Application of Cluster Analysis and Development of A Lifecycle Environmental Performance Indicator to Categorise Construction Materials

[1]  R. Harrison,et al.  AIRUSE-LIFE+: a harmonized PM speciation and source apportionment in five southern European cities , 2015 .

[2]  Morton Lippmann,et al.  Particulate matter neurotoxicity in culture is size-dependent. , 2013, Neurotoxicology.

[3]  Roberta Vecchi,et al.  A study on nighttime–daytime PM10 concentration and elemental composition in relation to atmospheric dispersion in the urban area of Milan (Italy) , 2007 .

[4]  Ulrich Pöschl,et al.  Atmospheric aerosols: composition, transformation, climate and health effects. , 2005, Angewandte Chemie.

[5]  M. Casazza,et al.  Some remarks on PM2.5 , 2003 .

[6]  C. Tanner,et al.  Projected number of people with Parkinson disease in the most populous nations, 2005 through 2030 , 2007, Neurology.

[7]  G Gilli,et al.  Meteo-climatic conditions influence the contribution of endotoxins to PM10 in an urban polluted environment. , 2010, Journal of environmental monitoring : JEM.

[8]  Roy M Harrison,et al.  Estimation of the contributions of brake dust, tire wear, and resuspension to nonexhaust traffic particles derived from atmospheric measurements. , 2012, Environmental science & technology.

[9]  Joel Schwartz,et al.  Exposure to particulate air pollution and cognitive decline in older women. , 2011, Archives of internal medicine.

[10]  J. Schwartz,et al.  Traffic-Related Air Pollution and Cognitive Function in a Cohort of Older Men , 2010, Environmental health perspectives.

[11]  Takahiro Kobayashi,et al.  Changes in neurotransmitter levels and proinflammatory cytokine mRNA expressions in the mice olfactory bulb following nanoparticle exposure. , 2008, Toxicology and applied pharmacology.

[12]  C. Henríquez-Roldán,et al.  Long-term Air Pollution Exposure Is Associated with Neuroinflammation, an Altered Innate Immune Response, Disruption of the Blood-Brain Barrier, Ultrafine Particulate Deposition, and Accumulation of Amyloid β-42 and α-Synuclein in Children and Young Adults , 2008, Toxicologic pathology.

[13]  Roderic B. Mast,et al.  Data and Maps , 2015 .

[14]  J. D. Robertson,et al.  Copper, iron and zinc in Alzheimer's disease senile plaques , 1998, Journal of the Neurological Sciences.

[15]  E Dybing,et al.  Release of inflammatory cytokines, cell toxicity and apoptosis in epithelial lung cells after exposure to ambient air particles of different size fractions. , 2004, Toxicology in vitro : an international journal published in association with BIBRA.

[16]  John Peterson Myers,et al.  Environmental exposures and gene regulation in disease etiology. , 2007, Ciencia & saude coletiva.

[17]  Cristina Pignata,et al.  Mutagenic properties of PM2.5 urban pollution in the northern Italy: the nitro-compounds contribution. , 2009, Environment international.

[18]  Christer Johansson,et al.  Road traffic emission factors for heavy metals , 2009 .

[19]  Kazuhiko Sakamoto,et al.  Estimation of the contribution of ultrafine particles to lung deposition of particle-bound mutagens in the atmosphere. , 2011, The Science of the total environment.

[20]  Marco Casazza,et al.  PM10 size distribution of metals and environmental-sanitary risk analysis in the city of Torino. , 2014, Chemosphere.

[21]  Hongtu Zhu,et al.  The impact of environmental metals in young urbanites' brains. , 2013, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.

[22]  Marco Casazza,et al.  Thirty-years assessment of size-fractionated particle mass concentrations in a polluted urban area and its implications for the regulatory framework , 2013 .

[23]  Rosalind J Wright,et al.  Association of black carbon with cognition among children in a prospective birth cohort study. , 2007, American journal of epidemiology.

[24]  J. Olesen,et al.  Cost of disorders of the brain in Europe , 2005, European journal of neurology.

[25]  B. Fowler,et al.  Roles of biomarkers in evaluating interactions among mixtures of lead, cadmium and arsenic. , 2008, Toxicology and applied pharmacology.

[26]  J. Kleinjans,et al.  Genotoxicity and physicochemical characteristics of traffic‐related ambient particulate matter , 2005, Environmental and molecular mutagenesis.

[27]  M. Fang,et al.  Cytotoxicity of PM(2.5) and PM(2.5--10) ambient air pollutants assessed by the MTT and the Comet assays. , 2000, Mutation research.

[28]  H. Gerwig,et al.  Influence of seasons, air mass origin and day of the week on size-segregated chemical composition of aerosol particles at a kerbside , 2009 .

[29]  A. Hartwig,et al.  Carcinogenic metal compounds: recent insight into molecular and cellular mechanisms , 2008, Archives of Toxicology.

[30]  Fabrice Cazier,et al.  Genotoxic potential of Polycyclic Aromatic Hydrocarbons-coated onto airborne Particulate Matter (PM 2.5) in human lung epithelial A549 cells. , 2008, Cancer letters.

[31]  Lucia Migliore,et al.  Environmental-induced oxidative stress in neurodegenerative disorders and aging. , 2009, Mutation research.

[32]  Raymond Agius,et al.  Total and water-soluble trace metal content of urban background PM10, PM2.5 and black smoke in Edinburgh, UK , 2005 .

[33]  J. Olesen,et al.  The burden of brain diseases in Europe , 2003, European journal of neurology.

[34]  Michela Ponzio,et al.  Mutagenic properties of PM2.5 air pollution in the Padana Plain (Italy) before and in the course of XX Winter Olympic Games of "Torino 2006". , 2008, Environment international.

[35]  J. McDonald,et al.  Air pollution & the brain: Subchronic diesel exhaust exposure causes neuroinflammation and elevates early markers of neurodegenerative disease , 2011, Journal of Neuroinflammation.

[36]  K E Noll,et al.  Size distribution of atmospheric giant particles. , 1971, Atmospheric environment.