type plasminogen activator protects against myocardial ischemia/reperfusion injury in cats: role of the endothelium. , 1996, The Journal of pharmacology and experimental therapeutics.
[19] F E Schmidt,et al. Leukocyte depletion of blood cardioplegia attenuates reperfusion injury. , 1996, The Annals of thoracic surgery.
[20] J S Beckman,et al. Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly. , 1996, The American journal of physiology.
[21] H. Sato,et al. Adenosine A2-receptor activation inhibits neutrophil-mediated injury to coronary endothelium. , 1996, The American journal of physiology.
[22] A. M. Lefer,et al. The role of nitric oxide and cell adhesion molecules on the microcirculation in ischaemia-reperfusion. , 1996, Cardiovascular research.
[23] B. Lucchesi,et al. Selective inhibition of the alternative complement pathway by sCR1[desLHR-A] protects the rabbit isolated heart from human complement-mediated damage. , 1996, Immunopharmacology.
[24] F. Lallemand,et al. Preconditioning prevents chronic reperfusion-induced coronary endothelial dysfunction in rats. , 1996, The American journal of physiology.
[25] A. M. Lefer,et al. Direct measurement of nitric oxide release from vascular endothelial cells. , 1996, Journal of applied physiology.
[26] H. Oku,et al. [Simultaneous surgical revascularization for myocardial and peripheral vascular ischemia]. , 1996, Kyobu geka. The Japanese journal of thoracic surgery.
[27] L. Horwitz,et al. An oligosaccharide sialyl-Lewis(x) analogue does not reduce myocardial infarct size after ischemia and reperfusion in dogs. , 1996, Circulation.
[28] G. Firestein,et al. Carbohydrate- and CD18-dependent neutrophil adhesion to cardiac myocytes: effects of adenosine. , 1996, Cardiovascular research.
[29] M. Thiel,et al. Effect of adenosine on the expression of β2 integrins and L‐selectin of human polymorphonuclear leukocytes in vitro , 1996, Journal of leukocyte biology.
[30] M. Arai,et al. An anti-CD18 antibody limits infarct size and preserves left ventricular function in dogs with ischemia and 48-hour reperfusion. , 1996, Journal of the American College of Cardiology.
[31] W. Buurman,et al. Adenosine inhibits cytokine release and expression of adhesion molecules by activated human endothelial cells. , 1996, The American journal of physiology.
[32] J. Gott,et al. Depletion of Blood Cardioplegia Attenuates Reperfusion Injury , 1996 .
[33] A. Buda,et al. Effects of a monoclonal antibody directed against P-selectin after myocardial ischemia and reperfusion. , 1996, The American journal of physiology.
[34] A. Buda,et al. Nitric oxide attenuates neutrophil-mediated myocardial contractile dysfunction after ischemia and reperfusion. , 1996, Circulation research.
[35] H. Sato,et al. L-Arginine inhibits neutrophil adherence and coronary artery dysfunction. , 1996, Cardiovascular research.
[36] A. M. Lefer,et al. Inhibition of nitric oxide biosynthesis promotes P-selectin expression in platelets. Role of protein kinase C. , 1995, Arteriosclerosis, thrombosis, and vascular biology.
[37] W. DeCampli,et al. Simultaneous manipulation of the nitric oxide and prostanoid pathways reduces myocardial reperfusion injury. , 1995, The Journal of thoracic and cardiovascular surgery.
[38] R. Schulz,et al. Inhibition of nitric oxide synthesis protects the isolated working rabbit heart from ischaemia-reperfusion injury. , 1995, Cardiovascular research.
[39] A. M. Lefer,et al. Attenuation of myocardial ischemia-reperfusion injury with nitric oxide replacement therapy. , 1995, The Annals of thoracic surgery.
[40] A. Buda,et al. Intracoronary nitric oxide improves postischemic coronary blood flow and myocardial contractile function. , 1995, The American journal of physiology.
[41] J. Hammon,et al. Supplemental L-arginine during cardioplegic arrest and reperfusion avoids regional postischemic injury. , 1995, The Journal of thoracic and cardiovascular surgery.
[42] K. Stöcker,et al. Phorbol ester-stimulated adherence of neutrophils to endothelial cells is reduced by adenosine A2 receptor agonists. , 1995, Journal of immunology.
[43] P. Libby,et al. Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines. , 1995, The Journal of clinical investigation.
[44] T. Williams,et al. Neutrophil chemoattractants generated in two phases during reperfusion of ischemic myocardium in the rabbit. Evidence for a role for C5a and interleukin-8. , 1995, The Journal of clinical investigation.
[45] P. Kubes,et al. Therapeutic potential of inhibiting leukocyte rolling in ischemia/reperfusion. , 1995, The Journal of clinical investigation.
[46] J. D. Chambers,et al. C5a- and tumor necrosis factor-alpha-induced leukocytosis occurs independently of beta 2 integrins and L-selectin: differential effects on neutrophil adhesion molecule expression in vivo. , 1995, Blood.
[47] D. Van Wylen,et al. Adenosine deaminase inhibition augments interstitial adenosine but does not attenuate myocardial infarction. , 1995, Cardiovascular research.
[48] B. Lucchesi,et al. Cardioprotective effects of heparin or N-acetylheparin in an in vivo model of myocardial ischaemic and reperfusion injury. , 1995, Cardiovascular research.
[49] D. Yellon,et al. Inhibition of nitric oxide synthesis reduces infarct size by an adenosine-dependent mechanism. , 1995, Circulation.
[50] R. Cummings,et al. P-selectin glycoprotein ligand-1 mediates rolling of human neutrophils on P-selectin , 1995, The Journal of cell biology.
[51] D. Sheridan,et al. Neutrophil interactions with endothelium and platelets: possible role in the development of cardiovascular injury. , 1995, European heart journal.
[52] A. M. Lefer,et al. Cardioprotective effects of a C1 esterase inhibitor in myocardial ischemia and reperfusion. , 1995, Circulation.
[53] J. Zweier,et al. Direct Measurement of Nitric Oxide Generation in the Ischemic Heart Using Electron Paramagnetic Resonance Spectroscopy (*) , 1995, The Journal of Biological Chemistry.
[54] G. Schmid-Schönbein,et al. Modification of leukocyte adhesion in spontaneously hypertensive rats by adrenal corticosteroids , 1995, Journal of leukocyte biology.
[55] A. M. Lefer,et al. Time course of coronary vascular endothelial adhesion molecule expression during reperfusion of the ischemic feline myocardium , 1995, Journal of leukocyte biology.
[56] T. Möröy,et al. Complement Activation and Inhibition , 1995 .
[57] M. Erion,et al. Inhibition of neutrophil adhesion by adenosine and an adenosine kinase inhibitor. The role of selectins. , 1995, Journal of immunology.
[58] S. Rendig,et al. Limitation of reperfusion injury by a monoclonal antibody to C5a during myocardial infarction in pigs. , 1995, The American journal of physiology.
[59] J. Pernow,et al. The protective effect of L-arginine on myocardial injury and endothelial function following ischaemia and reperfusion in the pig. , 1994, European heart journal.
[60] J. Svendsen. The microcirculation of the heart. With special reference to the role of reactive oxygen metabolites in the microvascular reperfusion injury. , 1994, Danish medical bulletin.
[61] A. Buda,et al. A novel sialyl LewisX analog attenuates neutrophil accumulation and myocardial necrosis after ischemia and reperfusion. , 1994, Circulation.
[62] A. M. Lefer,et al. Inhibition of endothelial-derived nitric oxide promotes P-selectin expression and actions in the rat microcirculation. , 1994, Gastroenterology.
[63] A. M. Lefer,et al. Humanized monoclonal antibody DREG-200 directed against I-selectin protects in feline myocardial reperfusion injury. , 1994, The Journal of pharmacology and experimental therapeutics.
[64] B. Lucchesi,et al. Effects of heparin and N-acetyl heparin on ischemia/reperfusion-induced alterations in myocardial function in the rabbit isolated heart. , 1994, Circulation research.
[65] P. Kubes,et al. NO donors prevent integrin-induced leukocyte adhesion but not P-selectin-dependent rolling in postischemic venules. , 1994, The American journal of physiology.
[66] M. Entman,et al. Postreperfusion inflammation: a model for reaction to injury in cardiovascular disease. , 1994, Cardiovascular research.
[67] P. Ward,et al. C5a-induced expression of P-selectin in endothelial cells. , 1994, The Journal of clinical investigation.
[68] A. M. Lefer,et al. Polymorphonuclear leukocyte-induced vasocontraction and endothelial dysfunction. Role of selectins. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[69] A. M. Lefer,et al. Role of P-selectin in microvascular leukocyte-endothelial interaction in splanchnic ischemia-reperfusion. , 1994, The American journal of physiology.
[70] J. Hammon,et al. Coronary artery endothelial dysfunction after global ischemia, blood cardioplegia, and reperfusion. , 1994, The Annals of thoracic surgery.
[71] J. Perrard,et al. Molecular evidence for induction of intracellular adhesion molecule-1 in the viable border zone associated with ischemia-reperfusion injury of the dog heart. , 1994, Circulation.
[72] J. Gorcsan,et al. A Potential Role for Nitric Oxide in Myocardial Stunning , 1994, Journal of cardiac surgery.
[73] J. Mehta,et al. Adenosine protects against attenuation of flow reserve and myocardial function after coronary occlusion and reperfusion. , 1994, American heart journal.
[74] A. M. Lefer,et al. Quantification of neutrophil migration following myocardial ischemia and reperfusion in cats and dogs , 1994, Journal of leukocyte biology.
[75] D. G. L. Wylen,et al. Effect of ischemic preconditioning on interstitial purine metabolite and lactate accumulation during myocardial ischemia. , 1994 .
[76] U. Ikeda,et al. Neutrophil Adherence to Rat Cardiac Myocyte by Proinflammatory Cytokines , 1994, Journal of cardiovascular pharmacology.
[77] C. Smith,et al. P-selectin and ICAM-1-dependent adherence reactions: role in the genesis of postischemic no-reflow. , 1994, The American journal of physiology.
[78] J. Beckman,et al. Peroxynitrite, a product of superoxide and nitric oxide, produces coronary vasorelaxation in dogs. , 1994, The Journal of pharmacology and experimental therapeutics.
[79] J. Schofer,et al. Early accumulation of the terminal complement-complex in the ischaemic myocardium after reperfusion. , 1994, European heart journal.
[80] A. M. Lefer,et al. Sialyl Lewisx-containing oligosaccharide attenuates myocardial reperfusion injury in cats. , 1994, The Journal of clinical investigation.
[81] P. Kubes,et al. Molecular mechanisms involved in superoxide-induced leukocyte-endothelial cell interactions in vivo. , 1994, The American journal of physiology.
[82] W. Baumgartner,et al. Complement and neutrophil activation during cardiopulmonary bypass: a study in the complement-deficient dog. , 1994, The Annals of thoracic surgery.
[83] D. Lefer,et al. Therapeutic role of nitric oxide donors in the treatment of cardiovascular disease , 1994 .
[84] B. Lucchesi,et al. Complement activation and inhibition in myocardial ischemia and reperfusion injury. , 1994, Annual review of pharmacology and toxicology.
[85] S. Thom,et al. Involvement of platelet-endothelial cell adhesion molecule-1 in neutrophil recruitment in vivo. , 1993, Science.
[86] M. Weisfeldt,et al. Soluble Complement Receptor Type 1 Inhibits the Complement Pathway and Prevents Contractile Failure in the Postischemic Heart Evidence That Complement Activation Is Required for Neutrophil‐Mediated Reperfusion Injury , 1993, Circulation.
[87] J. Beckman. Nitric oxide and peroxynitrite , 1993 .
[88] T. Gardner,et al. Inhibition of neutrophil adhesion reduces myocardial infarct size. , 1993, The Annals of thoracic surgery.
[89] D. Lefer,et al. Antineutrophil and Myocardial Protecting Actions of a Novel Nitric Oxide Donor After Acute Myocardial Ischemia and Reperfusion in Dogs , 1993, Circulation.
[90] J. Zweier,et al. Cardioprotective Actions of a Monoclonal Antibody Against CD‐18 in Myocardial Ischemia‐Reperfusion Injury , 1993, Circulation.
[91] M. Entman,et al. Regulation of intercellular adhesion molecule-1 (ICAM-1) in ischemic and reperfused canine myocardium. , 1993, The Journal of clinical investigation.
[92] J. Smith,et al. Acting via A2 receptors, adenosine inhibits the upregulation of Mac-1 (Cd11b/CD18) expression on FMLP-stimulated neutrophils. , 1993, American journal of respiratory cell and molecular biology.
[93] J. Vinten-johansen,et al. Receptor‐Mediated Cardioprotective Effects of Endogenous Adenosine Are Exerted Primarily During Reperfusion After Coronary Occlusion in the Rabbit , 1993, Circulation.
[94] G. A. Limb,et al. Early and Late Effects of Leukopenic Reperfusion on the Recovery of Cardiac Contractile Function Studies in the Transplanted and Isolated Blood‐Perfused Rat Heart , 1993, Circulation.
[95] D. Phillips,et al. PECAM-1 is required for transendothelial migration of leukocytes , 1993, The Journal of experimental medicine.
[96] A. M. Lefer,et al. Monoclonal Antibody to L‐Selectin Attenuates Neutrophil Accumulation and Protects Ischemic Reperfused Cat Myocardium , 1993, Circulation.
[97] D. Yellon,et al. Inhibition of nitric oxide limits infarct size in the in situ rabbit heart. , 1993, Biochemical and biophysical research communications.
[98] W. Schlack,et al. Adenosine A2-Receptor Activation at Reperfusion Reduces Infarct Size and Improves Myocardial Wall Function in Dog Heart , 1993, Journal of cardiovascular pharmacology.
[99] A. M. Lefer,et al. In vivo neutralization of P-selectin protects feline heart and endothelium in myocardial ischemia and reperfusion injury. , 1993, The Journal of clinical investigation.
[100] R. Bolli. Role of neutrophils in myocardial stunning after brief ischaemia: the end of a six year old controversy (1987-1993) , 1993, Cardiovascular research.
[101] R. Engler,et al. Severe neutrophil depletion by leucocyte filters or cytotoxic drug does not improve recovery of contractile function in stunned porcine myocardium. , 1993, Cardiovascular research.
[102] J S Beckman,et al. Pathological implications of nitric oxide, superoxide and peroxynitrite formation. , 1993, Biochemical Society transactions.
[103] R. Virmani,et al. Myocardial infarct extension during reperfusion after coronary artery occlusion: pathologic evidence. , 1993, Journal of the American College of Cardiology.
[104] W. Baumgartner,et al. Complement inhibition with soluble complement receptor type 1 in cardiopulmonary bypass. , 1993, The Annals of thoracic surgery.
[105] A. M. Lefer,et al. Diminished basal nitric oxide release after myocardial ischemia and reperfusion promotes neutrophil adherence to coronary endothelium. , 1993, Circulation research.
[106] Xinliang Ma,et al. S-Nitroso-N-acetylpenicillamine is a Potent Inhibitor of Neutrophil-Endothelial Interaction , 1993 .
[107] W. Sluiter,et al. Leukocyte adhesion molecules on the vascular endothelium: their role in the pathogenesis of cardiovascular disease and the mechanisms underlying their expression. , 1993, Journal of cardiovascular pharmacology.
[108] D. Lefer,et al. Endothelial and Myocardial Cell Protection by a Cysteine‐Containing Nitric Oxide Donor After Myocardial Ischemia and Reperfusion , 1993, Journal of cardiovascular pharmacology.
[109] A. M. Lefer,et al. Endothelial dysfunction and neutrophil adherence as critical events in the development of reperfusion injury. , 1993, Agents and actions. Supplements.
[110] J. Harlan,et al. Cytokine-induced adhesion molecule expression on human umbilical vein endothelial cells is not regulated by cyclic adenosine monophosphate accumulation. , 1993, Life sciences.
[111] D. Lefer,et al. Intracoronary L-arginine during reperfusion improves endothelial function and reduces infarct size. , 1992, The American journal of physiology.
[112] J. Svendsen,et al. Effects of exogenous oxygen derived free radicals on myocardial capillary permeability, vascular tone, and incidence of ventricular arrhythmias in the canine heart. , 1992, Cardiovascular research.
[113] W. Dreyer,et al. Kinetics of C5a release in cardiac lymph of dogs experiencing coronary artery ischemia-reperfusion injury. , 1992, Circulation research.
[114] M. Entman,et al. Neutrophil induced oxidative injury of cardiac myocytes. A compartmented system requiring CD11b/CD18-ICAM-1 adherence. , 1992, The Journal of clinical investigation.
[115] A. M. Lefer,et al. Cardiac venous endothelial dysfunction after myocardial ischemia and reperfusion in dogs. , 1992, The American journal of physiology.
[116] A. M. Lefer,et al. Beneficial effects of SPM-5185, a cysteine-containing NO donor in myocardial ischemia-reperfusion. , 1992, The American journal of physiology.
[117] S. Abramson,et al. Nitric oxide, an endothelial cell relaxation factor, inhibits neutrophil superoxide anion production via a direct action on the NADPH oxidase. , 1992, The Journal of clinical investigation.
[118] A. M. Lefer,et al. Coronary Endothelial and Cardiac Protective Effects of a Monoclonal Antibody to Intercellular Adhesion Molecule‐1 in Myocardial Ischemia and Reperfusion , 1992, Circulation.
[119] Simon C Watkins,et al. Negative inotropic effects of cytokines on the heart mediated by nitric oxide. , 1992, Science.
[120] A. M. Lefer,et al. The Role of L‐Arginine in Ameliorating Reperfusion Injury After Myocardial Ischemia in the Cat , 1992, Circulation.
[121] R. Mentzer,et al. Cardiac microdialysis in isolated rat hearts: interstitial purine metabolites during ischemia. , 1992, The American journal of physiology.
[122] S. Houser,et al. Cardiac-derived neutrophil chemotactic factors: detection in coronary sinus effluents of patients undergoing myocardial revascularization. , 1992, The Journal of thoracic and cardiovascular surgery.
[123] S. Abramson,et al. Neutrophil adherence to endothelium is enhanced via adenosine A1 receptors and inhibited via adenosine A2 receptors. , 1992, Journal of immunology.
[124] R. Virmani,et al. The effects of intravenous infusions of selective adenosine A1-receptor and A2-receptor agonists on myocardial reperfusion injury. , 1992, American heart journal.
[125] A. M. Lefer,et al. Cardioprotection and attenuation of endothelial dysfunction by organic nitric oxide donors in myocardial ischemia-reperfusion. , 1992, The Journal of pharmacology and experimental therapeutics.
[126] H. Lutz,et al. Coronary artery endothelial cell and smooth muscle dysfunction after global myocardial ischemia. , 1992, The Annals of thoracic surgery.
[127] L. Horwitz,et al. Role of leukocytes in coronary vascular endothelial injury due to ischemia and reperfusion. , 1991, Circulation research.
[128] R. Engler,et al. Intracoronary C5a induces myocardial ischemia by mechanisms independent of the neutrophil: leukocyte filters desensitize the myocardium to C5a , 1991, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[129] A. M. Lefer,et al. Antibody to CD-18 exerts endothelial and cardiac protective effects in myocardial ischemia and reperfusion. , 1991, The Journal of clinical investigation.
[130] G. Zimmerman,et al. Coexpression of GMP-140 and PAF by endothelium stimulated by histamine or thrombin: a juxtacrine system for adhesion and activation of neutrophils , 1991, The Journal of cell biology.
[131] S. Albelda,et al. Molecular and cellular properties of PECAM-1 (endoCAM/CD31): a novel vascular cell-cell adhesion molecule , 1991, The Journal of cell biology.
[132] A. M. Lefer,et al. Neutrophil-mediated vasoconstriction and endothelial dysfunction in low-flow perfusion-reperfused cat coronary artery. , 1991, Circulation research.
[133] J. Downey,et al. Preconditioning Causes Improved Wall Motion as Well as Smaller Infarcts After Transient Coronary Occlusion in Rabbits , 1991, Circulation.
[134] W. Dreyer,et al. Neutrophil Accumulation in Ischemic Canine Myocardium: Insights Into Time Course, Distribution, and Mechanism of Localization During Early Reperfusion , 1991, Circulation.
[135] P. Kubes,et al. Nitric oxide: an endogenous modulator of leukocyte adhesion. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[136] A. M. Lefer,et al. Role of endothelial dysfunction in the pathogenesis of reperfusion injury after myocardial ischemia , 1991, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[137] H. Nathan. Nifedipine after coronary bypass grafting. , 1991, The Annals of thoracic surgery.
[138] G. Buckberg,et al. Studies of controlled reperfusion after ischemia. XXII. Reperfusate composition: effects of leukocyte depletion of blood and blood cardioplegic reperfusates after acute coronary occlusion. , 1991, The Journal of thoracic and cardiovascular surgery.
[139] A. M. Lefer,et al. Cardioprotective effects of authentic nitric oxide in myocardial ischemia with reperfusion , 1991, Critical care medicine.
[140] J. Parratt,et al. Failure of cyclo-oxygenase inhibition to protect against arrhythmias induced by ischaemia and reperfusion: implications for the role of prostaglandins as endogenous myocardial protective substances. , 1991, Cardiovascular research.
[141] G. Angelini,et al. ASPIRIN AND BLOOD LOSS. REPLY , 1991 .
[142] J. Svendsen. Myocardial capillary permeability for small hydrophilic indicators during normal physiological conditions and after ischemia and reperfusion. , 1991, Acta physiologica Scandinavica. Supplementum.
[143] R. Virmani,et al. Reduction of myocardial reperfusion injury by intravenous adenosine administered during the early reperfusion period. , 1991, Circulation.
[144] A. M. Lefer,et al. Time course and mechanism of endothelial dysfunction in isolated ischemic- and hypoxic-perfused rat hearts. , 1990, The American journal of physiology.
[145] D. Das,et al. Prevention of Myocardial Reperfusion Injury in Experimental Coronary Revascularization Following Ischemic Arrest by a Novel Antiinflammatory Drug, ONO-3144 , 1990, Journal of cardiovascular pharmacology.
[146] W. W. Bailey,et al. Complement activation and lung permeability during cardiopulmonary bypass. , 1990, The Annals of thoracic surgery.
[147] A. M. Lefer,et al. Time course of endothelial dysfunction and myocardial injury during myocardial ischemia and reperfusion in the cat. , 1990, Circulation.
[148] G. R. Carson,et al. Soluble human complement receptor type 1: in vivo inhibitor of complement suppressing post-ischemic myocardial inflammation and necrosis. , 1990, Science.
[149] J. V. van Mourik,et al. Characterization of the enhanced adhesion of neutrophil leukocytes to thrombin-stimulated endothelial cells. , 1990, Journal of immunology.
[150] B. Cronstein,et al. The adenosine/neutrophil paradox resolved: human neutrophils possess both A1 and A2 receptors that promote chemotaxis and inhibit O2 generation, respectively. , 1990, The Journal of clinical investigation.
[151] J. Gorski,et al. PECAM-1 (CD31) cloning and relation to adhesion molecules of the immunoglobulin gene superfamily. , 1990, Science.
[152] Rodger P. McEver,et al. Rapid neutrophil adhesion to activated endothelium mediated by GMP-140 , 1990, Nature.
[153] B. Babior. The respiratory burst oxidase of human neutrophils , 1990 .
[154] G. Johnson,et al. Cardioprotective effects of acidified sodium nitrite in myocardial ischemia with reperfusion. , 1990, The Journal of pharmacology and experimental therapeutics.
[155] D. Janero,et al. Influence of cardioprotective cyclooxygenase and lipoxygenase inhibitors on peroxidative injury to myocardial-membrane phospholipid. , 1989, Biochemical pharmacology.
[156] A. Buda,et al. Neutrophil depletion fails to modify myocardial no reflow and functional recovery after coronary reperfusion. , 1989, Journal of the American College of Cardiology.
[157] L. Becker,et al. Neutrophil depletion limited to reperfusion reduces myocardial infarct size after 90 minutes of ischemia. Evidence for neutrophil-mediated reperfusion injury. , 1989, Circulation.
[158] W. Dreyer,et al. Canine Neutrophil Activation by Cardiac Lymph Obtained During Reperfiision of Ischemic Myocardium , 1989, Circulation research.
[159] K. Mizuno,et al. Monoclonal Antibody , 2020, Definitions.
[160] M. Tonnesen. Neutrophil-endothelial cell interactions: mechanisms of neutrophil adherence to vascular endothelium. , 1989, The Journal of investigative dermatology.
[161] A. Stephenson,et al. Regulation of human neutrophil functions by adenine nucleotides. , 1989, Journal of immunology.
[162] B. Babior,et al. The respiratory burst oxidase , 1994, Basic life sciences.
[163] E. Smith,et al. Temporal relation between neutrophil accumulation and myocardial reperfusion injury. , 1988, The American journal of physiology.
[164] D. Blake,et al. Inflammation: Basic Principles and Clinical Correlates , 1988 .
[165] R. Virmani,et al. Reduction of reperfusion injury in the canine preparation by intracoronary adenosine: importance of the endothelium and the no-reflow phenomenon. , 1987, Circulation.
[166] G. Schmid-Schönbein. Capillary plugging by granulocytes and the no-reflow phenomenon in the microcirculation. , 1987, Federation proceedings.
[167] L. Horwitz,et al. Reperfusion after acute coronary occlusion in dogs impairs endothelium-dependent relaxation to acetylcholine and augments contractile reactivity in vitro. , 1987, The Journal of clinical investigation.
[168] D. Schrier,et al. The effects of adenosine agonists on human neutrophil function. , 1986, Journal of immunology.
[169] F. Rossi. The O2- -forming NADPH oxidase of the phagocytes: nature, mechanisms of activation and function. , 1986, Biochimica et biophysica acta.
[170] T. Orszulak,et al. Oxygen free radical generation during cardiopulmonary bypass: correlation with complement activation. , 1986, Circulation.
[171] G. Buckberg,et al. Studies of controlled reperfusion after ischemia. , 1986, The Journal of thoracic and cardiovascular surgery.
[172] G. Weissmann,et al. Adenosine: an endogenous inhibitor of neutrophil-mediated injury to endothelial cells. , 1986, The Journal of clinical investigation.
[173] G. Schmid-Schönbein,et al. Role of leukocytes in response to acute myocardial ischemia and reflow in dogs. , 1986, The American journal of physiology.
[174] G. Schmid-Schönbein,et al. Accumulation of polymorphonuclear leukocytes during 3-h experimental myocardial ischemia. , 1986, The American journal of physiology.
[175] P. Vanhoutte,et al. Superoxide anions and hyperoxia inactivate endothelium-derived relaxing factor. , 1986, The American journal of physiology.
[176] S. Moncada,et al. Superoxide anion is involved in the breakdown of endothelium-derived vascular relaxing factor , 1986, Nature.
[177] G. Kammer. ADENOSINE , 1986, The Lancet.
[178] H. Ohto,et al. A novel leukocyte differentiation antigen: two monoclonal antibodies TM2 and TM3 define a 120-kd molecule present on neutrophils, monocytes, platelets, and activated lymphoblasts. , 1985, Blood.
[179] S. B. Kramer,et al. Adenosine; a physiologic modulator of superoxide anion generation by human neutrophils. Adenosine acts via an A2 receptor on human neutrophils. , 1985, Journal of immunology.
[180] M. Entman,et al. Selective Accumulation of the First Component of Complement and Leukocytes in Ischemic Canine Heart Muscle: A Possible Initiator of an Extra Myocardial Mechanism of Ischemic Injury , 1985, Circulation research.
[181] S Westaby,et al. Complement and the damaging effects of cardiopulmonary bypass. , 1983, The Journal of thoracic and cardiovascular surgery.
[182] W. J. Powell,et al. Early membrane damage during coronary reperfusion in dogs. Detection by radiolabeled anticardiac myosin (Fab')2. , 1983, The Journal of clinical investigation.
[183] M. Schork,et al. Reduction of the Extent of Ischemic Myocardial Injury by Neutrophil Depletion in the Dog , 1983, Circulation.
[184] L. McManus,et al. Complement localization in ischemic baboon myocardium. , 1983, Laboratory investigation; a journal of technical methods and pathology.
[185] G. Schmid-Schönbein,et al. Leukocyte capillary plugging in myocardial ischemia and reperfusion in the dog. , 1983, The American journal of pathology.
[186] E. Blackstone,et al. Complement activation during cardiopulmonary bypass: evidence for generation of C3a and C5a anaphylatoxins. , 1981, The New England journal of medicine.
[187] L. Gordon,et al. Modulation of neutrophil function by lysozyme. Potential negative feedback system of inflammation. , 1979, The Journal of clinical investigation.
[188] R. Kloner,et al. The "no-reflow" phenomenon after temporary coronary occlusion in the dog. , 1974, The Journal of clinical investigation.
[189] Role of causality in ensuring unconditional security of relativistic quantum cryptography , .
[190] D. Garcia-Dorado,et al. Cardiovascular Research , 1966 .